Example of Cellular Microbiology format
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Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format
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Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format Example of Cellular Microbiology format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Cellular Microbiology — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Microbiology #40 of 150 down down by 23 ranks
Virology #21 of 69 down down by 9 ranks
Immunology #74 of 202 down down by 40 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 489 Published Papers | 3160 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 11/06/2020
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Related Journals

open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 11.0
SJR: 3.719
SNIP: 1.882
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American Society for Microbiology

Quality:  
High
CiteRatio: 8.9
SJR: 2.617
SNIP: 1.372
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Nature

Quality:  
High
CiteRatio: 28.2
SJR: 7.305
SNIP: 3.41
open access Open Access

Frontiers Media

Quality:  
High
CiteRatio: 6.5
SJR: 1.812
SNIP: 1.485

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.43

20% from 2018

Impact factor for Cellular Microbiology from 2016 - 2019
Year Value
2019 3.43
2018 4.288
2017 4.41
2016 4.554
graph view Graph view
table view Table view

6.5

14% from 2019

CiteRatio for Cellular Microbiology from 2016 - 2020
Year Value
2020 6.5
2019 7.6
2018 8.1
2017 8.8
2016 8.7
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 20% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 14% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.542

21% from 2019

SJR for Cellular Microbiology from 2016 - 2020
Year Value
2020 1.542
2019 1.954
2018 2.126
2017 2.52
2016 2.707
graph view Graph view
table view Table view

0.992

3% from 2019

SNIP for Cellular Microbiology from 2016 - 2020
Year Value
2020 0.992
2019 1.019
2018 0.986
2017 1.064
2016 1.077
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 21% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 3% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Cellular Microbiology

Guideline source: View

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Wiley

Cellular Microbiology

Cellular Microbiology aims to publish significant contributions on the intersection of microbial and host-cell biology that strongly advance understanding of unique biological processes and mechanisms. The scope includes the host cell responses and interactions elicited by pro...... Read More

Microbiology

Virology

Immunology

Immunology and Microbiology

i
Last updated on
11 Jun 2020
i
ISSN
1462-5814
i
Impact Factor
High - 1.136
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
apa
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Citation Type
Numbered
[25]
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Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1111/J.1462-5822.2007.00901.X
Lounging in a lysosome: the intracellular lifestyle of Coxiella burnetii.
Daniel E. Voth1, Robert A. Heinzen
01 Apr 2007 - Cellular Microbiology

Abstract:

Summary Most intracellular parasites employ sophisticated mechanisms to direct biogenesis of a vacuolar replicative niche that circumvents default maturation through the endolysosomal cascade. However, this is not the case of the Q fever bacterium, Coxiella burnetii. This hardy, obligate intracellular pathogen has evolved to... Summary Most intracellular parasites employ sophisticated mechanisms to direct biogenesis of a vacuolar replicative niche that circumvents default maturation through the endolysosomal cascade. However, this is not the case of the Q fever bacterium, Coxiella burnetii. This hardy, obligate intracellular pathogen has evolved to not only survive, but to thrive, in the harshest of intracellular compartments: the phagolysosome. Following internalization, the nascent Coxiella phagosome ultimately develops into a large and spacious parasitophorous vacuole (PV) that acquires lysosomal characteristics such as acidic pH, acid hydrolases and cationic peptides, defences designed to rid the host of intruders. However, transit of Coxiella to this environment is initially stalled, a process that is apparently modulated by interactions with the autophagic pathway. Coxiella actively participates in biogenesis of its PV by synthesizing proteins that mediate phagosome stalling, autophagic interactions, and development and maintenance of the mature vacuole. Among the potential mechanisms mediating these processes is deployment of a type IV secretion system to deliver effector proteins to the host cytosol. Here we summarize our current understanding of the cellular events that occur during parasitism of host cells by Coxiella. read more read less

Topics:

