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Andrej Tarkowski
Researcher at University of Gothenburg
Publications - 69
Citations - 5625
Andrej Tarkowski is an academic researcher from University of Gothenburg. The author has contributed to research in topics: Arthritis & Staphylococcus aureus. The author has an hindex of 37, co-authored 69 publications receiving 5272 citations. Previous affiliations of Andrej Tarkowski include Sahlgrenska University Hospital.
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Resistin, an adipokine with potent proinflammatory properties
TL;DR: The results indicate that resistin is a new and important member of the cytokine family with potent regulatory functions and makes it a novel and interesting therapeutic target in chronic inflammatory diseases such as rheumatoid arthritis.
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Discovery of endogenous catecholamines in lymphocytes and evidence for catecholamine regulation of lymphocyte function via an autocrine loop.
TL;DR: Results suggest the presence of an autocrine loop whereby lymphocytes down-regulate their own activity, and intracellular dopamine levels are shown to be increased by extra-cellular dopamine, suggesting a cellular-uptake mechanism.
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Catalase (KatA) and Alkyl Hydroperoxide Reductase (AhpC) Have Compensatory Roles in Peroxide Stress Resistance and Are Required for Survival, Persistence, and Nasal Colonization in Staphylococcus aureus
Kate Cosgrove,Graham Coutts,Ing-Marie Jonsson,Andrej Tarkowski,John F. Kokai-Kun,James J. Mond,Simon J. Foster +6 more
TL;DR: Oxidative-stress resistance is an important factor in the ability of S. aureus to persist in the hospital environment and so contribute to the spread of human disease.
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Host response to Bothrops asper snake venom. Analysis of edema formation, inflammatory cells, and cytokine release in a mouse model.
TL;DR: The present observations suggest that venom, besides its cytotoxic properties, induces early hematologic and immunologic alterations that may be of relevance in future treatment modalities.
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The Common Vaccine Adjuvant Aluminum Hydroxide Up-Regulates Accessory Properties of Human Monocytes via an Interleukin-4-Dependent Mechanism
TL;DR: The findings suggest that aluminum hydroxide directly stimulates monocytes to produce proinflammatory cytokines activating T cells, which can induce an increase in the expression of MHC class II molecules on monocytes, which leads to enhanced accessory functions of monocytes.