Example of Journal of Neuroimmune Pharmacology format
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Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format
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Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format Example of Journal of Neuroimmune Pharmacology format
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open access Open Access
recommended Recommended

Journal of Neuroimmune Pharmacology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Neuroscience (miscellaneous) #2 of 24 up up by 3 ranks
Pharmacology #45 of 297 up up by 20 ranks
Immunology and Allergy #41 of 182 up up by 10 ranks
Immunology #49 of 202 up up by 13 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 204 Published Papers | 1573 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/07/2020
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Related Journals

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Quality:  
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SJR: 5.06
SNIP: 2.281
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CiteRatio: 8.4
SJR: 2.078
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SJR: 1.864
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Quality:  
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CiteRatio: 53.9
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.113

6% from 2018

Impact factor for Journal of Neuroimmune Pharmacology from 2016 - 2019
Year Value
2019 4.113
2018 3.87
2017 3.664
2016 3.339
graph view Graph view
table view Table view

7.7

7% from 2019

CiteRatio for Journal of Neuroimmune Pharmacology from 2016 - 2020
Year Value
2020 7.7
2019 7.2
2018 6.9
2017 6.3
2016 8.4
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 6% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 7% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.219

1% from 2019

SJR for Journal of Neuroimmune Pharmacology from 2016 - 2020
Year Value
2020 1.219
2019 1.235
2018 1.333
2017 1.379
2016 1.774
graph view Graph view
table view Table view

0.854

12% from 2019

SNIP for Journal of Neuroimmune Pharmacology from 2016 - 2020
Year Value
2020 0.854
2019 0.965
2018 0.889
2017 0.817
2016 1.069
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 1% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 12% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Neuroimmune Pharmacology

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Springer

Journal of Neuroimmune Pharmacology

Approved by publishing and review experts on SciSpace, this template is built as per for Journal of Neuroimmune Pharmacology formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 247 authors to write and format their manuscripts to this journal.

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Last updated on
12 Jul 2020
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ISSN
1557-1904
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Open Access
Hybrid
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S11481-009-9164-4
Heterogeneity of Microglial Activation in the Innate Immune Response in the Brain
Carol A. Colton1

Abstract:

The immune response in the brain has been widely investigated and while many studies have focused on the proinflammatory cytotoxic response, the brain’s innate immune system demonstrates significant heterogeneity. Microglia, like other tissue macrophages, participate in repair and resolution processes after infection or injur... The immune response in the brain has been widely investigated and while many studies have focused on the proinflammatory cytotoxic response, the brain’s innate immune system demonstrates significant heterogeneity. Microglia, like other tissue macrophages, participate in repair and resolution processes after infection or injury to restore normal tissue homeostasis. This review examines the mechanisms that lead to reduction of self-toxicity and to repair and restructuring of the damaged extracellular matrix in the brain. Part of the resolution process involves switching macrophage functional activation to include reduction of proinflammatory mediators, increased production and release of anti-inflammatory cytokines, and production of cytoactive factors involved in repair and reconstruction of the damaged brain. Two partially overlapping and complimentary functional macrophage states have been identified and are called alternative activation and acquired deactivation. The immunosuppressive and repair processes of each of these states and how alternative activation and acquired deactivation participate in chronic neuroinflammation in the brain are discussed. read more read less

Topics:

Neuroinflammation (56%)56% related to the paper, Proinflammatory cytokine (53%)53% related to the paper, Innate immune system (52%)52% related to the paper, Microglia (51%)51% related to the paper, Immune system (51%)51% related to the paper
View PDF
799 Citations
Journal Article DOI: 10.1007/S11481-006-9025-3
Blood-brain barrier: structural components and function under physiologic and pathologic conditions
Yuri Persidsky1, Servio H. Ramirez1, James Haorah1, Georgette D. Kanmogne1

Abstract:

