D
Donald D. Anderson
Researcher at University of Iowa
Publications - 148
Citations - 4357
Donald D. Anderson is an academic researcher from University of Iowa. The author has contributed to research in topics: Medicine & Ankle. The author has an hindex of 35, co-authored 120 publications receiving 3749 citations. Previous affiliations of Donald D. Anderson include University of Iowa Hospitals and Clinics & University of California, San Francisco.
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Journal ArticleDOI
Post-traumatic osteoarthritis: improved understanding and opportunities for early intervention
Donald D. Anderson,Susan Chubinskaya,Farshid Guilak,James A. Martin,Theodore R. Oegema,Steven A. Olson,Joseph A. Buckwalter,Joseph A. Buckwalter +7 more
TL;DR: Recent advances in understanding of the structural damage and the acute biological response following joint injury are highlighted, and important directions for future research are identified.
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LOGISMOS—Layered Optimal Graph Image Segmentation of Multiple Objects and Surfaces: Cartilage Segmentation in the Knee Joint
Yin Yin,Xiangmin Zhang,Rachel Marie-Crane Williams,Xiaodong Wu,Donald D. Anderson,Milan Sonka +5 more
TL;DR: The reported LOGISMOS framework provides robust and accurate segmentation of the knee joint bone and cartilage surfaces of the femur, tibia, and patella, and the developed framework can be applied to a broad range of multiobject multisurface segmentation problems.
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Insulin-Like Growth Factor I Accelerates Functional Recovery from Achilles Tendon Injury in a Rat Model
TL;DR: It is demonstrated that via a possible antiinflammatory mechanism, insulin-like growth factor I reduces maximum functional deficit and accelerates recovery after Achilles tendon injury.
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Contact stress aberrations following imprecise reduction of simple tibial plateau fractures.
Thomas D. Brown,Donald D. Anderson,James V. Nepola,Robert J. Singerman,Douglas R. Pedersen,Richard A. Brand +5 more
TL;DR: The present laboratory results suggest that nominally factor‐of‐two peak local pressure elevations, provided that they occur over only small portions of the cartilage surface, are probably within the long‐term overall tolerance range of an articular joint.
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The Roles of Mechanical Stresses in the Pathogenesis of Osteoarthritis: Implications for Treatment of Joint Injuries.
TL;DR: Advances in understanding of how altering mechanical stresses can lead to remodeling of osteoarthritic joints and how excessive stress causes loss of articular cartilage provide the basis for new biologic and mechanical approaches to the prevention and treatment of OA.