Example of BMJ Open Gastroenterology format
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Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format
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Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format Example of BMJ Open Gastroenterology format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

BMJ Open Gastroenterology — Template for authors

Categories Rank Trend in last 3 yrs
Gastroenterology #60 of 136 up up by 8 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 208 Published Papers | 789 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 05/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.8

3% from 2019

CiteRatio for BMJ Open Gastroenterology from 2016 - 2020
Year Value
2020 3.8
2019 3.9
2018 3.5
2017 2.9
graph view Graph view
table view Table view

1.24

29% from 2019

SJR for BMJ Open Gastroenterology from 2017 - 2020
Year Value
2020 1.24
2019 0.958
2018 1.206
2017 0.483
graph view Graph view
table view Table view

1.025

9% from 2019

SNIP for BMJ Open Gastroenterology from 2017 - 2020
Year Value
2020 1.025
2019 0.941
2018 0.756
2017 0.581
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 3% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 29% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 9% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

BMJ Open Gastroenterology

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BMJ Publishing Group

BMJ Open Gastroenterology

BMJ Open Gastroenterology is an online-only, peer-reviewed, Open Access gastroenterology journal, dedicated to publishing high-quality medical research from all disciplines and therapeutic areas of gastroenterology. The journal publishes all research study types, from study pr...... Read More

Medicine

i
Last updated on
04 Jun 2020
i
Acceptance Rate
75%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1136/BMJGAST-2017-000145
Intestinal microbiota is altered in patients with colon cancer and modified by probiotic intervention

Abstract:

Objective The colonic microbiota is altered in patients with colorectal cancer (CRC). We investigated the microbiota composition of patients with colon cancer compared with controls devoid of neoplastic or inflammatory disease and the potential to modify the colonic microbiota with probiotics. Design Biopsy samples were obtai... Objective The colonic microbiota is altered in patients with colorectal cancer (CRC). We investigated the microbiota composition of patients with colon cancer compared with controls devoid of neoplastic or inflammatory disease and the potential to modify the colonic microbiota with probiotics. Design Biopsy samples were obtained from the normal mucosa and tumour during colonoscopy from 15 patients with colon cancer. Subsequent patient-matched samples were taken at surgery from the tumour and nearby mucosa from the patients with cancer, eight of whom had received two daily tablets totalling 1.4×1010 CFUs Bifidobacterium lactis Bl-04 and 7×109 CFUs Lactobacillus acidophilus NCFM. Faecal samples were obtained after colonoscopy prior to starting the intervention and at surgery. In addition, 21 mucosal biopsies from non-cancer controls were obtained during colonoscopy followed by later faecal samples. The colonic and faecal microbiota was assessed by 16S rRNA gene amplicon sequencing. Results The tumour microbiota was characterised by increased microbial diversity and enrichment of several taxa including Fusobacterium, Selenomonas and Peptostreptococcus compared with the control microbiota. Patients with colon cancer that received probiotics had an increased abundance of butyrate-producing bacteria, especially Faecalibacterium and Clostridiales spp in the tumour, non-tumour mucosa and faecal microbiota. CRC-associated genera such as Fusobacterium and Peptostreptococcus tended to be reduced in the faecal microbiota of patients that received probiotics. Conclusions Patients with colon cancer harbour a distinct microbiota signature in the tumour tissue and nearby mucosa, which was altered with probiotic intervention. Our results show promise for potential therapeutic benefits in CRC by manipulation of the microbiota. Trial registration number NCT03072641; Results. read more read less

Topics:

Colorectal cancer (53%)53% related to the paper, Peptostreptococcus (51%)51% related to the paper
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236 Citations
open accessOpen access Journal Article DOI: 10.1136/BMJGAST-2017-000144
Alteration in the gastric microbiota and its restoration by probiotics in patients with functional dyspepsia.

Abstract:

Objective The objective of this study was to comparatively analyse the gastric fluid (GF) microbiota between patients with functional dyspepsia (FD) and healthy controls (HC), and to assess the effect of probiotics on the microbiota. Design Twenty-four Japanese patients with FD who met the Rome III definition and 21 age-match... Objective The objective of this study was to comparatively analyse the gastric fluid (GF) microbiota between patients with functional dyspepsia (FD) and healthy controls (HC), and to assess the effect of probiotics on the microbiota. Design Twenty-four Japanese patients with FD who met the Rome III definition and 21 age-matched and gender-matched HC volunteers were enrolled. The patients with FD had been treated with LG21, a probiotic strain. The GF was sampled after an overnight fast using a nasogastric tube. The bile acids concentration was determined by ELISA. The V3-V4 region of 16S rRNA gene was amplified using bacterial DNA from the GF, and then about 30 000 high-quality amplicons per sample were grouped into operational taxonomic units for analyses. Results The ratio of GF samples in which the bile acids were detectable was significantly greater in the FD than in the HC groups. In the bacterial composition analysis at the phylum level, the GF microbiota had a Bacteroidetes > Proteobacteria abundance and an absence of Acidobacteria in the FD group, in contrast, the GF microbiota had a Bacteroidetes Proteobacteria abundance and the presence of Acidobacteria in the HC group. Probiotic therapy in patients with FD shifted the composition of the GF microbiota to that observed in the HC volunteers. Conclusions Alteration in the GF microbiota was found in patients with FD compared with HC volunteers. Reflux of the small intestinal contents, including bile acid and intestinal bacteria, to the stomach was suggested to induce a bacterial composition change and be involved in the pathophysiology underlying FD. Probiotics appear effective in the treatment of FD through the normalisation of gastric microbiota. Trial registration number UMINCTR 000022026; Results. read more read less
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101 Citations
open accessOpen access Journal Article DOI: 10.1136/BMJGAST-2015-000075
Statins, antidiabetic medications and liver histology in patients with diabetes with non-alcoholic fatty liver disease.

Abstract:

Background Type-2 diabetes mellitus (T2DM) is a risk factor for progressive non-alcoholic fatty liver disease (NAFLD). Drugs commonly prescribed in patients with T2DM may affect liver histology by interfering with lipid metabolism and insulin resistance/secretion. Aim We studied if statins or antidiabetic agents were associat... Background Type-2 diabetes mellitus (T2DM) is a risk factor for progressive non-alcoholic fatty liver disease (NAFLD). Drugs commonly prescribed in patients with T2DM may affect liver histology by interfering with lipid metabolism and insulin resistance/secretion. Aim We studied if statins or antidiabetic agents were associated with non-alcoholic steatohepatitis (NASH) and significant fibrosis (SF). Methods We performed a cross-sectional study of 346 diabetics with biopsy-proven NAFLD. T2DM was defined as fasting glucose ≥7 mmol/L or glycated haemoglobin ≥6.5% and/or use of antidiabetics. NASH was defined according to the FLIP algorithm and SF as F2–4 Kleiner9s stages. Results 84% of patients were on antidiabetic therapy and 45% on statins. NASH and SF were present in 57% and 48% of patients. Statin-treated patients were older, more frequently male and with poorer glycaemic control despite more frequent antidiabetic therapy than those without statins; however, the prevalence of NASH (57%vs56%, p=0.868) and SF (48%vs48%, p=0.943) was not different between statin users and non-users. NASH was more common in patients on metformin or insulin than in those not treated with these drugs (60%vs47%, p=0.026; 68%vs53%, p=0.017). SF was more common in those treated with sulfonylureas (57%vs44%, p=0.030). Multivariate analyses confirmed that use of statins was independently and negatively associated with both NASH (OR (95% CI) 0.57 (0.32 to 1.01), p=0.055) and SF (OR (95% CI) 0.47 (0.26 to 0.84), p=0.011). Moreover, we found independent associations between insulin use and NASH (OR (95% CI) 2.24 (1.11 to 4.54), p=0.025) and sulfonylureas use and SF (OR (95% CI) 2.04 (1.11 to 3.74), p=0.022). Conclusions Several medications used in patients with diabetes are differently associated with NAFLD histology. Statin use is negatively associated, while insulin and sulfonylureas are positively associated with NASH and SF. A wider use of statins may be warranted in this high-risk population. read more read less

Topics:

Insulin resistance (53%)53% related to the paper, Fatty liver (53%)53% related to the paper, Statin (52%)52% related to the paper, Steatohepatitis (52%)52% related to the paper, Diabetes mellitus (52%)52% related to the paper
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92 Citations
open accessOpen access Journal Article DOI: 10.1136/BMJGAST-2017-000139
Efficacy of dietary and physical activity intervention in non-alcoholic fatty liver disease: a systematic review
Susan Kenneally1, Joanna H. Sier2, J. Bernadette Moore2, J. Bernadette Moore1

Abstract:

Background Non-alcoholic fatty liver disease (NAFLD) is the most common cause of chronic liver disease worldwide, with prevalence above 30% in many adult populations. Strongly associated with obesity, weight loss through diet and physical activity is the mainstay of its management. Weight loss can be difficult to achieve and ... Background Non-alcoholic fatty liver disease (NAFLD) is the most common cause of chronic liver disease worldwide, with prevalence above 30% in many adult populations. Strongly associated with obesity, weight loss through diet and physical activity is the mainstay of its management. Weight loss can be difficult to achieve and maintain however, and uncertainty exists as to which lifestyle changes are most effective. Objective The aim of this work was to systematically evaluate randomised controlled trials assessing diet, exercise or combination interventions aimed at reducing steatosis or markers of NAFLD activity. Design Medline, Scopus and Cochrane databases were searched from 1 January 1980 through to 31 July 2016, for intervention trials assessing the effects of diet, weight loss, exercise or any combination thereof, on NAFLD disease markers in human adults. Risk of publication bias and study quality was assessed using the American Dietetic Association Quality Criteria Checklist. Results From a total of... read more read less

Topics:

Fatty liver (55%)55% related to the paper, Weight loss (54%)54% related to the paper, Chronic liver disease (52%)52% related to the paper, Publication bias (51%)51% related to the paper
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89 Citations
open accessOpen access Journal Article DOI: 10.1136/BMJGAST-2017-000166
Mechanisms of liver disease in patients infected with HIV
Matthew B Kaspar1, Richard K. Sterling

Abstract:

Objective To describe the various mechanisms of liver disease in patients with HIV infection, and to link these mechanisms to disease states which may utilise them. Background Non-AIDS causes of morbidity and mortality are becoming increasingly common in patients chronically infected with HIV. In particular, liver-related dis... Objective To describe the various mechanisms of liver disease in patients with HIV infection, and to link these mechanisms to disease states which may utilise them. Background Non-AIDS causes of morbidity and mortality are becoming increasingly common in patients chronically infected with HIV. In particular, liver-related diseases have risen to become one of the leading causes of non-AIDS-related death. A thorough understanding of the mechanisms driving the development of liver disease in these patients is essential when evaluating and caring for these patients. Methods The literature regarding mechanisms of liver disease by which different disease entities may cause hepatic injury and fibrosis was reviewed and synthesised. Results A number of discrete mechanisms of injury were identified, to include: oxidative stress, mitochondrial injury, lipotoxicity, immune-mediated injury, cytotoxicity, toxic metabolite accumulation, gut microbial translocation, systemic inflammation, senescence and nodular regenerative hyperplasia. Disease states may use any number of these mechanisms to exert their effect on the liver. Conclusions The mechanisms by which liver injury may occur in patients with HIV infection are numerous. Most disease states use multiple mechanisms to cause hepatic injury and fibrosis. read more read less

Topics:

Liver disease (59%)59% related to the paper, Cirrhosis (53%)53% related to the paper, Disease (53%)53% related to the paper, Nodular regenerative hyperplasia (52%)52% related to the paper, Hepatitis (52%)52% related to the paper
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88 Citations
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BMJ Open Gastroenterology format uses unsrt citation style.

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Frequently asked questions

1. Can I write BMJ Open Gastroenterology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the BMJ Open Gastroenterology guidelines and auto format it.

2. Do you follow the BMJ Open Gastroenterology guidelines?

Yes, the template is compliant with the BMJ Open Gastroenterology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in BMJ Open Gastroenterology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the BMJ Open Gastroenterology citation style.

4. Can I use the BMJ Open Gastroenterology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for BMJ Open Gastroenterology.

5. Can I use a manuscript in BMJ Open Gastroenterology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper BMJ Open Gastroenterology that you can download at the end.

6. How long does it usually take you to format my papers in BMJ Open Gastroenterology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in BMJ Open Gastroenterology.

7. Where can I find the template for the BMJ Open Gastroenterology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per BMJ Open Gastroenterology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the BMJ Open Gastroenterology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. BMJ Open Gastroenterology an online tool or is there a desktop version?

SciSpace's BMJ Open Gastroenterology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like BMJ Open Gastroenterology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like BMJ Open Gastroenterology?”

11. What is the output that I would get after using BMJ Open Gastroenterology?

After writing your paper autoformatting in BMJ Open Gastroenterology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is BMJ Open Gastroenterology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for BMJ Open Gastroenterology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for BMJ Open Gastroenterology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In BMJ Open Gastroenterology?

The 5 most common citation types in order of usage for BMJ Open Gastroenterology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the BMJ Open Gastroenterology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per BMJ Open Gastroenterology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download BMJ Open Gastroenterology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in BMJ Open Gastroenterology Endnote style according to Elsevier guidelines.

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