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James L. Rudolph

Researcher at Brown University

Publications -  186
Citations -  8165

James L. Rudolph is an academic researcher from Brown University. The author has contributed to research in topics: Delirium & Medicine. The author has an hindex of 42, co-authored 149 publications receiving 6690 citations. Previous affiliations of James L. Rudolph include Harvard University & United States Department of Veterans Affairs.

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The anticholinergic risk scale and anticholinergic adverse effects in older persons.

TL;DR: Higher ARS scores are associated with statistically significantly increased risk of anticholinergic adverse effects in older patients, and these scores were associated with increased risk in the GEM cohort and in the primary care cohort.
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Orthogeriatric care models and outcomes in hip fracture patients: a systematic review and meta-analysis.

TL;DR: A meta-analysis found that orthogeriatric collaboration was associated with a significant reduction of in-hospital mortality and long-term mortality and that the best model of orthogueriatric collaboration and if these partnerships improve functional outcomes is needed.
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Derivation and validation of a preoperative prediction rule for delirium after cardiac surgery.

TL;DR: Using 4 preoperative characteristics, clinicians can determine cardiac surgery patients' risk for delirium using a prediction rule developed in a cohort and validated in an independent cohort.
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Postoperative Delirium: Acute Change with Long-Term Implications

TL;DR: Care improvements such as identifyingDelirium risk preoperatively; training surgeons, anesthesiologists, and nurses to screen for delirium; implementing deliriam prevention programs; and developing standardized deliria treatment protocols may reduce the risk of delirity and its associated morbidity.
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Delirium accelerates cognitive decline in Alzheimer disease

TL;DR: The information from this study provides the foundation for future randomized intervention studies to determine whether prevention of delirium might ameliorate or delay cognitive decline in patients with Alzheimer disease (AD).