E
Elliot D. Rosenstein
Researcher at Overlook Medical Center
Publications - 67
Citations - 2117
Elliot D. Rosenstein is an academic researcher from Overlook Medical Center. The author has contributed to research in topics: Rheumatoid arthritis & Arthritis. The author has an hindex of 21, co-authored 65 publications receiving 2016 citations. Previous affiliations of Elliot D. Rosenstein include Beth Israel Deaconess Medical Center & George Washington University.
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Adenosine; a physiologic modulator of superoxide anion generation by human neutrophils. Adenosine acts via an A2 receptor on human neutrophils.
TL;DR: Adenosine, derived from damaged tissues, may serve as a specific, endogenous modulator of superoxide anion generation by activated neutrophil through interaction at this newly described receptor on human neutrophils.
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Hypothesis: The Humoral Immune Response to Oral Bacteria Provides a Stimulus for the Development of Rheumatoid Arthritis
TL;DR: It is suggested that individuals predisposed to periodontal infection are exposed to antigens generated by PAD, with deiminated fibrin as a likely candidate, which become systemic immunogens and lead to intraarticular inflammation.
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Occupancy of adenosine receptors raises cyclic AMP alone and in synergy with occupancy of chemoattractant receptors and inhibits membrane depolarization.
Bruce N. Cronstein,Sara B. Kramer,Elliot D. Rosenstein,Helen M. Korchak,Gerald Weissmann,Rochelle Hirschhorn +5 more
TL;DR: Adenosine occupies an A2 receptor on neutrophils to raise intracellular cAMP in synergy with occupancy of the FMLP receptor, and indicates that cAMP is not the second messenger for inhibition of O2- generation by adenosine and its analogues.
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Pilot study of dietary fatty acid supplementation in the treatment of adult periodontitis.
TL;DR: In this paper, the anti-inflammatory effects of both n-3 and n-6 polyunsaturated fatty acids (PUFA) have been demonstrated in vitro and in many disease states, in particular in the treatment of rheumatoid arthritis.
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Giant Cell Myocarditis: Most Fatal of Autoimmune Diseases
TL;DR: The clinical and immunopathogenetic similarities with classical rheumatologic diseases, the differential diagnosis with sarcoidosis and other inflammatory conditions, and the use of standard immunosuppressive medications make GCM a disease process that should be added to the rheumologist's expertise.