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Jay M. Campbell

Researcher at University of Wisconsin-Madison

Publications -  7
Citations -  2211

Jay M. Campbell is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Endothelial stem cell & HEK 293 cells. The author has an hindex of 6, co-authored 7 publications receiving 1943 citations.

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Collagen reorganization at the tumor-stromal interface facilitates local invasion

TL;DR: Three tumor-associated collagen signatures (TACS) are observed and defined that provide novel markers to locate and characterize tumors and should provide indications that a tumor is, or could become, invasive, and may serve as part of a strategy to help identify and characterize breast tumors in animal and human tissues.
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The C. elegans Hook Protein, ZYG-12, Mediates the Essential Attachment between the Centrosome and Nucleus

TL;DR: It is proposed that dynein and ZYG-12 move the centrosomes toward the nucleus, followed by a ZYg-12/SUN-1-dependent anchorage.
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Involvement of the actin cytoskeleton and homotypic membrane fusion in ER dynamics in Caenorhabditis elegans.

TL;DR: Investigation into the dynamics and regulation of the endoplasmic reticulum in the early Caenorhabditis elegans embryo provides evidence that homotypic membrane fusion is required to form the sheet structure in theEarly embryo.
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Cortical granule exocytosis in C. elegans is regulated by cell cycle components including separase.

TL;DR: In this paper, the authors identified cortical granules in Caenorhabditis elegans and found that degranulation occurs in a wave that initiates in the vicinity of the meiotic spindle during anaphase I. They concluded that osmotic sensitivity phenotypes occur because of defects in trafficking of cortical granule and subsequent formation of an impermeable eggshell.
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Regulation of endothelial cell barrier function by antibody-driven affinity modulation of platelet endothelial cell adhesion molecule-1 (PECAM-1).

TL;DR: The finding that the adhesive properties of PECAM-1 are regulatable suggests novel approaches for controlling endothelial cell migration and barrier function in a variety of vascular permeability disorders.