J
Jesse D. Schold
Researcher at Cleveland Clinic
Publications - 373
Citations - 14365
Jesse D. Schold is an academic researcher from Cleveland Clinic. The author has contributed to research in topics: Transplantation & Kidney transplantation. The author has an hindex of 54, co-authored 327 publications receiving 12115 citations. Previous affiliations of Jesse D. Schold include Wright State University & Cleveland Clinic Lerner College of Medicine.
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Journal ArticleDOI
Lack of Improvement in Renal Allograft Survival Despite a Marked Decrease in Acute Rejection Rates Over the Most Recent Era
TL;DR: This work analyzed data provided by the Scientific Registry of Transplant Recipients regarding all adult first renal transplants between 1995 and 2000 to investigate how acute rejection rates have evolved on a national level in the U.S and how this has impacted graft survival in the most recent era of kidney transplantation.
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Acute Kidney Injury Is Associated With Increased Long-Term Mortality After Cardiothoracic Surgery
Charles Hobson,Sinan Yavas,Mark S. Segal,Jesse D. Schold,Curtis G. Tribble,A. Joseph Layon,Azra Bihorac +6 more
TL;DR: The risk of death associated with AKI after cardiothoracic surgery remains high for 10 years regardless of other risk factors, even for those patients with complete renal recovery, so improved renal protection and closer postdischarge follow-up of renal function may be warranted.
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Long-term renal allograft survival: have we made significant progress or is it time to rethink our analytic and therapeutic strategies?
TL;DR: This work aimed, now with sufficient follow up available, to calculate real half‐lives of renal allograft survival improvement between 1988 and 1995 using projections of half-lives based on limited actual follow up.
Journal ArticleDOI
Long-term risk of mortality and acute kidney injury during hospitalization after major surgery.
Azra Bihorac,Sinan Yavas,Sophie Subbiah,Charles Hobson,Jesse D. Schold,Andrea Gabrielli,A. Joseph Layon,Mark S. Segal +7 more
TL;DR: In a large single-center cohort of patients discharged after major surgery, AKI with even small changes in sCr level during hospitalization was associated with an independent long-term risk of death.
Journal ArticleDOI
Excessive fructose intake induces the features of metabolic syndrome in healthy adult men: role of uric acid in the hypertensive response
Santos E. Perez-Pozo,Jesse D. Schold,Takahiko Nakagawa,Takahiko Nakagawa,Laura G. Sánchez-Lozada,Laura G. Sánchez-Lozada,Richard J. Johnson,Richard J. Johnson,J López Lillo +8 more
TL;DR: High doses of fructose raise the BP and cause the features of metabolic syndrome and lower the uric acid level prevents the increase in mean arterial blood pressure.