Example of Cardiovascular Drugs and Therapy format
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Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format Example of Cardiovascular Drugs and Therapy format
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Cardiovascular Drugs and Therapy — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Pharmacology (medical) #35 of 246 up up by 27 ranks
Cardiology and Cardiovascular Medicine #56 of 317 up up by 21 ranks
Pharmacology #61 of 297 up up by 34 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 259 Published Papers | 1679 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 10/06/2020
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Related Journals

open access Open Access

SAGE

Quality:  
Good
CiteRatio: 4.3
SJR: 0.787
SNIP: 0.865
open access Open Access

Wiley

Quality:  
Good
CiteRatio: 4.2
SJR: 0.818
SNIP: 0.742
open access Open Access

SAGE

Quality:  
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CiteRatio: 6.1
SJR: 1.333
SNIP: 1.061
open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.8
SJR: 1.164
SNIP: 1.22

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.069

3% from 2018

Impact factor for Cardiovascular Drugs and Therapy from 2016 - 2019
Year Value
2019 4.069
2018 4.181
2017 2.771
2016 2.82
graph view Graph view
table view Table view

6.5

8% from 2019

CiteRatio for Cardiovascular Drugs and Therapy from 2016 - 2020
Year Value
2020 6.5
2019 6.0
2018 5.5
2017 5.0
2016 5.3
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 3% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 8% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.108

19% from 2019

SJR for Cardiovascular Drugs and Therapy from 2016 - 2020
Year Value
2020 1.108
2019 1.36
2018 1.338
2017 1.349
2016 1.067
graph view Graph view
table view Table view

1.077

7% from 2019

SNIP for Cardiovascular Drugs and Therapy from 2016 - 2020
Year Value
2020 1.077
2019 1.011
2018 0.864
2017 0.79
2016 0.831
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 19% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 7% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Cardiovascular Drugs and Therapy

Guideline source: View

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Springer

Cardiovascular Drugs and Therapy

Designed to objectively cover the world of cardiovascular pharmacology and therapy, and to bring you the information you need most in a timely and useful format, Cardiovascular Drugs and Therapy takes a fresh and energetic look at advances in this dynamic field. Homing in on t...... Read More

Medicine

i
Last updated on
09 Jun 2020
i
ISSN
0920-3206
i
Impact Factor
Medium - 0.803
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/S10557-005-5686-Z
Mild to moderate muscular symptoms with high-dosage statin therapy in hyperlipidemic patients--the PRIMO study.
Eric Bruckert, Gilles Hayem, Sylvie Dejager1, Caroline Yau1, Bernard Bégaud

Abstract:

Objectives: To characterize the risk factors, rate of occurrence, onset, nature and impact of mild to moderate muscular symptoms with high-dosage HMG-CoA reductase inhibitor (statin) therapy in general practice. Objectives: To characterize the risk factors, rate of occurrence, onset, nature and impact of mild to moderate muscular symptoms with high-dosage HMG-CoA reductase inhibitor (statin) therapy in general practice. read more read less

Topics:

Statin (50%)50% related to the paper
1,134 Citations
open accessOpen access Journal Article DOI: 10.1007/S10557-016-6711-0
Anthracycline Chemotherapy and Cardiotoxicity
John Mcgowan1, Robin Chung1, Angshuman Maulik1, Izabela Piotrowska1, J Malcolm Walker1, Derek M. Yellon1

Abstract:

Anthracycline chemotherapy maintains a prominent role in treating many forms of cancer. Cardiotoxic side effects limit their dosing and improved cancer outcomes expose the cancer survivor to increased cardiovascular morbidity and mortality. The basic mechanisms of cardiotoxicity may involve direct pathways for reactive oxygen... Anthracycline chemotherapy maintains a prominent role in treating many forms of cancer. Cardiotoxic side effects limit their dosing and improved cancer outcomes expose the cancer survivor to increased cardiovascular morbidity and mortality. The basic mechanisms of cardiotoxicity may involve direct pathways for reactive oxygen species generation and topoisomerase 2 as well as other indirect pathways. Cardioprotective treatments are few and those that have been examined include renin angiotensin system blockade, beta blockers, or the iron chelator dexrazoxane. New treatments exploiting the ErbB or other novel pro-survival pathways, such as conditioning, are on the cardioprotection horizon. Even in the forthcoming era of targeted cancer therapies, the substantial proportion of today's anthracycline-treated cancer patients may become tomorrow's cardiac patient. read more read less

Topics:

Cardiotoxicity (58%)58% related to the paper, Dexrazoxane (55%)55% related to the paper, Anthracycline (55%)55% related to the paper, Cancer (53%)53% related to the paper, Cardiovascular agent (53%)53% related to the paper
View PDF
596 Citations
Journal Article DOI: 10.1023/A:1007757724505
Randomized, double-blind, placebo-controlled trial of fish oil and mustard oil in patients with suspected acute myocardial infarction: the Indian experiment of infarct survival--4.

