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Henrik Zetterberg

Researcher at Sahlgrenska University Hospital

Publications -  2261
Citations -  104794

Henrik Zetterberg is an academic researcher from Sahlgrenska University Hospital. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 125, co-authored 1736 publications receiving 72452 citations. Previous affiliations of Henrik Zetterberg include University of Gothenburg & Luleå University of Technology.

Papers
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Cerebrospinal fluid and plasma biomarkers in Alzheimer disease.

TL;DR: The rationales behind and the diagnostic performances of the core cerebrospinal fluid biomarkers for AD, namely total tau, phosphorylated tau and the 42 amino acid form of amyloid-β are presented.
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Association between CSF biomarkers and incipient Alzheimer's disease in patients with mild cognitive impairment: a follow-up study

TL;DR: The association between pathological CSF and progression to Alzheimer's disease was much stronger than, and independent of, established risk factors including age, sex, education, APOE genotype, and plasma homocysteine.
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CSF and blood biomarkers for the diagnosis of Alzheimer's disease: a systematic review and meta-analysis

TL;DR: The core CSF biomarkers of neurodegeneration (T-tau, P-tAU, and Aβ42), CSF NFL, and plasma T-t Tau were strongly associated with Alzheimer's disease and the core biomarkers were strong associated with mild cognitive impairment due to Alzheimer's Disease.
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Defeating Alzheimer's disease and other dementias: a priority for European science and society

TL;DR: This poster aims to demonstrate the efforts towards in-situ applicability of EMMARM, which aims to provide real-time information about the physical and cognitive properties of Alzheimer's disease and other dementias.
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Gut microbiome alterations in Alzheimer's disease.

TL;DR: The gut microbiome of AD participants has decreased microbial diversity and is compositionally distinct from control age- and sex-matched individuals, which adds AD to the growing list of diseases associated with gut microbial alterations, as well as suggest that gut bacterial communities may be a target for therapeutic intervention.