Example of Environmental and Molecular Mutagenesis format
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Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format
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Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format Example of Environmental and Molecular Mutagenesis format
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open access Open Access

Environmental and Molecular Mutagenesis — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Health, Toxicology and Mutagenesis #28 of 134 down down by 14 ranks
Epidemiology #29 of 99 down down by 8 ranks
Genetics (clinical) #29 of 87 down down by 6 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 257 Published Papers | 1516 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 11/07/2020
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Related Journals

open access Open Access

Springer

Quality:  
Good
CiteRatio: 3.5
SJR: 0.774
SNIP: 1.015
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Wiley

Quality:  
Medium
CiteRatio: 3.3
SJR: 1.301
SNIP: 0.659
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recommended Recommended

PLOS

Quality:  
High
CiteRatio: 9.0
SJR: 3.587
SNIP: 1.457
open access Open Access
recommended Recommended

Cambridge University Press

Quality:  
High
CiteRatio: 8.4
SJR: 1.718
SNIP: 1.845

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.131

24% from 2018

Impact factor for Environmental and Molecular Mutagenesis from 2016 - 2019
Year Value
2019 3.131
2018 2.528
2017 3.254
2016 3.575
graph view Graph view
table view Table view

5.9

26% from 2019

CiteRatio for Environmental and Molecular Mutagenesis from 2016 - 2020
Year Value
2020 5.9
2019 4.7
2018 5.9
2017 6.7
2016 6.4
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 24% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 26% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.0

30% from 2019

SJR for Environmental and Molecular Mutagenesis from 2016 - 2020
Year Value
2020 1.0
2019 0.772
2018 1.002
2017 1.119
2016 1.51
graph view Graph view
table view Table view

1.132

31% from 2019

SNIP for Environmental and Molecular Mutagenesis from 2016 - 2020
Year Value
2020 1.132
2019 0.867
2018 0.867
2017 0.875
2016 0.989
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 30% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 31% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Environmental and Molecular Mutagenesis

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Wiley

Environmental and Molecular Mutagenesis

Environmental and Molecular Mutagenesis publishes original research articles on environmental mutagenesis. It will publish manuscripts in the six general areas of mechanisms of mutagenesis; genomics; DNA damage; replication, recombination, and repair; public health; and DNA te...... Read More

Health, Toxicology and Mutagenesis

Genetics(clinical)

Epidemiology

Environmental Science

i
Last updated on
10 Jul 2020
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ISSN
0893-6692
i
Impact Factor
High - 1.022
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
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Bibliography Name
apa
i
Citation Type
Numbered
[25]
i
Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1002/(SICI)1098-2280(2000)35:3<206::AID-EM8>3.0.CO;2-J
Single cell gel/comet assay: guidelines for in vitro and in vivo genetic toxicology testing.

Abstract:

Atthe International Workshop on Genotoxicity Test Procedures (IWGTP) held in Washington, DC, March 25-26, 1999, an expert panel met to develop guidelines for the use of the single-cell gel (SCG)/Comet assay in genetic toxicology. The expert panel reached a consensus that the optimal version of the Comet assay for identifying ... Atthe International Workshop on Genotoxicity Test Procedures (IWGTP) held in Washington, DC, March 25-26, 1999, an expert panel met to develop guidelines for the use of the single-cell gel (SCG)/Comet assay in genetic toxicology. The expert panel reached a consensus that the optimal version of the Comet assay for identifying agents with genotoxic activity was the alkaline (pH > 13) version of the assay developed by Singh et al. [1988]. The pH > 13 version is capable of detecting DNA single-strand breaks (SSB), alkali-labile sites (ALS), DNA-DNA/DNA-protein cross-linking, and SSB associated with incomplete excision repair sites. Relative to other genotoxicity tests, the advantages of the SCG assay include its demonstrated sensitivity for detecting low levels of DNA damage, the requirement for small numbers of cells per sample, its flexibility, its low costs, its ease of application, and the short time needed to complete a study. The expert panel decided that no single version of the alkaline (pH > 13) Comet assay was clearly superior. However, critical technical steps within the assay were discussed and guidelines developed for preparing slides with agarose gels, lysing cells to liberate DNA, exposing the liberated DNA to alkali to produce single-stranded DNA and to express ALS as SSB, electrophoresing the DNA using pH > 13 alkaline conditions, alkali neutralization, DNA staining, comet visualization, and data collection. Based on the current state of knowledge, the expert panel developed guidelines for conducting in vitro or in vivo Comet assays. The goal of the expert panel was to identify minimal standards for obtaining reproducible and reliable Comet data deemed suitable for regulatory submission. The expert panel used the current Organization for Economic Co-operation and Development (OECD) guidelines for in vitro and in vivo genetic toxicological studies as guides during the development of the corresponding in vitro and in vivo SCG assay guidelines. Guideline topics considered included initial considerations, principles of the test method, description of the test method, procedure, results, data analysis and reporting. Special consideration was given by the expert panel to the potential adverse effect of DNA degradation associated with cytotoxicity on the interpretation of Comet assay results. The expert panel also discussed related SCG methodologies that might be useful in the interpretation of positive Comet data. The related methodologies discussed included: (1) the use of different pH conditions during electrophoreses to discriminate between DNA strand breaks and ALS; (2) the use of repair enzymes or antibodies to detect specific classes of DNA damage; (3) the use of a neutral diffusion assay to identify apoptotic/necrotic cells; and (4) the use of the acellular SCG assay to evaluate the ability of a test substance to interact directly with DNA. The alkaline (pH > 13) Comet assay guidelines developed by the expert panel represent a work in progress. Additional information is needed before the assay can be critically evaluated for its utility in genetic toxicology. The information needed includes comprehensive data on the different sources of variability (e.g., cell to cell, gel to gel, run to run, culture to culture, animal to animal, experiment to experiment) intrinsic to the alkaline (pH > 3) SCG assay, the generation of a large database based on in vitro and in vivo testing using these guidelines, and the results of appropriately designed multilaboratory international validation studies. read more read less

