Nan Yao

TL;DR: Recent progress is reviewed in determining the role and regulation of plant pcd responses that accompany both resistance and susceptible interactions and the mechanisms by which plant pCD occurs during these different interactions.

TL;DR: In this paper, a new mitochondrial gene, WA352, which originated recently in wild rice, confers the wild abortive male sterility (CMS-WA) because the protein it encodes interacts with the nuclear-encoded mitochondrial protein COX11.

TL;DR: The first ceramide kinase (CERK) mutant in any organism is characterized, called accelerated cell death 5, which accumulates CERK substrates and shows enhanced disease symptoms during pathogen attack and apoptotic-like cell death dependent on defense signaling late in development.

TL;DR: It is reported that a new mitochondrial gene, WA352, which originated recently in wild rice, confers CMS-WA because the protein it encodes interacts with the nuclear-encoded mitochondrial protein COX11, and can be suppressed by two restorer-of-fertility genes, suggesting the existence of different mechanisms to counteract deleterious cytoplasmic factors.

TL;DR: These results strongly suggest that chloroplast Hsp70 is targeted by the P. syringae HopI1 effector to promote bacterial virulence by suppressing plant defenses.