S
Sabine A. Eming
Researcher at University of Cologne
Publications - 157
Citations - 12421
Sabine A. Eming is an academic researcher from University of Cologne. The author has contributed to research in topics: Wound healing & Inflammation. The author has an hindex of 43, co-authored 141 publications receiving 9897 citations. Previous affiliations of Sabine A. Eming include Harvard University & Max Planck Society.
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Journal ArticleDOI
Wound repair and regeneration: Mechanisms, signaling, and translation
TL;DR: In this review, emerging concepts in tissue regeneration and repair are highlighted, and some perspectives on how to translate current knowledge into viable clinical approaches for treating patients with wound-healing pathologies are provided.
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Inflammation in wound repair: molecular and cellular mechanisms.
TL;DR: Cellular and molecular mechanisms controlling inflammation in cutaneous tissue repair are reviewed and a rationale for targeting the inflammatory phase in order to modulate the outcome of the healing response is provided.
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Differential Roles of Macrophages in Diverse Phases of Skin Repair
Tina Lucas,Ari Waisman,Rajeev Ranjan,Jürgen Roes,Thomas Krieg,Werner Müller,Axel Roers,Sabine A. Eming +7 more
TL;DR: The results demonstrate that macrophages exert distinct functions during the diverse phases of skin repair, which are crucial to control the natural sequence of repair events.
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Inflammation and metabolism in tissue repair and regeneration
TL;DR: The current understanding of the role of inflammation and cell metabolism in tissue-regenerative responses is reviewed, emerging concepts that may expand therapeutic perspectives are highlighted, and where important knowledge gaps remain are discussed.
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Engineering the Growth Factor Microenvironment with Fibronectin Domains to Promote Wound and Bone Tissue Healing
Mikaël M. Martino,Federico Tortelli,Mayumi Mochizuki,Stephanie Traub,Dror Ben-David,Gisela A. Kuhn,Ralph Müller,Erella Livne,Sabine A. Eming,Jeffrey A. Hubbell +9 more
TL;DR: Preclinical demonstrations in rodent models show promise for the use of the FN III9-10/12-14–modified matrices in humans to heal chronic wounds and repair bones, and shows potent synergistic signaling and morphogenesis between α5β1 integrin and the growth factor receptors.