Topics:

Comet assay (61%)61% related to the paper, Electrophoreses (52%)52% related to the paper, Genotoxicity (51%)51% related to the paper
View PDF
4,583 Citations
open accessOpen access Journal Article DOI: 10.1002/EM.22087
Mechanisms of DNA damage, repair, and mutagenesis.
Nimrat Chatterjee1, Graham C. Walker1

Abstract:

Living organisms are continuously exposed to a myriad of DNA damaging agents that can impact health and modulate disease-states. However, robust DNA repair and damage-bypass mechanisms faithfully protect the DNA by either removing or tolerating the damage to ensure an overall survival. Deviations in this fine-tuning are known... Living organisms are continuously exposed to a myriad of DNA damaging agents that can impact health and modulate disease-states. However, robust DNA repair and damage-bypass mechanisms faithfully protect the DNA by either removing or tolerating the damage to ensure an overall survival. Deviations in this fine-tuning are known to destabilize cellular metabolic homeostasis, as exemplified in diverse cancers where disruption or deregulation of DNA repair pathways results in genome instability. Because routinely used biological, physical and chemical agents impact human health, testing their genotoxicity and regulating their use have become important. In this introductory review, we will delineate mechanisms of DNA damage and the counteracting repair/tolerance pathways to provide insights into the molecular basis of genotoxicity in cells that lays the foundation for subsequent articles in this issue. Environ. Mol. Mutagen. 58:235-263, 2017. © 2017 Wiley Periodicals, Inc. read more read less

Topics:

DNA damage (59%)59% related to the paper, Nucleotide excision repair (59%)59% related to the paper, DNA repair (59%)59% related to the paper, DNA mismatch repair (57%)57% related to the paper, Genome instability (56%)56% related to the paper
954 Citations
Journal Article DOI: 10.1002/EM.2850130104
Identification of aneuploidy‐inducing agents using cytokinesis‐blocked human lymphocytes and an antikinetochore antibody
David A. Eastmond1, James D. Tucker1

Abstract:

The identification of agents causing aneuploidy in humans, a condition associated with carcinogenesis and birth defects, is currently limited due to the highly skilled and time-consuming nature of cytogenetic analyses. We report the development of a new simple and rapid assay to identify aneuploidy-inducing agents (aneuploido... The identification of agents causing aneuploidy in humans, a condition associated with carcinogenesis and birth defects, is currently limited due to the highly skilled and time-consuming nature of cytogenetic analyses. We report the development of a new simple and rapid assay to identify aneuploidy-inducing agents (aneuploidogens). The assay involves the chemical- or radiation-induced formation of micronuclei in cytokinesis-blocked human lymphocytes and the use of an antikinetochore antibody to determine whether the micronuclei contain centromeres--a condition indicating a high potential for aneuploidy. All agents tested produced dose-related increases in the frequency of micronucleated cells. The micronucleated cells induced by the known aneuploidogens--colchicine, vincristine sulfate, and diethylstilbestrol--contained kinetochore-positive micronuclei 92, 87, and 76% of the time, respectively. In contrast, the micronucleated cells induced by the potent clastogens--ionizing radiation and sodium arsenite--contained kinetochore-positive micronuclei only 3 and 19% of the time, respectively. These results indicate that this relatively simple assay can discriminate between aneuploidogens and clastogens and may allow a more rapid identification of environmental and therapeutic agents with aneuploidy-inducing potential. read more read less