The blood–brain barrier (BBB) is the specialized system of brain microvascular endothelial cells (BMVEC) that shields the brain from toxic substances in the blood, supplies brain tissues with nutrients, and filters harmful compounds from the brain back to the bloodstream. The close interaction between BMVEC and other componen... The blood–brain barrier (BBB) is the specialized system of brain microvascular endothelial cells (BMVEC) that shields the brain from toxic substances in the blood, supplies brain tissues with nutrients, and filters harmful compounds from the brain back to the bloodstream. The close interaction between BMVEC and other components of the neurovascular unit (astrocytes, pericytes, neurons, and basement membrane) ensures proper function of the central nervous system (CNS). Transport across the BBB is strictly limited through both physical (tight junctions) and metabolic barriers (enzymes, diverse transport systems). A functional polarity exists between the luminal and abluminal membrane surfaces of the BMVEC. As a result of restricted permeability, the BBB is a limiting factor for the delivery of therapeutic agents into the CNS. BBB breakdown or alterations in transport systems play an important role in the pathogenesis of many CNS diseases (HIV-1 encephalitis, Alzheimer's disease, ischemia, tumors, multiple sclerosis, and Parkinson's disease). Proinflammatory substances and specific disease-associated proteins often mediate such BBB dysfunction. Despite seemingly diverse underlying causes of BBB dysfunction, common intracellular pathways emerge for the regulation of the BBB structural and functional integrity. Better understanding of tight junction regulation and factors affecting transport systems will allow the development of therapeutics to improve the BBB function in health and disease. read more read less

Topics:

Blood–brain barrier (56%)56% related to the paper
768 Citations
Journal Article DOI: 10.1007/S11481-006-9015-5
Microglia Biology in Health and Disease
Gwenn A. Garden1, Thomas Möller1

Abstract:

Microglia cells are resident central nervous system (CNS) leukocytes that regulate innate immunity and participate in adaptive immune responses in CNS tissue. However, microglia cells also appear to play an important role during normal function of the mature nervous system. In response to injury, ischemia, and inflammatory st... Microglia cells are resident central nervous system (CNS) leukocytes that regulate innate immunity and participate in adaptive immune responses in CNS tissue. However, microglia cells also appear to play an important role during normal function of the mature nervous system. In response to injury, ischemia, and inflammatory stimuli, microglia cells assume an activated phenotype associated with proliferation, migration to the site of injury, phagocytosis of cellular debris, and elaboration (Power and Proudfoot 2001) of both neurotoxic and neurotrophic factors. Recent reports strongly suggest that regulating microglia function may be a fruitful future therapeutic target for the prevention of neurological dysfunction in a variety of CNS injuries and chronic diseases. Thus, developing a thorough understanding of extracellular signals that activate microglia as well as a complete catalogue of microglia responses to activating stimuli in both the healthy and diseased state are crucial scientific endeavors. This review presents the current understanding of the biology of microglia during normal CNS function as well as in response to CNS injury or neurodegenerative disease. In addition, microglia modulate both the activation and down-regulation of the adaptive immune response in the CNS. Evidence that microglia cells play a primary role in regulating CNS immune responses will also be discussed. read more read less

Topics:

Neuroinflammation (66%)66% related to the paper, Microglia (54%)54% related to the paper, Acquired immune system (53%)53% related to the paper, Innate immune system (51%)51% related to the paper, Neurotrophic factors (51%)51% related to the paper
475 Citations
open accessOpen access Journal Article DOI: 10.1007/S11481-020-09944-5
The Natural History, Pathobiology, and Clinical Manifestations of SARS-CoV-2 Infections.

Abstract:

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the etiological agent of coronavirus disease 2019 (COVID-19). SARS-CoV-2, is a positive-sense single-stranded RNA virus with epithelial cell and respiratory system proclivity. Like its predecessor, SARS-CoV, COVID-19 can lead to life-threatening disease. Due to w... Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the etiological agent of coronavirus disease 2019 (COVID-19). SARS-CoV-2, is a positive-sense single-stranded RNA virus with epithelial cell and respiratory system proclivity. Like its predecessor, SARS-CoV, COVID-19 can lead to life-threatening disease. Due to wide geographic impact affecting an extremely high proportion of the world population it was defined by the World Health Organization as a global public health pandemic. The infection is known to readily spread from person-to-person. This occurs through liquid droplets by cough, sneeze, hand-to-mouth-to-eye contact and through contaminated hard surfaces. Close human proximity accelerates SARS-CoV-2 spread. COVID-19 is a systemic disease that can move beyond the lungs by blood-based dissemination to affect multiple organs. These organs include the kidney, liver, muscles, nervous system, and spleen. The primary cause of SARS-CoV-2 mortality is acute respiratory distress syndrome initiated by epithelial infection and alveolar macrophage activation in the lungs. The early cell-based portal for viral entry is through the angiotensin-converting enzyme 2 receptor. Viral origins are zoonotic with genomic linkages to the bat coronaviruses but without an identifiable intermediate animal reservoir. There are currently few therapeutic options, and while many are being tested, although none are effective in curtailing the death rates. There is no available vaccine yet. Intense global efforts have targeted research into a better understanding of the epidemiology, molecular biology, pharmacology, and pathobiology of SARS-CoV-2. These fields of study will provide the insights directed to curtailing this disease outbreak with intense international impact. Graphical Abstract. read more read less

