C
Catrin Albrecht
Researcher at Leibniz Association
Publications - 76
Citations - 4377
Catrin Albrecht is an academic researcher from Leibniz Association. The author has contributed to research in topics: Oxidative stress & Inflammation. The author has an hindex of 33, co-authored 73 publications receiving 3955 citations. Previous affiliations of Catrin Albrecht include University of Turin & University of Düsseldorf.
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Journal ArticleDOI
Inhaled particles and lung cancer. Part A: Mechanisms.
TL;DR: Since most of the proposed molecular mechanisms underlying particle‐related carcinogenesis have been derived from in vitro studies, there is a need for future studies that evaluate the implication of these mechanisms for in vivo lung cancer development and transgenic and gene knockout animal models may provide a useful tool.
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Cellular responses to nanoparticles: Target structures and mechanisms
Klaus Unfried,Catrin Albrecht,Lars-Oliver Klotz,Anna von Mikecz,Susanne Grether-Beck,Roel P. F. Schins +5 more
TL;DR: An integrated research protocol is proposed to identify fundamental cellular responses to NP in order to complement current toxicological screening strategies with a mechanism-based approach.
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Endocytosis, oxidative stress and IL-8 expression in human lung epithelial cells upon treatment with fine and ultrafine TiO2: role of the specific surface area and of surface methylation of the particles.
Seema Singh,Tingming Shi,Tingming Shi,Rodger Duffin,Rodger Duffin,Catrin Albrecht,Damien van Berlo,Doris Höhr,Bice Fubini,Gianmario Martra,Ivana Fenoglio,Paul J. A. Borm,Paul J. A. Borm,Roel P. F. Schins +13 more
TL;DR: The present data indicate that ultrafine TiO(2), even as aggregates/agglomerates, can trigger inflammatory responses that appear to be driven by their large surface area.
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Cytotoxicity and oxidative DNA damage by nanoparticles in human intestinal Caco-2 cells
TL;DR: The use of engineered nanoparticles in the food sector is anticipated to increase dramatically, whereas their potential hazards for the gastrointestinal tract are still largely unknown as mentioned in this paper, and the potential hazards of nanoparticles are investigated.
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Inhaled particles and lung cancer, part B: paradigms and risk assessment.
TL;DR: Differences in antioxidant protection between different rodent species correlate with susceptibility to PSP‐induced carcinogenicity and support the need for detailed studies on antioxidant response in humans, and scarce data available on humans do not support the overload concept but suggest a role for persistent lung inflammation.