Example of Molecular Brain format
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Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format Example of Molecular Brain format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Molecular Brain — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Molecular Biology #176 of 382 down down by 69 ranks
Cellular and Molecular Neuroscience #48 of 88 down down by 20 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 421 Published Papers | 2248 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 14/06/2020
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Related Journals

open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 7.3
SJR: 2.628
SNIP: 1.713
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Frontiers Media

Quality:  
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CiteRatio: 8.4
SJR: 1.989
SNIP: 1.224
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Springer

Quality:  
High
CiteRatio: 12.8
SJR: 2.928
SNIP: 1.815
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 19.1
SJR: 5.565
SNIP: 2.172

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.686

16% from 2018

Impact factor for Molecular Brain from 2016 - 2019
Year Value
2019 4.686
2018 4.051
2017 3.449
2016 3.41
graph view Graph view
table view Table view

5.3

15% from 2019

CiteRatio for Molecular Brain from 2016 - 2020
Year Value
2020 5.3
2019 6.2
2018 6.8
2017 6.6
2016 6.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 16% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 15% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.748

10% from 2019

SJR for Molecular Brain from 2016 - 2020
Year Value
2020 1.748
2019 1.945
2018 2.109
2017 1.805
2016 2.106
graph view Graph view
table view Table view

1.079

1% from 2019

SNIP for Molecular Brain from 2016 - 2020
Year Value
2020 1.079
2019 1.087
2018 1.201
2017 0.938
2016 1.01
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 10% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 1% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Molecular Brain

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Springer

Molecular Brain

Approved by publishing and review experts on SciSpace, this template is built as per for Molecular Brain formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 327 authors to write and format their manuscripts to this journal.

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Last updated on
14 Jun 2020
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ISSN
1606-8610
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/1756-6606-5-14
The molecular biology of memory: CAMP, PKA, CRE, CREB-1, CREB-2, and CPEB
Eric R. Kandel1
14 May 2012 - Molecular Brain

Abstract:

The analysis of the contributions to synaptic plasticity and memory of cAMP, PKA, CRE, CREB-1, CREB-2, and CPEB has recruited the efforts of many laboratories all over the world. These are six key steps in the molecular biological delineation of short-term memory and its conversion to long-term memory for both implicit (proce... The analysis of the contributions to synaptic plasticity and memory of cAMP, PKA, CRE, CREB-1, CREB-2, and CPEB has recruited the efforts of many laboratories all over the world. These are six key steps in the molecular biological delineation of short-term memory and its conversion to long-term memory for both implicit (procedural) and explicit (declarative) memory. I here first trace the background for the clinical and behavioral studies of implicit memory that made a molecular biology of memory storage possible, and then detail the discovery and early history of these six molecular steps and their roles in explicit memory. read more read less

Topics:

Implicit memory (62%)62% related to the paper, Explicit memory (57%)57% related to the paper, CPEB (56%)56% related to the paper, CREB (54%)54% related to the paper
View PDF
738 Citations
open accessOpen access Journal Article DOI: 10.1186/1756-6606-4-3
APP processing in Alzheimer's disease
Yun-wu Zhang1, Robert C. Thompson2, Han Zhang1, Han Zhang2, Huaxi Xu2, Huaxi Xu1
07 Jan 2011 - Molecular Brain

Abstract:

An important pathological feature of Alzheimer's disease (AD) is the presence of extracellular senile plaques in the brain. Senile plaques are composed of aggregations of small peptides called β-amyloid (Aβ). Multiple lines of evidence demonstrate that overproduction/aggregation of Aβ in the brain is a primary cause of AD and... An important pathological feature of Alzheimer's disease (AD) is the presence of extracellular senile plaques in the brain. Senile plaques are composed of aggregations of small peptides called β-amyloid (Aβ). Multiple lines of evidence demonstrate that overproduction/aggregation of Aβ in the brain is a primary cause of AD and inhibition of Aβ generation has become a hot topic in AD research. Aβ is generated from β-amyloid precursor protein (APP) through sequential cleavages first by β-secretase and then by γ-secretase complex. Alternatively, APP can be cleaved by α-secretase within the Aβ domain to release soluble APPα and preclude Aβ generation. Cleavage of APP by caspases may also contribute to AD pathologies. Therefore, understanding the metabolism/processing of APP is crucial for AD therapeutics. Here we review current knowledge of APP processing regulation as well as the patho/physiological functions of APP and its metabolites. read more read less

