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Saverio Sartore

Researcher at University of Padua

Publications -  131
Citations -  9139

Saverio Sartore is an academic researcher from University of Padua. The author has contributed to research in topics: Myosin & Myocyte. The author has an hindex of 53, co-authored 131 publications receiving 8941 citations. Previous affiliations of Saverio Sartore include National Research Council.

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Three myosin heavy chain isoforms in type 2 skeletal muscle fibres.

TL;DR: The existence of the 2X-MHC isoform was confirmed by immunoblotting analysis using muscles containing 2X fibres as a major component, such as the normal and hyperthyroid diaphragm, and the soleus muscle after high frequency chronic stimulation.
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Contribution of Adventitial Fibroblasts to Neointima Formation and Vascular Remodeling: From Innocent Bystander to Active Participant

TL;DR: It is hypothesize that during the response to vascular injuries in the adult, “activation” of adventitial fibroblasts is, at least in part, reminiscent of a developmental program that also invests, although with distinct spatiotemporal features, medial VSMCs.
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Number and Function of Endothelial Progenitor Cells as a Marker of Severity for Diabetic Vasculopathy

TL;DR: It is demonstrated that EPC decrease is related to PAD severity and that E PC function is altered in diabetic subjects with PAD, strengthening the pathogenetic role of EPC dysregulation in diabetic vasculopathy.
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Technical notes on endothelial progenitor cells: ways to escape from the knowledge plateau.

TL;DR: This review focuses on some aspects related to the two methods used to assess circulating EPCs: flow cytometry and cell culture, which uncover the many traps hidden in the choice of the right protocol, and suggest the best solutions on the basis of evidence and background theories.
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Diabetes impairs progenitor cell mobilisation after hindlimb ischaemia-reperfusion injury in rats.

TL;DR: Defective ischaemia-induced bone marrow mobilisation of EPCs impedes compensatory angiogenesis in ischaemic tissues of diabetic animals and growth factor administration together with blood glucose control may offer a rational therapeutic strategy for diabetic ischaems syndromes.