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Ayanthi A. Richards
Researcher at University of Queensland
Publications - 16
Citations - 1999
Ayanthi A. Richards is an academic researcher from University of Queensland. The author has contributed to research in topics: Adiponectin & Adipokine. The author has an hindex of 14, co-authored 16 publications receiving 1913 citations. Previous affiliations of Ayanthi A. Richards include Princess Alexandra Hospital.
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Journal ArticleDOI
Lipid rafts and caveolae as portals for endocytosis: new insights and common mechanisms.
TL;DR: This review examines the current evidence for the involvement of rafts and caveolae in endocytosis and the molecular players involved in their regulation.
Journal ArticleDOI
Adiponectin--a key adipokine in the metabolic syndrome.
Jonathan P. Whitehead,Ayanthi A. Richards,Ingrid J. Hickman,Graeme A. Macdonald,Johannes B. Prins +4 more
TL;DR: Given the low levels of adiponectin in subjects with the metabolic syndrome, and the beneficial effect of the adipokine in animal studies, there is exciting potential for adiponECTin replacement therapy in insulin resistance and related disorders.
Journal ArticleDOI
Adiponectin Multimerization Is Dependent on Conserved Lysines in the Collagenous Domain: Evidence for Regulation of Multimerization by Alterations in Posttranslational Modifications
Ayanthi A. Richards,Tim Stephens,Hayley K. Charlton,Alun Jones,Graeme A. Macdonald,Johannes B. Prins,Jonathan P. Whitehead +6 more
TL;DR: Gl glucose-induced increases in HMW multimer production from human adipose explants correlated with changes in the two-dimensional electrophoresis profile of adiponectin isoforms, suggesting that adiponECTin multimer composition is affected by changes in PTM in response to physiological factors.
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Acute lipopolysaccharide priming boosts inflammasome activation independently of inflammasome sensor induction.
Kate Schroder,Vitaliya Sagulenko,Alina Zamoshnikova,Ayanthi A. Richards,Jasmyn A. Cridland,Katharine M. Irvine,Katryn J. Stacey,Matthew J. Sweet +7 more
TL;DR: It is demonstrated that rapid LPS priming ofNLRP3 inflammasome activation can occur independently of NLRP3 induction, since the priming effect of LPS is still apparent at short pre-treatment times in which NLRP2 protein expression remains unchanged.
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Inhibitors of COP-mediated Transport and Cholera Toxin Action Inhibit Simian Virus 40 Infection
TL;DR: It is shown that SV40 requires COPI- and COPII-dependent transport steps for successful infection and a potent inhibition of SV40 infection by the dipeptide N-benzoyl-oxycarbonyl-Gly-Phe-amide, which also inhibits late events in cholera toxin action is demonstrated.