Example of Acta Neurologica Scandinavica format
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Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format Example of Acta Neurologica Scandinavica format
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open access Open Access

Acta Neurologica Scandinavica — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Neurology (clinical) #74 of 343 up up by 5 ranks
Neurology #44 of 156 up up by 3 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 571 Published Papers | 3330 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 24/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.684

6% from 2018

Impact factor for Acta Neurologica Scandinavica from 2016 - 2019
Year Value
2019 2.684
2018 2.852
2017 3.126
2016 3.087
graph view Graph view
table view Table view

5.8

5% from 2019

CiteRatio for Acta Neurologica Scandinavica from 2016 - 2020
Year Value
2020 5.8
2019 5.5
2018 5.1
2017 5.2
2016 5.2
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 6% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.967

3% from 2019

SJR for Acta Neurologica Scandinavica from 2016 - 2020
Year Value
2020 0.967
2019 0.943
2018 1.225
2017 1.116
2016 1.219
graph view Graph view
table view Table view

1.145

0% from 2019

SNIP for Acta Neurologica Scandinavica from 2016 - 2020
Year Value
2020 1.145
2019 1.143
2018 1.052
2017 1.169
2016 1.077
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 3% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 0% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Acta Neurologica Scandinavica

Guideline source: View

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Wiley

Acta Neurologica Scandinavica

Acta Neurologica Scandinavica aims to publish manuscripts of a high scientific quality representing original clinical, diagnostic or experimental work in neuroscience. The journla's scope is to act as an international forum for the dissemination of information advancing the sc...... Read More

Medicine

i
Last updated on
23 Jun 2020
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ISSN
0001-6314
i
Impact Factor
High - 1.094
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
apa
i
Citation Type
Numbered
[25]
i
Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1111/J.1600-0404.1984.TB07822.X
Models for studying long-term recovery following forebrain ischemia in the rat. 2. A 2-vessel occlusion model
Maj-Lis Smith1, George Bendek1, Nils Dahlgren1, Ingmar Rosén1, Tadeusz Wieloch1, Bo K. Siesjö1

Abstract:

A model is described in which transient ischemia is induced in rats anaesthetized with N2O:O2 (70:30) by bilateral carotid artery clamping combined with a lowering of mean arterial blood pressure to 50 mm Hg, the latter being achieved by bleeding, or by bleeding supplemented with administration of trimetaphan or phentolamine.... A model is described in which transient ischemia is induced in rats anaesthetized with N2O:O2 (70:30) by bilateral carotid artery clamping combined with a lowering of mean arterial blood pressure to 50 mm Hg, the latter being achieved by bleeding, or by bleeding supplemented with administration of trimetaphan or phentolamine. By the use of intubation, muscle paralysis with suxamethonium chloride, and insertion of tail arterial and venous catheters, it was possible to induce reversible ischemia for long-term recovery studies. Autoradiographic measurements of local CBF showed that the procedure reduced CBF in neocortical areas, hippocampus, and caudoputamen to near-zero values, flow rates in a number of subcortical areas being variable. Administration of trimethaphane or phentolamine did not affect ischemic and postischemic flow rates, nor did they alter recovery of EEG and sensory-evoked responses, but trimetaphan blunted the changes in plasma concentrations of adrenaline and noradrenaline. Recovery experiments showed that 10 min of ischemia gave rise to clear signs of permanent brain damage, with a small number of animals developing postischemic seizures that led to the death of the animals in status epilepticus. After 15 min of ischemia, such alterations were more pronounced, and the majority of animals died. It is concluded that the short revival times noted are explained by the fact that the model induces near-complete ischemia, and that recovery following forebrain ischemia is critically dependent on residual flow rates during the period of ischemia. read more read less

Topics:

Ischemia (60%)60% related to the paper, Phentolamine (50%)50% related to the paper
759 Citations
Journal Article DOI: 10.1111/J.1600-0404.1996.TB05866.X
Evolution of the neuropathology of Alzheimer's disease
Heiko Braak1, Eva Braak1

Abstract:

Our knowledge of the etiology and pathogenesis of Alzheimer's disease is limited. The most conspicuous changes seen in the brain are deposits of insoluble proteins in both extracellular and intraneuronal locations. The extracellular deposits consist primarily of a specific A4 amyloid protein. The significance of these deposit... Our knowledge of the etiology and pathogenesis of Alzheimer's disease is limited. The most conspicuous changes seen in the brain are deposits of insoluble proteins in both extracellular and intraneuronal locations. The extracellular deposits consist primarily of a specific A4 amyloid protein. The significance of these deposits remains to be determined, as they are often found in the cerebral cortex of non-demented elderly persons. More telling is the gradual accumulation of insoluble fibrous material within some neurons that consists mainly of abnormally phosphorylated tau protein. Six stages of increasingly severe cortical destruction can be distinguished. Stages I and II are characterized by neurofibrillary changes that are largely confined to the transentorhinal region, whereas stages III and IV are marked by severe involvement of both the entorhinal and transentorhinal regions. Isocortical destruction occurs during stages V and VI. This progression in cortical pathology correlates with the gradual worsening of clinical symptoms. read more read less

Topics:

Neuropathology (51%)51% related to the paper, Tau protein (51%)51% related to the paper, Alzheimer's disease (51%)51% related to the paper
662 Citations
open accessOpen access Journal Article
Transient global amnesia.

