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José Manuel Rodríguez-Sánchez

Researcher at University of Cantabria

Publications -  54
Citations -  3503

José Manuel Rodríguez-Sánchez is an academic researcher from University of Cantabria. The author has contributed to research in topics: First episode & Schizophrenia. The author has an hindex of 29, co-authored 54 publications receiving 3097 citations.

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Construct validity of the Trail Making Test: Role of task-switching, working memory, inhibition/interference control, and visuomotor abilities

TL;DR: The results suggest that T MT-A requires mainly visuoperceptual abilities, TMT-B reflects primarily working memory and secondarily task-switching ability, while B-A minimizes visu operceptual and working memory demands, providing a relatively pure indicator of executive control abilities.
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Antipsychotic-induced weight gain in chronic and first-episode psychotic disorders: a systematic critical reappraisal.

TL;DR: Recent studies carried out in young patients with first-episode psychosis (FEP), along with methodological artefacts in studies of chronic populations, suggest that the magnitude of weight gain reported by much of the literature could be an underestimation of the true magnitude of this adverse effect.
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Trail Making Test in traumatic brain injury, schizophrenia, and normal ageing: Sample comparisons and normative data

TL;DR: Normal ageing impaired both direct and derived TMT indices, as revealed by lower scores in the healthy elderly group as compared with young (16-24) and middle-aged (25-54) healthy participants.
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White Matter Integrity and Cognitive Impairment in First-Episode Psychosis

TL;DR: Deficits in executive and motor functioning in patients with first-episode psychosis are associated with reductions in white matter integrity in the major fasciculi that connect the frontal and temporal cortices as well as in pathways connecting cortical and subcortical regions.
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Cognitive dysfunction in first-episode psychosis: the processing speed hypothesis

TL;DR: Speed of information processing may be considered a core cognitive deficit in schizophrenia and might be mediating a broader diversity of cognitive disturbances.