Example of Journal of Clinical Periodontology format
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Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format
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Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format Example of Journal of Clinical Periodontology format
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open access Open Access

Journal of Clinical Periodontology — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Periodontics #2 of 12 -
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 620 Published Papers | 6608 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 02/06/2020
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Related Journals

open access Open Access
recommended Recommended

Wiley

Quality:  
High
CiteRatio: 15.0
SJR: 3.725
SNIP: 4.092
open access Open Access

Medknow

Quality:  
Good
CiteRatio: 2.3
SJR: 0.289
SNIP: 1.104

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.241

26% from 2018

Impact factor for Journal of Clinical Periodontology from 2016 - 2019
Year Value
2019 5.241
2018 4.164
2017 4.046
2016 3.477
graph view Graph view
table view Table view

10.7

45% from 2019

CiteRatio for Journal of Clinical Periodontology from 2016 - 2020
Year Value
2020 10.7
2019 7.4
2018 7.1
2017 6.9
2016 8.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 26% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 45% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.456

40% from 2019

SJR for Journal of Clinical Periodontology from 2016 - 2020
Year Value
2020 3.456
2019 2.475
2018 2.581
2017 2.079
2016 2.484
graph view Graph view
table view Table view

3.236

28% from 2019

SNIP for Journal of Clinical Periodontology from 2016 - 2020
Year Value
2020 3.236
2019 2.527
2018 2.173
2017 1.968
2016 2.049
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 40% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 28% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Journal of Clinical Periodontology

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Wiley

Journal of Clinical Periodontology

The aim of the Journal of Clinical Periodontology is to provide the platform for exchange of scientific and clinical progress in the field of Periodontology and allied disciplines, and to do so at the highest possible level. The Journal publishes original contributions of high...... Read More

Periodontics

Dentistry

i
Last updated on
02 Jun 2020
i
ISSN
0303-6979
i
Impact Factor
High - 1.688
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
apa
i
Citation Type
Numbered
[25]
i
Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1111/J.1600-051X.1998.TB02419.X
Microbial complexes in subgingival plaque

Abstract:

It has been recognized for some time that bacterial species exist in complexes in subgingival plaque. The purpose of the present investigation was to attempt to define such communities using data from large numbers of plaque samples and different clustering and ordination techniques. Subgingival plaque samples were taken from... It has been recognized for some time that bacterial species exist in complexes in subgingival plaque. The purpose of the present investigation was to attempt to define such communities using data from large numbers of plaque samples and different clustering and ordination techniques. Subgingival plaque samples were taken from the mesial aspect of each tooth in 185 subjects (mean age 51 +/- 16 years) with (n = 160) or without (n = 25) periodontitis. The presence and levels of 40 subgingival taxa were determined in 13,261 plaque samples using whole genomic DNA probes and checkerboard DNA-DNA hybridization. Clinical assessments were made at 6 sites per tooth at each visit. Similarities between pairs of species were computed using phi coefficients and species clustered using an averaged unweighted linkage sort. Community ordination was performed using principal components analysis and correspondence analysis. 5 major complexes were consistently observed using any of the analytical methods. One complex consisted of the tightly related group: Bacteroides forsythus, Porphyromonas gingivalis and Treponema denticola. The 2nd complex consisted of a tightly related core group including members of the Fusobacterium nucleatum/periodonticum subspecies, Prevotella intermedia, Prevotella nigrescens and Peptostreptococcus micros. Species associated with this group included: Eubacterium nodatum, Campylobacter rectus, Campylobacter showae, Streptococcus constellatus and Campylobacter gracilis. The 3rd complex consisted of Streptococcus sanguis, S. oralis, S. mitis, S. gordonii and S. intermedius. The 4th complex was comprised of 3 Capnocytophaga species, Campylobacter concisus, Eikenella corrodens and Actinobacillus actinomycetemcomitans serotype a. The 5th complex consisted of Veillonella parvula and Actinomyces odontolyticus. A. actinomycetemcomitans serotype b, Selenomonas noxia and Actinomyces naeslundii genospecies 2 (A. viscosus) were outliers with little relation to each other and the 5 major complexes. The 1st complex related strikingly to clinical measures of periodontal disease particularly pocket depth and bleeding on probing. read more read less

Topics:

Prevotella nigrescens (55%)55% related to the paper, Campylobacter rectus (55%)55% related to the paper, Campylobacter showae (54%)54% related to the paper, Eubacterium nodatum (54%)54% related to the paper, Dental plaque (52%)52% related to the paper
4,143 Citations
Journal Article DOI: 10.1111/J.1600-051X.2005.00642.X
Dimensional ridge alterations following tooth extraction. An experimental study in the dog
Mauricio G. Araújo1, Mauricio G. Araújo2, Jan Lindhe2

Abstract:

Objective: To study dimensional alterations of the alveolar ridge that occurred following tooth extraction as well as processes of bone modelling and remodelling associated with such change. Material and Methods: Twelve mongrel dogs were included in the study. In both quadrants of the mandible incisions were made in the cr... Objective: To study dimensional alterations of the alveolar ridge that occurred following tooth extraction as well as processes of bone modelling and remodelling associated with such change. Material and Methods: Twelve mongrel dogs were included in the study. In both quadrants of the mandible incisions were made in the crevice region of the 3rd and 4th premolars. Minute buccal and lingual full thickness flaps were elevated. The four premolars were hemi-sected. The distal roots were removed. The extraction sites were covered with the mobilized gingival tissue. The extractions of the roots and the sacrifice of the dogs were staggered in such a manner that all dogs contributed with sockets representing 1, 2, 4 and 8 weeks of healing. The animals were sacrificed and tissue blocks containing the extraction socket were dissected, decalcified in EDTA, embedded in paraffin and cut in the buccal–lingual plane. The sections were stained in haematoxyline–eosine and examined in the microscope. Results: It was demonstrated that marked dimensional alterations occurred during the first 8 weeks following the extraction of mandibular premolars. Thus, in this interval there was a marked osteoclastic activity resulting in resorption of the crestal region of both the buccal and the lingual bone wall. The reduction of the height of the walls was more pronounced at the buccal than at the lingual aspect of the extraction socket. The height reduction was accompanied by a “horizontal” bone loss that was caused by osteoclasts present in lacunae on the surface of both the buccal and the lingual bone wall. Conclusions: The resorption of the buccal/lingual walls of the extraction site occurred in two overlapping phases. During phase 1, the bundle bone was resorbed and replaced with woven bone. Since the crest of the buccal bone wall was comprised solely of bundle this modelling resulted in substantial vertical reduction of the buccal crest. Phase 2 included resorption that occurred from the outer surfaces of both bone walls. The reason for this additional bone loss is presently not understood. read more read less

Topics:

Bundle bone (60%)60% related to the paper, Socket preservation (60%)60% related to the paper, Bone regeneration (58%)58% related to the paper, Alveolar process (55%)55% related to the paper, Dental alveolus (55%)55% related to the paper
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1,308 Citations
Journal Article DOI: 10.1111/J.1600-051X.1997.TB01187.X
The interleukin-1 genotype as a severity factor in adult periodontal disease

Abstract:

Although specific bacteria, dental plaque, and age are associated with periodontal disease, there are currently no reliable predictors of periodontitis severity. Studies in twins have suggested a genetic contribution to the pathogenesis of periodontitis, but previous attempts to identify genetic markers have been unsuccessful... Although specific bacteria, dental plaque, and age are associated with periodontal disease, there are currently no reliable predictors of periodontitis severity. Studies in twins have suggested a genetic contribution to the pathogenesis of periodontitis, but previous attempts to identify genetic markers have been unsuccessful. The pro-inflammatory cytokines interleukin-1 (IL-1) and tumor necrosis factor alpha (TNF alpha) are key regulators of the host responses to microbial infection. IL-1 is also a major modulator of extracellular matrix catabolism and bone resorption. We report a specific genotype of the polymorphic IL-1 gene cluster that was associated with severity of periodontitis in non-smokers, and distinguished individuals with severe periodontitis from those with mild disease (odds ratio 18.9 for ages 40-60 years). Functionally, the specific periodontitis-associated IL-1 genotype comprises a variant in the IL-1B gene that is associated with high levels of IL-1 production. In smokers severe disease was not correlated with genotype. In this study, 86.0% of the severe periodontitis patients were accounted for by either smoking or the IL-1 genotype. This study demonstrates that specific genetic markers, that have been associated with increased IL-1 production, are a strong indicator of susceptibility to severe periodontitis in adults. read more read less

Topics:

