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Hassan M. Naif

Researcher at Nahrain University

Publications -  34
Citations -  2002

Hassan M. Naif is an academic researcher from Nahrain University. The author has contributed to research in topics: Viral replication & Monocyte. The author has an hindex of 26, co-authored 33 publications receiving 1935 citations. Previous affiliations of Hassan M. Naif include Westmead Hospital & National Institutes of Health.

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Journal ArticleDOI

HIV gp120 receptors on human dendritic cells.

TL;DR: A novel biotinylated gp120 assay was used to determine whether CLR or CD4 were predominant receptors on monocyte-derived DCs and ex vivo blood DCs, and confirmed that CLRs were the major receptors for gp120 on MDDCs.
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CCR5 Expression Correlates with Susceptibility of Maturing Monocytes to Human Immunodeficiency Virus Type 1 Infection

TL;DR: The concordance of levels of HIV DNA and p24 antigen suggested that the changes in susceptibility with monocyte maturation were at or immediately after entry and correlated well with CCR5 expression and inversely with CXCR4 expression.
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Persistent CCR5 Utilization and Enhanced Macrophage Tropism by Primary Blood Human Immunodeficiency Virus Type 1 Isolates from Advanced Stages of Disease and Comparison to Tissue-Derived Isolates

TL;DR: Results indicate that in the late stage of disease with increasing viral load and diversity, CCR5 utilization and M-tropism persist in blood and tissue and the replicative ability in macrophages increases.
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Pathogenesis of HIV infection

TL;DR: The interactions between viral phenotypes, tropism and co-receptor usage and how they influence HIV pathogenesis are the main themes addressed in this review.
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Molecular cloning and sequencing of an Australian isolate of proviral bovine leukaemia virus DNA: comparison with other isolates.

TL;DR: It is demonstrated that there are large tracts of amino acid sequence, particularly within the env and pol genes, that are highly conserved in different isolates that exist in regions containing epitopes important in virus infectivity.