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Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format
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Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format Example of Clinical Endocrinology format
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This content is only for preview purposes. The original open access content can be found here.
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Clinical Endocrinology — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Endocrinology, Diabetes and Metabolism #49 of 219 down down by 2 ranks
Endocrinology #36 of 117 down down by 4 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 768 Published Papers | 4719 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 15/06/2020
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Related Journals

open access Open Access

Springer

Quality:  
High
CiteRatio: 6.0
SJR: 1.141
SNIP: 1.158
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open access Open Access

Springer

Quality:  
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CiteRatio: 6.1
SJR: 1.034
SNIP: 1.318

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.38

17% from 2018

Impact factor for Clinical Endocrinology from 2016 - 2019
Year Value
2019 3.38
2018 2.897
2017 3.077
2016 3.327
graph view Graph view
table view Table view

6.1

5% from 2019

CiteRatio for Clinical Endocrinology from 2016 - 2020
Year Value
2020 6.1
2019 5.8
2018 5.8
2017 6.3
2016 6.7
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 17% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.055

15% from 2019

SJR for Clinical Endocrinology from 2016 - 2020
Year Value
2020 1.055
2019 1.245
2018 1.234
2017 1.359
2016 1.345
graph view Graph view
table view Table view

1.279

5% from 2019

SNIP for Clinical Endocrinology from 2016 - 2020
Year Value
2020 1.279
2019 1.221
2018 1.138
2017 1.259
2016 1.306
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 15% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 5% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Clinical Endocrinology

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Wiley

Clinical Endocrinology

Clinical Endocrinology publishes papers and reviews which focus on the clinical aspects of endocrinology, including the clinical application of molecular endocrinology. It features reviews, original papers, commentaries, cases of the month, book reviews and letters to the edit...... Read More

Endocrinology, Diabetes and Metabolism

Medicine

i
Last updated on
14 Jun 2020
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ISSN
0300-0664
i
Impact Factor
High - 1.338
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
apa
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Citation Type
Numbered
[25]
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Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1111/J.1365-2265.1977.TB01340.X
The spectrum of thyroid disease in a community: the whickham survey
W.M.G. Tunbridge1, D. C. Evered1, Reginald Hall1, D. Appleton1, M Brewis1, F. Clark1, J G Evans1, E. T. Young1, T. Bird1, P. A. Smith1
01 Dec 1977 - Clinical Endocrinology

Abstract:

SUMMARY A survey has been conducted in Whickham, County Durham, to determine the prevalence of thyroid disorders in the community. Two thousand seven hundred and seventy-nine people (82.4% of the available sample) were seen in the survey. The prevalence of overt hyperthyroidism was 19/1000 females rising to 27/1000 females w... SUMMARY A survey has been conducted in Whickham, County Durham, to determine the prevalence of thyroid disorders in the community. Two thousand seven hundred and seventy-nine people (82.4% of the available sample) were seen in the survey. The prevalence of overt hyperthyroidism was 19/1000 females rising to 27/1000 females when possible cases were included, compared with 1.6–2.3/1000 males. The prevalence of overt hyothyroidism was 14/1000 females rising to 19/1000 females when possible cases were included, compared with less than 1/1000 males. The prevalence of spontaneous overt hypothyroidism (excluding iatrogenic cases) was 10/1000 females or 15/1000 females including unconfirmed cases. Minor degrees of hypothyroidism were defined on the basis of elevated serum thyrotrophin (TSH) levels in the absence of obvious clinical features of hypothyroidism. TSH levels did not vary with age in males but increased markedly in females after the age of 45 years. The rise of TSH with age in females was virtually abolished when persons with thyroid antibodies were excluded from the sample. TSH levels above 6 mu/1 were shown to reflect a significant lowering of circulating thyroxine levels and showed a strong association with thyroid antibodies in both sexes, independent of age. Elevated TSH levels (>6mu/l) were recorded in 7.5% of females and 2.8% of males of all ages. Thyroglobulin antibodies were present in 2% of the sample. Thyroid cytoplasmic antibodies were present in 6.8% of the sample (females 10.3%, males 2.7%) and their frequency did not vary significantly with age in males but increased markedly in females over 45 years of age. 3% of the sample (females 5.1%, males 1.1%) had thyroid antibodies and elevated TSH levels and the relative risk of a high TSH level in subjects with antibodies was 20:1 for males and 13:1 for females, independent of age. Small goitres (palpable but not visible) were found in 8.6% of the sample and obvious goitres (palpable and visible) in 6.9%. Goitres were four times more common in females than in males and were most commonly found in younger rather than older females. TSH levels were slightly but not significantly lower in those with goitre than in those without goitre. There was a weak association between goitre and antibodies in females but not males. read more read less

Topics:

