P
Peng Li
Researcher at University of California, Irvine
Publications - 10
Citations - 775
Peng Li is an academic researcher from University of California, Irvine. The author has contributed to research in topics: Electroacupuncture & Rostral ventrolateral medulla. The author has an hindex of 9, co-authored 10 publications receiving 712 citations. Previous affiliations of Peng Li include Fudan University Shanghai Medical College.
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Reversal of Reflex-Induced Myocardial Ischemia by Median Nerve Stimulation A Feline Model of Electroacupuncture
TL;DR: Results suggest that stimulation of the median nerve to mimic electroacupuncture diminishes regional myocardial ischemia triggered by a sympathetically mediated increase in cardiac oxygen demand.
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Naloxone reverses inhibitory effect of electroacupuncture on sympathetic cardiovascular reflex responses.
Dong M. Chao,Lin L. Shen,Stephanie C. Tjen-A-Looi,Koullis F. Pitsillides,Peng Li,John C. Longhurst +5 more
TL;DR: The results indicate that the inhibitory effect of EA on the BK-induced pressor response and the consequent improvement of ischemic dysfunction is dependent on the activation of opioid receptors, specifically receptors located in the rVLM.
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Rostral ventrolateral medullary opioid receptor subtypes in the inhibitory effect of electroacupuncture on reflex autonomic response in cats
TL;DR: A significant portion of inhibition of the gallbladder pressor response by EA is related to activation of mu- and delta-opioid receptors in the rVLM, which appears to play a role in the EA-related modulation of cardiovascular reflex responses.
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Excitatory projections from arcuate nucleus to ventrolateral periaqueductal gray in electroacupuncture inhibition of cardiovascular reflexes.
TL;DR: Exitatory projections from the ARC to the vlPAG are essential to the EA inhibition of the reflex increase in BP induced by SN or gallbladder visceral afferent stimulation, suggesting that excitatory projection to the midbrain ventrolateral periaqueductal gray is essential to this effect.
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Long-loop pathways in cardiovascular electroacupuncture responses.
TL;DR: The vlPAG, particularly, the caudal vl PAG, is required for ARC inhibition of rVLM neuronal activation and subsequent EA-related cardiovascular activation.