Topics:

Aneugen (57%)57% related to the paper, Micronucleus test (57%)57% related to the paper
646 Citations
Comet assay in human biomonitoring studies: Reliability, validation, and applications

Abstract:

The comet assay (single-cell gel electrophoresis), which measures DNA strand breaks at the level of single cells, is very easily applied to human lymphocytes, and therefore lends itself to human biomonitoring studies. For the examination of DNA base oxidation (a specific marker of oxidative damage), the assay is modified by i... The comet assay (single-cell gel electrophoresis), which measures DNA strand breaks at the level of single cells, is very easily applied to human lymphocytes, and therefore lends itself to human biomonitoring studies. For the examination of DNA base oxidation (a specific marker of oxidative damage), the assay is modified by including a stage at which the DNA is incubated with a suitable lesion-specific endonuclease. Here we report on the reliability and reproducibility of this approach, from the level of comparing results from duplicate gels prepared from the same sample of cells, up to an assessment of the natural intra- and interindividual variability in lymphocyte DNA damage measured in groups of normal, healthy human volunteers. We applied the assay in investigations of human disease and occupational exposure of factory workers. read more read less

Topics:

Comet assay (62%)62% related to the paper, DNA damage (52%)52% related to the paper
635 Citations
Journal Article DOI: 10.1002/EM.20095
Carcinogenic polycyclic aromatic hydrocarbon-DNA adducts and mechanism of action.
William M. Baird1, Louisa A. Hooven1, Brinda Mahadevan1

Abstract:

Polycyclic aromatic hydrocarbons (PAHs) are a class of widespread environmental carcinogens. Most of our knowledge of their mechanisms of metabolic activation to DNA-binding "ultimate carcinogenic" metabolites has come from analysis of the DNA interaction products formed by these highly reactive intermediates. Studies of thei... Polycyclic aromatic hydrocarbons (PAHs) are a class of widespread environmental carcinogens. Most of our knowledge of their mechanisms of metabolic activation to DNA-binding "ultimate carcinogenic" metabolites has come from analysis of the DNA interaction products formed by these highly reactive intermediates. Studies of their role in forming DNA-binding intermediates identical to those formed in vivo from the PAH itself have also allowed identification of the particular cytochrome P450 enzymes involved in activating various structural classes of carcinogenic PAHs. It has been established that PAHs, after metabolic activation in vivo, are capable of inducing mutations in oncogenes and, by inducing multiple mutations, may result in tumors. PAHs also cause changes in cellular gap-junction communication similar to those caused by the tumor promoter 12-O-tetradecanoylphorbol-13-acetate. Thus, PAHs may also act through a promotional mechanism in addition to serving as tumor initiators. Previous studies on these mechanisms are described and summarized. read more read less

Topics:

Carcinogenic Polycyclic Aromatic Hydrocarbon (68%)68% related to the paper
596 Citations
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Frequently asked questions

1. Can I write Environmental and Molecular Mutagenesis in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Environmental and Molecular Mutagenesis guidelines and auto format it.

2. Do you follow the Environmental and Molecular Mutagenesis guidelines?

Yes, the template is compliant with the Environmental and Molecular Mutagenesis guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Environmental and Molecular Mutagenesis?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Environmental and Molecular Mutagenesis citation style.

4. Can I use the Environmental and Molecular Mutagenesis templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Environmental and Molecular Mutagenesis.

5. Can I use a manuscript in Environmental and Molecular Mutagenesis that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Environmental and Molecular Mutagenesis that you can download at the end.

6. How long does it usually take you to format my papers in Environmental and Molecular Mutagenesis?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Environmental and Molecular Mutagenesis.

7. Where can I find the template for the Environmental and Molecular Mutagenesis?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Environmental and Molecular Mutagenesis's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Environmental and Molecular Mutagenesis's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Environmental and Molecular Mutagenesis an online tool or is there a desktop version?

SciSpace's Environmental and Molecular Mutagenesis is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Environmental and Molecular Mutagenesis?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Environmental and Molecular Mutagenesis?”

11. What is the output that I would get after using Environmental and Molecular Mutagenesis?

After writing your paper autoformatting in Environmental and Molecular Mutagenesis, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Environmental and Molecular Mutagenesis's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Environmental and Molecular Mutagenesis?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Environmental and Molecular Mutagenesis. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Environmental and Molecular Mutagenesis?

The 5 most common citation types in order of usage for Environmental and Molecular Mutagenesis are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Environmental and Molecular Mutagenesis?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Environmental and Molecular Mutagenesis's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Environmental and Molecular Mutagenesis in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Environmental and Molecular Mutagenesis Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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