Coxiella burnetii (60%)60% related to the paper, Phagosome (54%)54% related to the paper, Phagolysosome (54%)54% related to the paper, Intracellular parasite (52%)52% related to the paper
View PDF
1,004 Citations
Journal Article DOI: 10.1111/J.1462-5822.2009.01323.X
Evolving concepts in biofilm infections
Luanne Hall-Stoodley, Paul Stoodley1
01 Jul 2009 - Cellular Microbiology

Abstract:

Several pathogens associated with chronic infections, including Pseudomonas aeruginosa in cystic fibrosis pneumonia, Haemophilus influenzae and Streptococcus pneumoniae in chronic otitis media, Staphylococcus aureus in chronic rhinosinusitis and enteropathogenic Escherichia coli in recurrent urinary tract infections, are link... Several pathogens associated with chronic infections, including Pseudomonas aeruginosa in cystic fibrosis pneumonia, Haemophilus influenzae and Streptococcus pneumoniae in chronic otitis media, Staphylococcus aureus in chronic rhinosinusitis and enteropathogenic Escherichia coli in recurrent urinary tract infections, are linked to biofilm formation. Biofilms are usually defined as surface-associated microbial communities, surrounded by an extracellular polymeric substance (EPS) matrix. Biofilm formation has been demonstrated for numerous pathogens and is clearly an important microbial survival strategy. However, outside of dental plaques, fewer reports have investigated biofilm development in clinical samples. Typically biofilms are found in chronic diseases that resist host immune responses and antibiotic treatment and these characteristics are often cited for the ability of bacteria to persist in vivo. This review examines some recent attempts to examine the biofilm phenotype in vivo and discusses the challenges and implications for defining a biofilm phenotype read more read less

Topics:

Biofilm (62%)62% related to the paper, Pseudomonas aeruginosa (53%)53% related to the paper
View PDF
990 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1462-5822.2005.00659.X
Neutrophil extracellular traps capture and kill Candida albicans yeast and hyphal forms
Constantin F. Urban1, Ulrike Reichard1, Volker Brinkmann1, Arturo Zychlinsky1
01 Apr 2006 - Cellular Microbiology

Abstract:

Neutrophils phagocytose and kill microbes upon phagolysosomal fusion. Recently we found that activated neutrophils form extracellular fibres that consist of granule proteins and chromatin. These neutrophil extracellular traps (NETs) degrade virulence factors and kill Gram positive and negative bacteria. Here we show for the f... Neutrophils phagocytose and kill microbes upon phagolysosomal fusion. Recently we found that activated neutrophils form extracellular fibres that consist of granule proteins and chromatin. These neutrophil extracellular traps (NETs) degrade virulence factors and kill Gram positive and negative bacteria. Here we show for the first time that Candida albicans, a eukaryotic pathogen, induces NET-formation and is susceptible to NET-mediated killing. C. albicans is the predominant aetiologic agent of fungal infections in humans, particularly in immunocompromised hosts. One major virulence trait of C. albicans is its ability to reversibly switch from singular budding cells to filamentous hyphae. We demonstrate that NETs kill both yeast-form and hyphal cells, and that granule components mediate fungal killing. Taken together our data indicate that neutrophils trap and kill ascomycetous yeasts by forming NETs. read more read less

Topics:

Candida albicans (60%)60% related to the paper, Neutrophil extracellular traps (59%)59% related to the paper, Extracellular Traps (53%)53% related to the paper, Corpus albicans (52%)52% related to the paper
View PDF
896 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1462-5822.2006.00751.X
Caspase-1-dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages.
Susan L. Fink1, Brad T. Cookson1
01 Nov 2006 - Cellular Microbiology

Abstract:

Salmonella enterica serovar Typhimurium invades host macrophages and induces a unique caspase-1-dependent pathway of cell death termed pyroptosis, which is activated during bacterial infection in vivo. We demonstrate DNA cleavage during pyroptosis results from caspase-1-stimulated nuclease activity. Although poly(ADP-ribose) ... Salmonella enterica serovar Typhimurium invades host macrophages and induces a unique caspase-1-dependent pathway of cell death termed pyroptosis, which is activated during bacterial infection in vivo. We demonstrate DNA cleavage during pyroptosis results from caspase-1-stimulated nuclease activity. Although poly(ADP-ribose) polymerase (PARP) activation by fragmented DNA depletes cellular ATP to cause lysis during oncosis, the rapid lysis characteristic of Salmonella-infected macrophages does not require PARP activity or DNA fragmentation. Membrane pores between 1.1 and 2.4 nm in diameter form during pyroptosis of host cells and cause swelling and osmotic lysis. Pore formation requires host cell actin cytoskeleton rearrangements and caspase-1 activity, as well as the bacterial type III secretion system (TTSS); however, insertion of functional TTSS translocons into the host membrane is not sufficient to directly evoke pore formation. Concurrent with pore formation, inflammatory cytokines are released from infected macrophages. This mechanism of caspase-1-mediated cell death provides additional experimental evidence supporting pyroptosis as a novel pathway of inflammatory programmed cell death. read more read less

Topics:

Pyroptosis (69%)69% related to the paper, Caspase 1 (63%)63% related to the paper, Actin cytoskeleton (54%)54% related to the paper, Poly ADP ribose polymerase (52%)52% related to the paper, Cytolysis (52%)52% related to the paper
View PDF
894 Citations
Journal Article DOI: 10.1111/J.1462-5822.2004.00361.X
The role and regulation of programmed cell death in plant–pathogen interactions
Jean T. Greenberg1, Nan Yao1
01 Mar 2004 - Cellular Microbiology

Abstract:

It is commonly known that animal pathogens often target and suppress programmed cell death (pcd) pathway components to manipulate their hosts. In contrast, plant pathogens often trigger pcd. In cases in which plant pcd accompanies disease resistance, an event called the hypersensitive response, the plant surveillance system h... It is commonly known that animal pathogens often target and suppress programmed cell death (pcd) pathway components to manipulate their hosts. In contrast, plant pathogens often trigger pcd. In cases in which plant pcd accompanies disease resistance, an event called the hypersensitive response, the plant surveillance system has learned to detect pathogen-secreted molecules in order to mount a defence response. In plants without genetic disease resistance, these secreted molecules serve as virulence factors that act through largely unknown mechanisms. Recent studies suggest that plant bacterial pathogens also secrete antiapoptotic proteins to promote their virulence. In contrast, a number of fungal pathogens secrete pcd-promoting molecules that are critical virulence factors. Here, we review recent progress in determining the role and regulation of plant pcd responses that accompany both resistance and susceptible interactions. We also review progress in discerning the mechanisms by which plant pcd occurs during these different interactions. read more read less

Topics:

Hypersensitive response (53%)53% related to the paper
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701 Citations
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Cellular Microbiology format uses apa citation style.

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Frequently asked questions

1. Can I write Cellular Microbiology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cellular Microbiology guidelines and auto format it.

2. Do you follow the Cellular Microbiology guidelines?

Yes, the template is compliant with the Cellular Microbiology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cellular Microbiology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cellular Microbiology citation style.

4. Can I use the Cellular Microbiology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cellular Microbiology.

5. Can I use a manuscript in Cellular Microbiology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cellular Microbiology that you can download at the end.

6. How long does it usually take you to format my papers in Cellular Microbiology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cellular Microbiology.

7. Where can I find the template for the Cellular Microbiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cellular Microbiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cellular Microbiology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Cellular Microbiology an online tool or is there a desktop version?

SciSpace's Cellular Microbiology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cellular Microbiology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Cellular Microbiology?”

11. What is the output that I would get after using Cellular Microbiology?

After writing your paper autoformatting in Cellular Microbiology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cellular Microbiology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cellular Microbiology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cellular Microbiology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cellular Microbiology?

The 5 most common citation types in order of usage for Cellular Microbiology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cellular Microbiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cellular Microbiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cellular Microbiology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cellular Microbiology Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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