Severe periodontitis (71%)71% related to the paper, Periodontitis (61%)61% related to the paper, Genotype (55%)55% related to the paper, Dental plaque (51%)51% related to the paper
1,194 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1600-051X.2008.01283.X
Peri-implant diseases: Consensus Report of the Sixth European Workshop on Periodontology
Jan Lindhe1, Joerg Meyle2

Abstract:

Issues related to peri-implant disease were discussed. It was observed that the most common lesions that occur, i.e. peri-implant mucositis and peri-implantitis are caused by bacteria. While the lesion of peri-implant mucositis resides in the soft tissues, peri-implantitis also affects the supporting bone. Peri-implant mucosi... Issues related to peri-implant disease were discussed. It was observed that the most common lesions that occur, i.e. peri-implant mucositis and peri-implantitis are caused by bacteria. While the lesion of peri-implant mucositis resides in the soft tissues, peri-implantitis also affects the supporting bone. Peri-implant mucositis occurs in about 80% of subjects (50% of sites) restored with implants, and peri-implantitis in between 28% and 56% of subjects (12-40% of sites). A number of risk indicators were identified including (i) poor oral hygiene, (ii) a history of periodontitis, (iii) diabetes and (iv) smoking. It was concluded that the treatment of peri-implant disease must include anti-infective measures. With respect to peri-implant mucositis, it appeared that non-surgical mechanical therapy caused the reduction in inflammation (bleeding on probing) but also that the adjunctive use of antimicrobial mouthrinses had a positive effect. It was agreed that the outcome of non-surgical treatment of peri-implantitis was unpredictable. The primary objective of surgical treatment in peri-implantitis is to get access to the implant surface for debridement and decontamination in order to achieve resolution of the inflammatory lesion. There was limited evidence that such treatment with the adjunctive use of systemic antibiotics could resolve a number of peri-implantitis lesions. There was no evidence that so-called regenerative procedures had additional beneficial effects on treatment outcome. read more read less

Topics:

Peri-implant mucositis (67%)67% related to the paper, Mucositis (65%)65% related to the paper, Periodontitis (52%)52% related to the paper, Peri-implantitis (51%)51% related to the paper, Bleeding on probing (51%)51% related to the paper
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1,185 Citations
Journal Article DOI: 10.1111/J.1600-051X.1982.TB02095.X
New attachment following surgical treatment of human periodontal disease.
Sture Nyman1, Jan Lindhe1, Thorkild Karring, Harald Rylander1

Abstract:

The present experiment was undertaken to test the hypothesis that new connective tissue attachment may form on a previously periodontitis involved root surface provided cells originating from the periodontal ligament are enabled to repopulate the root surface during healing. A mandibular incisor with advanced periodontal dise... The present experiment was undertaken to test the hypothesis that new connective tissue attachment may form on a previously periodontitis involved root surface provided cells originating from the periodontal ligament are enabled to repopulate the root surface during healing. A mandibular incisor with advanced periodontal disease of long standing (the distance between the cemento-enamel junction and the alveolar bone crest was 9 mm) was subjected to periodontal surgery using a technique which during healing prevented the dentogingival epithelium and the gingival connective tissue from reaching contact with the curetted root surface. Preference was hereby given to the periodontal ligament cells to repopulate the previously diseased root surface. After 3 months of healing a block biopsy containing the incisor and surrounding tissue was sampled. The histological analysis revealed that new cementum with inserting principal fibers had formed on the previously diseased root surface. This new attachment extended in coronal direction to a level 5 mm coronal to the alveolar bone crest. This finding suggests that new attachment can be achieved by cells originating from the periodontal ligament and demonstrates that the concept that the periodontitis affected root surface is a major preventive factor for new attachment is invalid. read more read less

Topics:

Clinical attachment loss (66%)66% related to the paper, Periodontal fiber (65%)65% related to the paper, Cementum (62%)62% related to the paper, Cementogenesis (60%)60% related to the paper, Dental alveolus (55%)55% related to the paper
1,040 Citations
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Journal of Clinical Periodontology format uses apa citation style.

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Frequently asked questions

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Yes, the template is compliant with the Journal of Clinical Periodontology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Clinical Periodontology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Clinical Periodontology citation style.

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12. Is Journal of Clinical Periodontology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Clinical Periodontology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Clinical Periodontology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Clinical Periodontology?

The 5 most common citation types in order of usage for Journal of Clinical Periodontology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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16. Can I download Journal of Clinical Periodontology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Clinical Periodontology Endnote style according to Elsevier guidelines.

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