Topics:

Transient global amnesia (78%)78% related to the paper, Amnesia (69%)69% related to the paper
630 Citations
Journal Article DOI: 10.1111/J.1600-0404.1995.TB07018.X
Multiple choice vocabulary test MWT as a valid and short test to estimate premorbid intelligence
Siegfried Lehrl1, Gerhard Triebig2, Bernd Fischer

Abstract:

The discrepancy between current and premorbid ability is a relevant indicator of acquired mental impairment, which itself is closely related to general cerebral dysfunction. The use of tests sensitive to cerebral dysfunction, raises relatively few problems compared with tests being resistant that are used to estimate premorbi... The discrepancy between current and premorbid ability is a relevant indicator of acquired mental impairment, which itself is closely related to general cerebral dysfunction. The use of tests sensitive to cerebral dysfunction, raises relatively few problems compared with tests being resistant that are used to estimate premorbid mental ability. For premorbid ability, verbal tests assessing knowledge, especially vocabulary, have been shown to be valid. A test, possibly more insensitive to brain dysfunction than the ones usually administered, is the multiple choice vocabulary test (MWT = Mehrfachwahl-Wortschatz-Test). At present only German versions are available. They are presented in some detail because of their advantages. Construction of the MWT is simple, and it can be easily administered in about five minutes. The results correlate fairly well with global IQ in healthy adults (median of r = 0.72 in 22 samples) and are more insensitive to current disturbances than such tests as the WAIS vocabulary test. The limitations of premorbid tests with respect to diagnostic validity are discussed. It is concluded, that studies which do not control premorbid intelligence have to be considered as a "malpractice" and should not be accepted by scientists. read more read less

Topics:

Wechsler Adult Intelligence Scale (53%)53% related to the paper, Intelligence quotient (52%)52% related to the paper, Test validity (51%)51% related to the paper
574 Citations
Journal Article DOI: 10.1111/J.1600-0404.2004.00324.X
The psychiatric comorbidity of epilepsy
Athanasios Gaitatzis1, Michael R. Trimble1, Josemir W. Sander1

Abstract:

Several studies have assessed the prevalence of psychiatric disorders in epilepsy. They are characterized by considerable heterogeneity, because of differences in the population setting and type of study. A non-systematic review of the literature allows us to draw some useful, although not definite, conclusions. Six per cent ... Several studies have assessed the prevalence of psychiatric disorders in epilepsy. They are characterized by considerable heterogeneity, because of differences in the population setting and type of study. A non-systematic review of the literature allows us to draw some useful, although not definite, conclusions. Six per cent of people with epilepsy in the general population appear to suffer from a psychiatric disorder, while this rises to 10-20% in populations with temporal lobe and/or refractory epilepsy. Mood disorders are the most common culprit (24-74%), particularly depression (30%), followed by anxiety disorders (10-25%), psychoses (2-7%) and personality disorders (1-2%). This comorbidity appears to be related to endogenous and exogenous (including iatrogenic) factors and to the severity and chronicity of epilepsy. Conditions such as schizophrenia-like psychosis of epilepsy and interictal dysphoric disorder are represented only in epilepsy. Adequate recognition and treatment of psychiatric conditions in epilepsy is essential for patient management because of their considerable burden in morbidity and quality of life. read more read less

Topics:

Generalized epilepsy (65%)65% related to the paper, Interictal dysphoric disorder (64%)64% related to the paper, Epilepsy (62%)62% related to the paper, Comorbidity (56%)56% related to the paper, Mood disorders (54%)54% related to the paper
503 Citations
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Acta Neurologica Scandinavica format uses apa citation style.

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Frequently asked questions

1. Can I write Acta Neurologica Scandinavica in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Acta Neurologica Scandinavica guidelines and auto format it.

2. Do you follow the Acta Neurologica Scandinavica guidelines?

Yes, the template is compliant with the Acta Neurologica Scandinavica guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Acta Neurologica Scandinavica?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Acta Neurologica Scandinavica citation style.

4. Can I use the Acta Neurologica Scandinavica templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Acta Neurologica Scandinavica.

5. Can I use a manuscript in Acta Neurologica Scandinavica that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Acta Neurologica Scandinavica that you can download at the end.

6. How long does it usually take you to format my papers in Acta Neurologica Scandinavica?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Acta Neurologica Scandinavica.

7. Where can I find the template for the Acta Neurologica Scandinavica?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Acta Neurologica Scandinavica's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Acta Neurologica Scandinavica's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Acta Neurologica Scandinavica an online tool or is there a desktop version?

SciSpace's Acta Neurologica Scandinavica is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Acta Neurologica Scandinavica?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Acta Neurologica Scandinavica?”

11. What is the output that I would get after using Acta Neurologica Scandinavica?

After writing your paper autoformatting in Acta Neurologica Scandinavica, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Acta Neurologica Scandinavica's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Acta Neurologica Scandinavica?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Acta Neurologica Scandinavica. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Acta Neurologica Scandinavica?

The 5 most common citation types in order of usage for Acta Neurologica Scandinavica are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Acta Neurologica Scandinavica?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Acta Neurologica Scandinavica's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Acta Neurologica Scandinavica in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Acta Neurologica Scandinavica Endnote style according to Elsevier guidelines.

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