Topics:

Disease (50%)50% related to the paper
View PDF
365 Citations
open accessOpen access Journal Article DOI: 10.1007/S11481-009-9176-0
Neuroinflammation in Parkinson’s Disease
Jae-Kyung Lee1, Thi Nguyet Anh Tran1, Malú G. Tansey1, Malú G. Tansey2

Abstract:

During the last two decades, a wealth of animal and human studies has implicated inflammation-derived oxidative stress and cytokine-dependent neurotoxicity in the progressive degeneration of the dopaminergic nigrostriatal pathway, the hallmark of Parkinson’s disease (PD). In this review, we discuss the various hypotheses rega... During the last two decades, a wealth of animal and human studies has implicated inflammation-derived oxidative stress and cytokine-dependent neurotoxicity in the progressive degeneration of the dopaminergic nigrostriatal pathway, the hallmark of Parkinson’s disease (PD). In this review, we discuss the various hypotheses regarding the role of microglia and other immune cells in PD pathogenesis and progression, the inflammatory mechanisms implicated in disease progression from pre-clinical and clinical studies, the recent evidence that systemic inflammation can trigger microglia activation in PD-relevant central nervous system regions, the synergism between gene products linked to parkinsonian phenotypes (α-synuclein, parkin, Nurr1, and regulator of G-protein signaling-10) and neuroinflammation in promoting neurodegeneration of the nigrostriatal pathway, and the latest update on meta-analysis of epidemiological studies on the risk-lowering effects of anti-inflammatory drug regimens. read more read less

Topics:

Neuroinflammation (58%)58% related to the paper, Parkin (57%)57% related to the paper, Parkinson's disease (52%)52% related to the paper, Neurodegeneration (52%)52% related to the paper, Nigrostriatal pathway (52%)52% related to the paper
View PDF
356 Citations
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Frequently asked questions

1. Can I write Journal of Neuroimmune Pharmacology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Neuroimmune Pharmacology guidelines and auto format it.

2. Do you follow the Journal of Neuroimmune Pharmacology guidelines?

Yes, the template is compliant with the Journal of Neuroimmune Pharmacology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Neuroimmune Pharmacology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Neuroimmune Pharmacology citation style.

4. Can I use the Journal of Neuroimmune Pharmacology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Neuroimmune Pharmacology.

5. Can I use a manuscript in Journal of Neuroimmune Pharmacology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Neuroimmune Pharmacology that you can download at the end.

6. How long does it usually take you to format my papers in Journal of Neuroimmune Pharmacology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Journal of Neuroimmune Pharmacology.

7. Where can I find the template for the Journal of Neuroimmune Pharmacology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Neuroimmune Pharmacology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Neuroimmune Pharmacology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Neuroimmune Pharmacology an online tool or is there a desktop version?

SciSpace's Journal of Neuroimmune Pharmacology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Journal of Neuroimmune Pharmacology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Journal of Neuroimmune Pharmacology?”

11. What is the output that I would get after using Journal of Neuroimmune Pharmacology?

After writing your paper autoformatting in Journal of Neuroimmune Pharmacology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Neuroimmune Pharmacology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Neuroimmune Pharmacology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Neuroimmune Pharmacology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Neuroimmune Pharmacology?

The 5 most common citation types in order of usage for Journal of Neuroimmune Pharmacology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Neuroimmune Pharmacology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Neuroimmune Pharmacology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Journal of Neuroimmune Pharmacology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Neuroimmune Pharmacology Endnote style according to Elsevier guidelines.

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