Example of Pulmonary Medicine format
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Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format
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Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format Example of Pulmonary Medicine format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Pulmonary Medicine — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Pulmonary and Respiratory Medicine #85 of 133 down down by 20 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 45 Published Papers | 85 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 16/06/2020
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Related Journals

open access Open Access

IOP Publishing

Quality:  
High
CiteRatio: 5.4
SJR: 0.967
SNIP: 1.105
open access Open Access

Springer

Quality:  
High
CiteRatio: 7.4
SJR: 0.979
SNIP: 1.483
open access Open Access

Springer

Quality:  
High
CiteRatio: 6.8
SJR: 1.174
SNIP: 1.449
open access Open Access

Springer

Quality:  
High
CiteRatio: 6.6
SJR: 1.846
SNIP: 1.501

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.9

32% from 2019

CiteRatio for Pulmonary Medicine from 2016 - 2020
Year Value
2020 1.9
2019 2.8
2018 2.8
2017 3.0
2016 5.0
graph view Graph view
table view Table view

0.528

7% from 2019

SJR for Pulmonary Medicine from 2016 - 2020
Year Value
2020 0.528
2019 0.566
2018 0.65
2017 0.56
2016 0.84
graph view Graph view
table view Table view

0.944

9% from 2019

SNIP for Pulmonary Medicine from 2016 - 2020
Year Value
2020 0.944
2019 1.033
2018 1.146
2017 0.827
2016 1.185
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 32% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 7% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 9% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Pulmonary Medicine

Guideline source: View

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Hindawi

Pulmonary Medicine

Pulmonary Medicine is a peer-reviewed, open access journal that publishes original research articles, review articles, and clinical studies in all areas of pulmonary medicine.... Read More

Medicine

i
Last updated on
16 Jun 2020
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ISSN
2090-1836
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Impact Factor
Medium - 0.796
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Acceptance Rate
52%
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Frequency
Not provided
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
unsrt
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Citation Type
Numbered
[25]
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Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2013/828939
Risk Factors for Tuberculosis
Padmanesan Narasimhan1, James G. Wood1, Chandini Raina MacIntyre1, Dilip Mathai2
12 Feb 2013 - Pulmonary Medicine

Abstract:

The risk of progression from exposure to the tuberculosis bacilli to the development of active disease is a two-stage process governed by both exogenous and endogenous risk factors. Exogenous factors play a key role in accentuating the progression from exposure to infection among which the bacillary load in the sputum and the... The risk of progression from exposure to the tuberculosis bacilli to the development of active disease is a two-stage process governed by both exogenous and endogenous risk factors. Exogenous factors play a key role in accentuating the progression from exposure to infection among which the bacillary load in the sputum and the proximity of an individual to an infectious TB case are key factors. Similarly endogenous factors lead in progression from infection to active TB disease. Along with well-established risk factors (such as human immunodeficiency virus (HIV), malnutrition, and young age), emerging variables such as diabetes, indoor air pollution, alcohol, use of immunosuppressive drugs, and tobacco smoke play a significant role at both the individual and population level. Socioeconomic and behavioral factors are also shown to increase the susceptibility to infection. Specific groups such as health care workers and indigenous population are also at an increased risk of TB infection and disease. This paper summarizes these factors along with health system issues such as the effects of delay in diagnosis of TB in the transmission of the bacilli. read more read less

Topics:

Risk factors for tuberculosis (61%)61% related to the paper, Endogenous Factors (56%)56% related to the paper, Tuberculosis (55%)55% related to the paper, Population (53%)53% related to the paper, Disease (53%)53% related to the paper
View PDF
515 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/745483
Role of TNF-Alpha, IFN-Gamma, and IL-10 in the Development of Pulmonary Tuberculosis.
28 Nov 2012 - Pulmonary Medicine

Abstract:

Host immune response against Mycobacterium tuberculosis is mediated by cellular immunity, in which cytokines and Th1 cells play a critical role. In the process of control of the infection by mycobacteria, TNF-alpha seems to have a primordial function. This cytokine acts in synergy with IFN-gamma, stimulating the production of... Host immune response against Mycobacterium tuberculosis is mediated by cellular immunity, in which cytokines and Th1 cells play a critical role. In the process of control of the infection by mycobacteria, TNF-alpha seems to have a primordial function. This cytokine acts in synergy with IFN-gamma, stimulating the production of reactive nitrogen intermediates (RNIs), thus mediating the tuberculostatic function of macrophages, and also stimulating the migration of immune cells to the infection site, contributing to granuloma formation, which controls the disease progression. IFN-gamma is the main cytokine involved in the immune response against mycobacteria, and its major function is the activation of macrophages, allowing them to exert its microbicidal role functions. Different from TNF-alpha and IFN-gamma, IL-10 is considered primarily an inhibitory cytokine, important to an adequate balance between inflammatory and immunopathologic responses. The increase in IL-10 levels seems to support the survival of mycobacteria in the host. Although there is not yet conclusive studies concerning a clear dichotomy between Th1 and Th2 responses, involving protective immunity and susceptibility to the disease, respectively, we can suggest that the knowledge about this responses based on the prevailing cytokine profile can help to elucidate the immune response related to the protection against M. tuberculosis. read more read less

Topics:

Immune system (56%)56% related to the paper, Cellular immunity (56%)56% related to the paper, Interleukin 10 (56%)56% related to the paper, Cytokine (56%)56% related to the paper, Tumor necrosis factor alpha (53%)53% related to the paper
View PDF
244 Citations
open accessOpen access Journal Article DOI: 10.1155/2020/6175964
Pulmonary Fibrosis in COVID-19 Survivors: Predictive Factors and Risk Reduction Strategies.
Ademola S. Ojo1, Simon A. Balogun2, Oyeronke T. Williams2, O S Ojo2
11 Aug 2020 - Pulmonary Medicine

Abstract:

Although pulmonary fibrosis can occur in the absence of a clear-cut inciting agent, and without a clinically clear initial acute inflammatory phase, it is more commonly associated with severe lung injury. This may be due to respiratory infections, chronic granulomatous diseases, medications, and connective tissue disorders. P... Although pulmonary fibrosis can occur in the absence of a clear-cut inciting agent, and without a clinically clear initial acute inflammatory phase, it is more commonly associated with severe lung injury. This may be due to respiratory infections, chronic granulomatous diseases, medications, and connective tissue disorders. Pulmonary fibrosis is associated with permanent pulmonary architectural distortion and irreversible lung dysfunction. Available clinical, radiographic, and autopsy data has indicated that pulmonary fibrosis is central to severe acute respiratory distress syndrome (SARS) and MERS pathology, and current evidence suggests that pulmonary fibrosis could also complicate infection by SARS-CoV-2. The aim of this review is to explore the current literature on the pathogenesis of lung injury in COVID-19 infection. We evaluate the evidence in support of the putative risk factors for the development of lung fibrosis in the disease and propose risk mitigation strategies. We conclude that, from the available literature, the predictors of pulmonary fibrosis in COVID-19 infection are advanced age, illness severity, length of ICU stay and mechanical ventilation, smoking and chronic alcoholism. With no proven effective targeted therapy against pulmonary fibrosis, risk reduction measures should be directed at limiting the severity of the disease and protecting the lungs from other incidental injuries. read more read less

Topics:

Pulmonary fibrosis (67%)67% related to the paper, Lung injury (60%)60% related to the paper, Lung (59%)59% related to the paper, Pneumonia (54%)54% related to the paper, Disease (50%)50% related to the paper
View PDF
196 Citations
open accessOpen access Journal Article DOI: 10.1155/2013/956081
Inspiratory Capacity during Exercise: Measurement, Analysis, and Interpretation
Jordan A. Guenette1, Roberto C. Chin2, Julia Cory3, Katherine A. Webb3, Denis E. O'Donnell3
07 Feb 2013 - Pulmonary Medicine

Abstract:

Cardiopulmonary exercise testing (CPET) is an established method for evaluating dyspnea and ventilatory abnormalities. Ventilatory reserve is typically assessed as the ratio of peak exercise ventilation to maximal voluntary ventilation. Unfortunately, this crude assessment provides limited data on the factors that limit the n... Cardiopulmonary exercise testing (CPET) is an established method for evaluating dyspnea and ventilatory abnormalities. Ventilatory reserve is typically assessed as the ratio of peak exercise ventilation to maximal voluntary ventilation. Unfortunately, this crude assessment provides limited data on the factors that limit the normal ventilatory response to exercise. Additional measurements can provide a more comprehensive evaluation of respiratory mechanical constraints during CPET (e.g., expiratory flow limitation and operating lung volumes). These measurements are directly dependent on an accurate assessment of inspiratory capacity (IC) throughout rest and exercise. Despite the valuable insight that the IC provides, there are no established recommendations on how to perform the maneuver during exercise and how to analyze and interpret the data. Accordingly, the purpose of this manuscript is to comprehensively examine a number of methodological issues related to the measurement, analysis, and interpretation of the IC. We will also briefly discuss IC responses to exercise in health and disease and will consider how various therapeutic interventions influence the IC, particularly in patients with chronic obstructive pulmonary disease. Our main conclusion is that IC measurements are both reproducible and responsive to therapy and provide important information on the mechanisms of dyspnea and exercise limitation during CPET. read more read less

Topics:

Inspiratory Capacity (52%)52% related to the paper
View PDF
138 Citations
open accessOpen access Journal Article DOI: 10.1155/2013/601737
Interferon-Gamma Release Assays versus Tuberculin Skin Testing for the Diagnosis of Latent Tuberculosis Infection: An Overview of the Evidence
Anete Trajman, R. E. Steffen1, Dick Menzies1
07 Feb 2013 - Pulmonary Medicine

Abstract:

A profusion of articles have been published on the accuracy and uses of interferon-gamma releasing assays. Here we review the clinical applications, advantages, and limitations of the tuberculin skin test and interferon-gamma release assays and provide an overview of the most recent systematic reviews conducted for different ... A profusion of articles have been published on the accuracy and uses of interferon-gamma releasing assays. Here we review the clinical applications, advantages, and limitations of the tuberculin skin test and interferon-gamma release assays and provide an overview of the most recent systematic reviews conducted for different indications for the use of these tests. We conclude that both tests are accurate to detect latent tuberculosis, although interferon-gamma release assays have higher specificity than tuberculin skin testing in BCG-vaccinated populations, particularly if BCG is received after infancy. However, both tests perform poorly to predict risk for progression to active tuberculosis. Interferon-gamma release assays have significant limitations in serial testing because of spontaneous variability and lack of a validated definition of conversion and reversion, making it difficult for clinicians to interpret changes in category (conversions and reversions). So far, the most important clinical evidence, that is, that isoniazid preventive therapy reduces the risk for progression to disease, has been produced only in tuberculin skin test-positive individuals. read more read less

Topics:

Latent tuberculosis (62%)62% related to the paper, Tuberculin (59%)59% related to the paper
View PDF
126 Citations
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With SciSpace, you do not need a word template for Pulmonary Medicine.

It automatically formats your research paper to Hindawi formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

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Time taken to format a paper and Compliance with guidelines

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Pulmonary Medicine format uses unsrt citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Pulmonary Medicine in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Pulmonary Medicine guidelines and auto format it.

2. Do you follow the Pulmonary Medicine guidelines?

Yes, the template is compliant with the Pulmonary Medicine guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Pulmonary Medicine?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Pulmonary Medicine citation style.

4. Can I use the Pulmonary Medicine templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Pulmonary Medicine.

5. Can I use a manuscript in Pulmonary Medicine that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Pulmonary Medicine that you can download at the end.

6. How long does it usually take you to format my papers in Pulmonary Medicine?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Pulmonary Medicine.

7. Where can I find the template for the Pulmonary Medicine?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pulmonary Medicine's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Pulmonary Medicine's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Pulmonary Medicine an online tool or is there a desktop version?

SciSpace's Pulmonary Medicine is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Pulmonary Medicine?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Pulmonary Medicine?”

11. What is the output that I would get after using Pulmonary Medicine?

After writing your paper autoformatting in Pulmonary Medicine, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Pulmonary Medicine's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Pulmonary Medicine?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Pulmonary Medicine. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Pulmonary Medicine?

The 5 most common citation types in order of usage for Pulmonary Medicine are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Pulmonary Medicine?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pulmonary Medicine's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Pulmonary Medicine in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Pulmonary Medicine Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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Researcher & Ex MS Word user
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