R
Robert A. Greenwald
Researcher at Long Island Jewish Medical Center
Publications - 59
Citations - 12638
Robert A. Greenwald is an academic researcher from Long Island Jewish Medical Center. The author has contributed to research in topics: Matrix metalloproteinase & Arthritis. The author has an hindex of 28, co-authored 59 publications receiving 11870 citations. Previous affiliations of Robert A. Greenwald include North Shore-LIJ Health System.
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Journal ArticleDOI
Development of criteria for the classification and reporting of osteoarthritis: Classification of osteoarthritis of the knee
Roy D. Altman,E. Asch,Daniel Bloch,Giles G. Bole,David G. Borenstein,Kenneth D. Brandt,Wallace C. Christy,Cooke Td,Robert A. Greenwald,Marc C. Hochberg,David S. Howell,David L. Kaplan,William J. Koopman,Selden Longley,Henry J. Mankin,Dennis J. McShane,Thomas A. Medsger,Robert F. Meenan,William M. Mikkelsen,Roland W. Moskowitz,William A. Murphy,B. Rothschild,Mark R. Segal,Leon Sokoloff,Frederick Wolfe +24 more
TL;DR: Variables from the medical history, physical examination, laboratory tests, and radiographs were used to develop sets of criteria that serve different investigative purposes and these proposed criteria utilize classification trees, or algorithms.
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The American College of Rheumatology criteria for the classification and reporting of osteoarthritis of the hip.
Roy D. Altman,Graciela S. Alarcón,D. Appelrouth,Daniel Bloch,David G. Borenstein,Kenneth D. Brandt,C. Brown,Cooke Td,W W Daniel,D. Feldman,Robert A. Greenwald,Marc C. Hochberg,David S. Howell,Robert W. Ike,P. Kapila,David L. Kaplan,William J. Koopman,Catherine Marino,Eric V. McDonald,Dennis J. McShane,Thomas A. Medsger,B. A. Michel,William A. Murphy,T. Osial,Rosalind Ramsey-Goldman,Bruce M. Rothschild,Frederick Wolfe +26 more
TL;DR: Clinical criteria for the classification of symptomatic idiopathic (primary) osteoarthritis of the hands were developed from data collected in a multicenter study and required that at least 3 of these 4 criteria be present to classify a patient as having OA of the hand.
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Tetracyclines inhibit connective tissue breakdown: new therapeutic implications for an old family of drugs.
TL;DR: Evidence is presented that this property of tetracycline may be therapeutically useful in retarding pathologic connective tissue breakdown, including bone resorption, and in the treatment of periodontal and several medical diseases.
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Hypothesis: The Humoral Immune Response to Oral Bacteria Provides a Stimulus for the Development of Rheumatoid Arthritis
TL;DR: It is suggested that individuals predisposed to periodontal infection are exposed to antigens generated by PAD, with deiminated fibrin as a likely candidate, which become systemic immunogens and lead to intraarticular inflammation.
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A Non-antibacterial Chemically-modified Tetracycline Inhibits Mammalian Collagenase Activity
TL;DR: In a preliminary in vivo study, pathologically-excessive collagenase in skin and gingiva was induced by rendering adult male rats diabetic, and the oral administration of CMT significantly reduced the excessive collagenase activity in both tissues, suggesting a lack of toxicity in this animal model.