Thyroid disease (53%)53% related to the paper, Anti-thyroid autoantibodies (52%)52% related to the paper
2,248 Citations
Journal Article DOI: 10.1111/J.1365-2265.1995.TB01894.X
The incidence of thyroid disorders in the community: a twenty-year follow-up of the Whickham Survey
01 Jul 1995 - Clinical Endocrinology

Abstract:

Summary BACKGROUND AND OBJECTIVE The original Whlckham Survey documented the prevalence of thyroid disorders in a randomly selected sample of 2779 adults which matched the population of Great Britain in age, sex and social class. The aim of the twenty-year follow-up survey was to determine the Incidence and natural history o... Summary BACKGROUND AND OBJECTIVE The original Whlckham Survey documented the prevalence of thyroid disorders in a randomly selected sample of 2779 adults which matched the population of Great Britain in age, sex and social class. The aim of the twenty-year follow-up survey was to determine the Incidence and natural history of thyroid disease in this cohort. DESIGN, PATIENTS AND MEASUREMENTS Subjects were traced at follow-up via the Electoral Register, General Practice registers, Gateshead Family Health Services Authority register and Office of Population Censuses and Surveys. Eight hundred and twenty-five subjects (30% of the sample) had died and, In addition to death certificates, two-thirds had Information from either hospital/General Practitioner notes or post-mortem reports to document morbidity prior to death. Of the 1877 known survivors, 96% participated in the follow-up study and 91 % were tested for clinical, biochemical and Immunological evidence of thyroid dysfunction. RESULTS Outcomes in terms of morbidity and mortality were determined for over 97% of the original sample. The mean Incidence (with 95% confidence Intervals) of spontaneous hypothyroidism in women was 3.5/1000 survivors/year (2.8-4.5) rising to 4.1/1000 survivors/year (3.3-5.0) for all causes of hypothyroidism and in men was 0.6/1000 survivors/year (0.3-1.2). The mean incidence of hyperthyroidism In women was 0.8/1000 survivors/year (0.5.1.4) and was negligible in men. Similar incidence rates were calculated for the deceased subjects. An estimate of the probability of the development of hypothyroidism and hyperthyroidism at a particular time, i.e. the hazard rate, showed an Increase with age In hypothyroidism but no age relation in hyperthyroidism. The frequency of goitre decreased with age with 10% of women and 2% of men having a goitre at follow-up, as compared to 23% and 5% in the same subjects respectively at the first survey. The presence of a goitre at either survey was not associated with any clinical or biochemical evidence of thyroid dysfunction. In women, an association was found between the development of a goitre and thyroid-antibody status at follow-up, but not initially. The risk of having developed hypothyroidism at follow-up was examined with respect to risk factors Identified at first survey. The odds ratios (with 95% confidence Intervals) of developing hypothyroidism with (a) raised serum TSH alone were 8 (3-20) for women and 44 (19-104) for men; (b) positive anti-thyroid antibodies alone were 8 (5-15) for women and 25 (10-63) for men; (c) both raised serum TSH and positive anti-thyroid antibodies were 38 (22-65) for women and 173 (81-370) for men. A loglt model Indicated that Increasing values of serum TSH above 2mU/l at first survey Increased the probability of developing hypothyroidism which was further Increased in the presence of anti-thyroid antibodies. Neither a positive family history of any form of thyroid disease nor parity of women at first survey was associated with Increased risk of developing hypothyroidism. Fasting cholesterol and triglyceride levels at first survey when corrected for age showed no association with the development of hypothyroidism In women. CONCLUSIONS This historical cohort study has provided Incidence data for thyroid disease over a twenty-year period for a representative cross-sectional sample of the population, and has allowed the determination of the importance of prognostic risk factors for thyroid disease Identified twenty years earlier. read more read less

Topics:

Thyroid disease (59%)59% related to the paper, Incidence (epidemiology) (52%)52% related to the paper, Population (52%)52% related to the paper, Odds ratio (52%)52% related to the paper, Cohort (52%)52% related to the paper
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2,134 Citations
Journal Article DOI: 10.1111/J.1365-2265.1974.TB03298.X
Sex-hormone-binding globulin.
David C. Anderson1
01 Jan 1974 - Clinical Endocrinology

Abstract:

A review was made to understand how plasma binding protein might influence sex-hormone action in target tissues. Steroids are predominately bound to plasma proteins and only unbound steroids enter the cells. Sex-hormone-binding globulin (SHBG) binds to both the main circulating steroid T and E2 but changes in SHBG concentrati... A review was made to understand how plasma binding protein might influence sex-hormone action in target tissues. Steroids are predominately bound to plasma proteins and only unbound steroids enter the cells. Sex-hormone-binding globulin (SHBG) binds to both the main circulating steroid T and E2 but changes in SHBG concentrations exert significant results. Increased SHBG levels increase estrogen production and decreases T activity; whereas increased androgens increase T action and inhibit SHBG production. These disturbances in hormone maintenance may lead to abnormal adult sex differentiation such as hirsutism and forms of hynaecomastia. By developing SHBG concentration measurement methods-responses of hirsutism to glucocorticoid or estrogem may be assessed. In addition the effect of thyroid hormones on SHBG may also have therapeutic implications in endocrine disease. read more read less

Topics:

Sex hormone-binding globulin (61%)61% related to the paper, Hormone (54%)54% related to the paper, Hormone receptor (53%)53% related to the paper, Estrogen (51%)51% related to the paper
View PDF
1,118 Citations
Journal Article DOI: 10.1046/J.1365-2265.1999.00639.X
Low maternal free thyroxine concentrations during early pregnancy are associated with impaired psychomotor development in infancy
01 Feb 1999 - Clinical Endocrinology

Abstract:

BACKGROUND Maternal thyroid function during early pregnancy is an important determinant of early fetal brain development because the fetal thyroid is unable to produce any T4 before 12–14 weeks' gestation. Overt maternal hypothyroidism as seen in severe iodine-deficient areas is associated with severely impaired neurological... BACKGROUND Maternal thyroid function during early pregnancy is an important determinant of early fetal brain development because the fetal thyroid is unable to produce any T4 before 12–14 weeks' gestation. Overt maternal hypothyroidism as seen in severe iodine-deficient areas is associated with severely impaired neurological development of the offspring. At present, it is not known whether low free T4 (fT4) levels during pregnancy in healthy women from iodine sufficient areas may affect fetal neurodevelopment. METHODS Neurodevelopment was assessed at 10 months of age in a cohort of 220 healthy children, born after uncomplicated pregnancies and deliveries, using the Bayley Scales of Infant Development. Maternal TSH, fT4 and TPO antibody status were assessed at 12 and 32 weeks' gestation. Maternal gestational fT4 concentration was defined as an independent parameter for child development. RESULTS Children of women with fT4 levels below the 5th (<9.8 pmol/l, n = 11) and 10th (<10.4 pmol/l, n = 22) percentiles at 12 weeks' gestation had significantly lower scores on the Bayley Psychomotor Developmental Index (PDI) scale at 10 months of age, compared to children of mothers with higher fT4 values (t test, mean difference: 14.1, 95% confidence interval (CI): 5.9–22 and 7.4, 95% CI: 1.1–13.9, respectively). At 32 weeks' gestation, no significant differences were found. In the group of women with the lowest 10th percentile fT4 concentrations at 12 weeks' gestation, a positive correlation was found between the mothers' fT4 concentration and children's PDI scores (linear regression, R: 0.46, P = 0.03). After correction for confounding variables, a fT4 concentration below the 10th percentile at 12 weeks' gestation was a significant risk factor for impaired psychomotor development (RR): 5.8, 95% CI: 1.3–12.6). CONCLUSIONS Low maternal plasma fT4 concentrations during early pregnancy may be an important risk factor for impaired infant development. read more read less

Topics:

Pregnancy (57%)57% related to the paper, Bayley Scales of Infant Development (56%)56% related to the paper, Maternal hypothyroidism (56%)56% related to the paper, Thyroid disease in pregnancy (55%)55% related to the paper, Thyroid function (54%)54% related to the paper
945 Citations
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Clinical Endocrinology format uses apa citation style.

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Frequently asked questions

1. Can I write Clinical Endocrinology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Clinical Endocrinology guidelines and auto format it.

2. Do you follow the Clinical Endocrinology guidelines?

Yes, the template is compliant with the Clinical Endocrinology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Clinical Endocrinology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Clinical Endocrinology citation style.

4. Can I use the Clinical Endocrinology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Clinical Endocrinology.

5. Can I use a manuscript in Clinical Endocrinology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Clinical Endocrinology that you can download at the end.

6. How long does it usually take you to format my papers in Clinical Endocrinology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Clinical Endocrinology.

7. Where can I find the template for the Clinical Endocrinology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical Endocrinology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Clinical Endocrinology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Clinical Endocrinology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Clinical Endocrinology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Clinical Endocrinology?”

11. What is the output that I would get after using Clinical Endocrinology?

After writing your paper autoformatting in Clinical Endocrinology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Clinical Endocrinology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Clinical Endocrinology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Clinical Endocrinology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Clinical Endocrinology?

The 5 most common citation types in order of usage for Clinical Endocrinology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Clinical Endocrinology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical Endocrinology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Clinical Endocrinology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Clinical Endocrinology Endnote style according to Elsevier guidelines.

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