J
Jean T. Greenberg
Researcher at University of Chicago
Publications - 88
Citations - 10770
Jean T. Greenberg is an academic researcher from University of Chicago. The author has contributed to research in topics: Arabidopsis & Pseudomonas syringae. The author has an hindex of 46, co-authored 83 publications receiving 10088 citations. Previous affiliations of Jean T. Greenberg include Harvard University & University of Colorado Boulder.
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Journal ArticleDOI
Priming in systemic plant immunity.
Ho Won Jung,Timothy J. Tschaplinski,Lin Wang,Lin Wang,Lin Wang,Jane Glazebrook,Jean T. Greenberg +6 more
TL;DR: Mutation of the AZELAIC ACID INDUCED 1 (AZI1) gene results in the specific loss of systemic immunity triggered by pathogen or azelaic acid and of the priming of SA induction in plants.
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The role and regulation of programmed cell death in plant–pathogen interactions
Jean T. Greenberg,Nan Yao +1 more
TL;DR: Recent progress is reviewed in determining the role and regulation of plant pcd responses that accompany both resistance and susceptible interactions and the mechanisms by which plant pCD occurs during these different interactions.
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Programmed cell death in plants : a pathogen-triggered response activated coordinately with multiple defense functions
Jean T. Greenberg,Allan Guo,Daniel F. Klessig,Daniel F. Klessig,Frederick M. Ausubel,Frederick M. Ausubel +5 more
TL;DR: In this paper, the authors describe mutant A. thaliana plants that contain lesions in a single accelerated cell death (ACD) gene called ACD2 and that bypass the need for pathogen exposure to induce the hypersensitive response (HR) to pathogens.
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Programmed cell death: a way of life for plants
TL;DR: In plants, PCD resembles either a common form of PCD seen in animals called apoptosis or it resembles a morphologically distinct form of cell death.
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Positive control of a global antioxidant defense regulon activated by superoxide-generating agents in Escherichia coli.
TL;DR: The identified genetic locus, soxR (superoxide response), that positively regulates 9 of these proteins during superoxide stress may operate as an inducible defense against xenobiotics in general.