Topics:

P3 peptide (62%)62% related to the paper, Senile plaques (57%)57% related to the paper, Amyloid precursor protein (52%)52% related to the paper
View PDF
723 Citations
open accessOpen access Journal Article DOI: 10.1186/1756-6606-5-35
Mitochondrial dysfunction associated with increased oxidative stress and α-synuclein accumulation in PARK2 iPSC-derived neurons and postmortem brain tissue.
06 Oct 2012 - Molecular Brain

Abstract:

Background Parkinson’s disease (PD) is a neurodegenerative disease characterized by selective degeneration of dopaminergic neurons in the substantia nigra (SN). The familial form of PD, PARK2, is caused by mutations in the parkin gene. parkin-knockout mouse models show some abnormalities, but they do not fully recapitulate t... Background Parkinson’s disease (PD) is a neurodegenerative disease characterized by selective degeneration of dopaminergic neurons in the substantia nigra (SN). The familial form of PD, PARK2, is caused by mutations in the parkin gene. parkin-knockout mouse models show some abnormalities, but they do not fully recapitulate the pathophysiology of human PARK2. read more read less

Topics:

Parkin (61%)61% related to the paper, Substantia nigra (60%)60% related to the paper, Alpha-synuclein (56%)56% related to the paper, Parkinson's disease (54%)54% related to the paper
View PDF
329 Citations
open accessOpen access Journal Article DOI: 10.1186/S13041-017-0340-9
Oxidative stress and cellular pathologies in Parkinson's disease.
Lesly Puspita1, Sun Young Chung2, Jae-Won Shim1
28 Nov 2017 - Molecular Brain

Abstract:

Parkinson’s disease (PD) is a chronic and progressive neurodegeneration of dopamine neurons in the substantia nigra. The reason for the death of these neurons is unclear; however, studies have demonstrated the potential involvement of mitochondria, endoplasmic reticulum, α-synuclein or dopamine levels in contributing to cellu... Parkinson’s disease (PD) is a chronic and progressive neurodegeneration of dopamine neurons in the substantia nigra. The reason for the death of these neurons is unclear; however, studies have demonstrated the potential involvement of mitochondria, endoplasmic reticulum, α-synuclein or dopamine levels in contributing to cellular oxidative stress as well as PD symptoms. Even though those papers had separately described the individual roles of each element leading to neurodegeneration, recent publications suggest that neurodegeneration is the product of various cellular interactions. This review discusses the role of oxidative stress in mediating separate pathological events that together, ultimately result in cell death in PD. Understanding the multi-faceted relationships between these events, with oxidative stress as a common denominator underlying these processes, is needed for developing better therapeutic strategies. read more read less

Topics:

Neurodegeneration (59%)59% related to the paper, Parkinson's disease (53%)53% related to the paper, Substantia nigra (52%)52% related to the paper, Oxidative stress (52%)52% related to the paper, Unfolded protein response (51%)51% related to the paper
View PDF
301 Citations
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With SciSpace, you do not need a word template for Molecular Brain.

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You can download a submission ready research paper in pdf, LaTeX and docx formats.

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Time taken to format a paper and Compliance with guidelines

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Frequently asked questions

1. Can I write Molecular Brain in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Molecular Brain guidelines and auto format it.

2. Do you follow the Molecular Brain guidelines?

Yes, the template is compliant with the Molecular Brain guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Molecular Brain?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Molecular Brain citation style.

4. Can I use the Molecular Brain templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Molecular Brain.

5. Can I use a manuscript in Molecular Brain that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Molecular Brain that you can download at the end.

6. How long does it usually take you to format my papers in Molecular Brain?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Molecular Brain.

7. Where can I find the template for the Molecular Brain?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Brain's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Molecular Brain's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Molecular Brain an online tool or is there a desktop version?

SciSpace's Molecular Brain is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Molecular Brain?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Molecular Brain?”

11. What is the output that I would get after using Molecular Brain?

After writing your paper autoformatting in Molecular Brain, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Molecular Brain's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Molecular Brain?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Molecular Brain. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Molecular Brain?

The 5 most common citation types in order of usage for Molecular Brain are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Molecular Brain?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Brain's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Molecular Brain in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Molecular Brain Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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