L
Lester J. Layfield
Researcher at University of Utah Hospital
Publications - 5
Citations - 548
Lester J. Layfield is an academic researcher from University of Utah Hospital. The author has contributed to research in topics: Cancer & Hypoxia-inducible factors. The author has an hindex of 5, co-authored 5 publications receiving 527 citations.
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Journal ArticleDOI
The National Cancer Institute Thyroid fine needle aspiration state of the science conference: a summation
Zubair W. Baloch,Edmund S. Cibas,Douglas P. Clark,Lester J. Layfield,Britt-Marie Ljung,Martha B. Pitman,Andrea Abati +6 more
TL;DR: The two-day "live" conference in October, attended by 154 registrants, gave the committees an in-depth opportunity to present their conclusions and debate controversial areas, and the establishment of an on-line educational cytopathology atlas.
Journal ArticleDOI
Consensus conference on second opinions in diagnostic anatomic pathology. Who, What, and When.
John E. Tomaszewski,Harry D. Bear,Julia A. Connally,Jonathan I. Epstein,Michael Feldman,Kathryn Foucar,Lester J. Layfield,Virginia A. LiVolsi,Ronald L. Sirota,Mark H. Stoler,Robin E. Stombler +10 more
TL;DR: A consensus conference on second Opinions in Diagnostic Anatomic Pathology: Who, What, and When John E. Tomaszewski (chair) and Harry D. Bear (chair), and Julia A. Bear, MD, PhD, FACS.
Journal ArticleDOI
Epidermal growth factor receptor gene amplification and protein expression in glioblastoma multiforme: prognostic significance and relationship to other prognostic factors.
TL;DR: The authors studied a series of 34 specimens with histologically proven GBM by immunohistochemistry for the presence of EGFR overexpression and by fluorescence in situ hybridization (FISH) for gene amplification of the EGFR gene to determine which variables were predictive of survival.
Journal ArticleDOI
Endolymphatic sac tumors in patients with and without von Hippel—Lindau disease: the role of genetic mutation, von Hippel—Lindau protein, and hypoxia inducible factor-1α expression
TL;DR: The sporadic and VHL disease-associated ELS tumors in this study had normal VHL-mediated HIF-1 regulation, a result ofnormal VHL gene expression in the case of the sporadic ELS tumor.
Book ChapterDOI
Metastatic Tumors and Lymphomas
TL;DR: With routine and special stains, distinction of metastatic carcinoma from a primary neoplasm of the thyroid is achievable, but, to assist with this, clinicians are expected to supply the history of malignancy on the requisition form.