Abstract:

In a randomized, placebo-controlled trial, the effects of treatment with fish oil (eicosapentaenoic acid, 1.08 g/day) and mustard oil (alpha-linolenic acid, 2.9 g/day) were compared for 1 year in the management of 122 patients (fish oil, group A), 120 patients (mustard oil, group B), and 118 patients (placebo, group C) with s... In a randomized, placebo-controlled trial, the effects of treatment with fish oil (eicosapentaenoic acid, 1.08 g/day) and mustard oil (alpha-linolenic acid, 2.9 g/day) were compared for 1 year in the management of 122 patients (fish oil, group A), 120 patients (mustard oil, group B), and 118 patients (placebo, group C) with suspected acute myocardial infarction (AMI). Treatments were administered about (mean) 18 hours after the symptoms of AMI in all three groups. The extent of cardiac disease, rise in cardiac enzymes, and lipid peroxides were comparable among the groups at entry into the study. After 1 year total cardiac events were significantly less in the fish oil and mustard oil groups compared with the placebo group (24.5% and 28% vs. 34.7%, p < 0.01). Nonfatal infarctions were also significantly less in the fish oil and mustard oil groups compared with the placebo group (13.0% and 15.0% vs. 25.4%, p < 0.05). Total cardiac deaths showed no significant reduction in the mustard oil group; however, the fish oil group had significantly less cardiac deaths compared with the placebo group (11.4% vs. 22.0%, p < 0.05). Apart from the decrease in the cardiac event rate, the fish oil and mustard oil groups also showed a significant reduction in total cardiac arrhythmias, left ventricular enlargement, and angina pectoris compared with the placebo group. Reductions in blood lipoproteins in the two intervention groups were modest and do not appear to be the cause of the benefit in the two groups. Diene conjugates showed a significant reduction in the fish oil and mustard oil groups, indicating that a part of the benefit may be caused by the reduction in oxidative stress. The findings of this study suggest that fish oil and mustard oil, possibly due to the presence of n-3 fatty acids, may provide rapid protective effects in patients with AMI. However, a large study is necessary to confirm this suggestion. read more read less

Topics:

Fish oil (56%)56% related to the paper, Mustard Plant (54%)54% related to the paper, Eicosapentaenoic acid (52%)52% related to the paper, Diet therapy (51%)51% related to the paper, Placebo (51%)51% related to the paper
583 Citations
Journal Article DOI: 10.1007/BF00877747
Elastic properties and windkessel function of the human aorta

Abstract:

An understanding of the role of the aortic elastic properties indicates their relevance at several sites of cardiovascular function. Acting as an elastic buffering chamber behind the heart (the Windkessel function), the aorta and some of the proximal large vessels store about 50% of the left ventricular stroke volume during s... An understanding of the role of the aortic elastic properties indicates their relevance at several sites of cardiovascular function. Acting as an elastic buffering chamber behind the heart (the Windkessel function), the aorta and some of the proximal large vessels store about 50% of the left ventricular stroke volume during systole. In diastole, the elastic forces of the aortic wall forward this 50% of the volume to the peripheral circulation, thus creating a nearly continuous peripheral blood flow. This systolic-diastolic interplay represents theWindkessel function, which has an influence not only on the peripheral circulation but also on the heart, resulting in a reduction of left ventricular afterload and improvement in coronary blood flow and left ventricular relaxation. The elastic resistance (or stiffness), which the aorta sets against its systolic distention, increases with aging, with an increase in blood pressure, and with pathological changes such as atherosclerosis. This increased stiffness leads to an increase in systolic blood pressure and a decrease in diastolic blood pressure at any given mean pressure, an increase in systolic blood velocity, an increase in left ventricular afterload, and a decrease in subendocardial blood supply during diastole, and must be considered a major pathophysiological factor, for example, in systolic hypertension. The elastic properties of the aortic Windkessel can be assessed in vivo in humans in several ways, most easily by measuring the pulse wave velocity along the aorta. The higher this velocity, the higher the elastic resistance, that is, the stiffness. Other methods depend on assessment of the ratio between pulse pressure and aortic volume changes (ΔP/ΔV), which can be assessed noninvasively by ultrasonic or tomographic methods. All assessments of vessel stiffness have to take into account the direct effect of current blood pressure, and thus judgements about influences of interventions rely on an unchanged blood pressure. Alternatively, to derive the “intrinsic” stiffness of the aortic wall one has to correct for the effect of the blood pressure present. Recently reports about pharmacologic influences on the elastic properties of the aorta have emerged in the literature. Angiotensin-converting enzyme (ACE) inhibitors and nitric oxide (NO) donors seem to directly reduce the elastic resistance of the aorta. This effect, in addition to other effects on blood pressure and the peripheral circulation, could have major clinical relevance as an additional mechanism for unloading the left ventricle, improving coronary circulation, and reducing the pulsatile stress of the arterial system. read more read less

Topics:

Afterload (67%)67% related to the paper, Ventricular pressure (64%)64% related to the paper, Pulse pressure (64%)64% related to the paper, Blood flow (60%)60% related to the paper, Aorta (60%)60% related to the paper
View PDF
383 Citations
Journal Article DOI: 10.1023/A:1011119003788
The EPHESUS trial: Eplerenone in patients with heart failure due to systolic dysfunction complicating acute myocardial infarction

