Alexandra Steckbauer

TL;DR: In this paper, the authors argue that ocean acidification from anthropogenic CO2 emissions is largely an open ocean syndrome and that a concept of anthro- pogenic impacts on marine pH, which is applicable across the entire ocean, from coastal to open-ocean environments, provides a superior framework to consider the multiple components of the anthropogenic perturbation of marine pH trajectories.

TL;DR: In this article, the authors observed diel pH changes in shallow (5-12 m) seagrass (Posidonia oceanica) meadows spanning 0.06 pH units in September to 0.24 units in June.

TL;DR: In this paper, the authors examined the recovery processes in a number of coastal areas managed for reducing nutrient inputs and thus, hypoxia (Northern Adriatic, Black Sea, Baltic Sea; Delaware Bay; and Danish Coastal Areas) reveals that recovery timescales following the return to normal oxygen conditions are much longer than those of loss following the onset of hypoxiosis, and typically involve decadal timesCales.

TL;DR: califying organisms have developed the capacity to alter the pH of their calcifying environment, or specifically within critical tissues where calcification occurs, thus achieving a homeostasis, and this capacity to control the conditions for calcification at the organism scale may buffer the full impacts of ocean acidification on an organism scale.

TL;DR: Oxygen depletion elicited significant and repeatable changes in general and species-specific reactions in virtually all organisms and most atypical (stress) behaviours were associated with specific oxygen thresholds: arm-tipping in the ophiuroid Ophiothrix quinquemaculata with the onset of mild hypoxia, for example.