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Rudy J. Castellani

Researcher at University of Maryland, Baltimore

Publications -  234
Citations -  13809

Rudy J. Castellani is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: Alzheimer's disease & Oxidative stress. The author has an hindex of 60, co-authored 229 publications receiving 12289 citations. Previous affiliations of Rudy J. Castellani include University of Yamanashi & Tohoku University.

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Consensus statement on concussion in sport—the 5th international conference on concussion in sport held in Berlin, October 2016

TL;DR: This document is developed for physicians and healthcare providers who are involved in athlete care, whether at a recreational, elite or professional level, and provides an overview of issues that may be of importance to healthcare providers involved in the management of SRC.
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Involvement of Oxidative Stress in Alzheimer Disease

TL;DR: A combination of dietary regimen of low total calorie and rich antioxidant nutrients and maintaining physical and intellectual activities may ultimately prove to be one of the most efficacious strategies for AD prevention.
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Oxidative stress and neurodegeneration

TL;DR: The findings support the idea that aldehyde‐ mediated modifications, in concert with oxyradical‐mediated modifications, are critical early pathogenic factors in Alzheimer's disease.
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Role of mitochondrial dysfunction in Alzheimer's disease.

TL;DR: The causes and consequences of mitochondrial disturbances in Alzheimer's disease as well as how this information might impact on therapeutic approaches to this disease are reviewed.
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Glycoxidation and oxidative stress in Parkinson disease and diffuse Lewy body disease

TL;DR: Immunolocalization of advanced glycation end-products and a marker of oxidative stress response induction provides evidence that glycoxidation and oxidative stress may be an important pathogenic factor in diseases characterized by Lewy body formation, and furthers the evidence that cytoskeletal proteins and their inclusions are susceptible to oxidative stress.