Abstract:

The importance of aldosterone in the pathophysiology of chronic heart failure (HF) has been established in previous studies [1–4] and is emphasized by the findings of the RALES trial [5]. In this study, aldosterone blockade with spironolactone resulted in a 30% reduction in total mortality and a 35% reduction in hospitalizati... The importance of aldosterone in the pathophysiology of chronic heart failure (HF) has been established in previous studies [1–4] and is emphasized by the findings of the RALES trial [5]. In this study, aldosterone blockade with spironolactone resulted in a 30% reduction in total mortality and a 35% reduction in hospitalizations for HF in patients with pre-existing chronic severe HF. Patients in the RALES trial also received standard therapy including an ACE inhibitor (if tolerated), a loop diuretic, and digoxin. While aldosterone receptor blockade has been proven beneficial in severe chronic HF due to systolic left ventricular (LV) dysfunction, its effects in patients with acute myocardial infarction (AMI) complicated by HF due to systolic left ventricular dysfunction are unknown. The pathophysiology of HF complicating AMI is complex. Factors such as the acute release of catecholamines, activation of the renin angiotensin aldosterone system, degree of ventricular remodeling, myocardial scar formation, extent of coronary artery disease, and residual ischemia may differ both quantitatively and qualitatively in patients with acute infarction compared to patients with chronic HF. Additionally, the extent of activation of cytokines, fibrinolytic balance, and activity of clotting factors may differ. Eplerenone was chosen for this study because of its demonstrated efficacy in an experimental model of AMI [6]. In clinical trials, eplerenone demonstrated efficacy similar to spironolactone in blocking aldosterone receptors, lowering blood pressure, and moderating hormonal and neurohormonal markers of HF [7,8]. However, eplerenone has significantly less affinity for androgen and progesterone receptors and should therefore be associated with a lower incidence of gynecomastia, breast pain and impotency in males, and diminished libido and menstrual irregularities in females [9–11]. While older patients suffering from refractory HF may tolerate these androgenic and progestational side effects, they may preclude widespread use of a nonspecific aldosterone antagonist in younger patients or in patients with less severe cardiac compromise. Since eplerenone is at least 100 times more specific in its affinity for aldosterone receptors than is spironolactone, if the hypothesis being tested in the EPHESUS trial proves correct, eplerenone has the potential to be used in a broad population to prevent progressive left ventricular remodeling, ventricular fibrosis, malignant arrhythmias, non-fatal AMI, and sudden cardiac death. We hypothesize that selective aldosterone receptor blockade with eplerenone will have a beneficial effect on survival and morbidity in patients with AMI complicated by HF due to systolic left ventricular dysfunction. This paper describes the background, design, and organization of a trial to test this hypothesis—the EPHESUS trial (Eplerenone Post-AMI Heart Failure Efficacy and Survival Study). read more read less

Topics:

Eplerenone (60%)60% related to the paper, Myocardial infarction complications (59%)59% related to the paper, Heart failure (56%)56% related to the paper, Myocardial infarction (55%)55% related to the paper, Heart disease (54%)54% related to the paper
349 Citations
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Cardiovascular Drugs and Therapy format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Cardiovascular Drugs and Therapy in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cardiovascular Drugs and Therapy guidelines and auto format it.

2. Do you follow the Cardiovascular Drugs and Therapy guidelines?

Yes, the template is compliant with the Cardiovascular Drugs and Therapy guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cardiovascular Drugs and Therapy?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cardiovascular Drugs and Therapy citation style.

4. Can I use the Cardiovascular Drugs and Therapy templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cardiovascular Drugs and Therapy.

5. Can I use a manuscript in Cardiovascular Drugs and Therapy that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cardiovascular Drugs and Therapy that you can download at the end.

6. How long does it usually take you to format my papers in Cardiovascular Drugs and Therapy?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cardiovascular Drugs and Therapy.

7. Where can I find the template for the Cardiovascular Drugs and Therapy?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cardiovascular Drugs and Therapy's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cardiovascular Drugs and Therapy's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Cardiovascular Drugs and Therapy an online tool or is there a desktop version?

SciSpace's Cardiovascular Drugs and Therapy is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cardiovascular Drugs and Therapy?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Cardiovascular Drugs and Therapy?”

11. What is the output that I would get after using Cardiovascular Drugs and Therapy?

After writing your paper autoformatting in Cardiovascular Drugs and Therapy, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cardiovascular Drugs and Therapy's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cardiovascular Drugs and Therapy?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cardiovascular Drugs and Therapy. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cardiovascular Drugs and Therapy?

The 5 most common citation types in order of usage for Cardiovascular Drugs and Therapy are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cardiovascular Drugs and Therapy?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cardiovascular Drugs and Therapy's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cardiovascular Drugs and Therapy in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cardiovascular Drugs and Therapy Endnote style according to Elsevier guidelines.

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