H
Hideki Fujii
Researcher at Kobe University
Publications - 132
Citations - 2896
Hideki Fujii is an academic researcher from Kobe University. The author has contributed to research in topics: Kidney disease & Internal medicine. The author has an hindex of 27, co-authored 109 publications receiving 2523 citations.
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Journal ArticleDOI
Clinical Practice Guideline for the Management of Chronic Kidney Disease-Mineral and Bone Disorder
Masafumi Fukagawa,Keitaro Yokoyama,Fumihiko Koiwa,Masatomo Taniguchi,Tetsuo Shoji,Junichiro James Kazama,Hirotaka Komaba,Ryoichi Ando,Takatoshi Kakuta,Hideki Fujii,Msasaaki Nakayama,Yugo Shibagaki,Seiji Fukumoto,Naohiko Fujii,Motoshi Hattori,Akira Ashida,Kunitoshi Iseki,Takashi Shigematsu,Yusuke Tsukamoto,Yoshiharu Tsubakihara,Tadashi Tomo,Hideki Hirakata,Tadao Akizawa +22 more
TL;DR: CKD-MBD Guideline Working Group, Japanese Society for Dialysis Therapy, and Tadao Akizawa thank the authors for their help and assistance in the development of this guideline.
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Depressed expression of Klotho and FGF receptor 1 in hyperplastic parathyroid glands from uremic patients
Hirotaka Komaba,Shunsuke Goto,Hideki Fujii,Yasuhiro Hamada,Akira Kobayashi,Koji Shibuya,Yoshihiro Tominaga,Naoki Otsuki,Ken-ichi Nibu,Kimie Nakagawa,Naoko Tsugawa,Toshio Okano,Riko Kitazawa,Masafumi Fukagawa +13 more
TL;DR: The results suggest that the depressed expression of the Klotho-FGFR1 complex in hyperplastic glands underlies the pathogenesis of SHPT and its resistance to extremely high FGF23 levels in uremic patients.
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Vitamin D Activates the Nrf2-Keap1 Antioxidant Pathway and Ameliorates Nephropathy in Diabetic Rats
Kentaro Nakai,Hideki Fujii,Keiji Kono,Shunsuke Goto,Riko Kitazawa,Sohei Kitazawa,Michinori Hirata,Masami Shinohara,Masafumi Fukagawa,Masafumi Fukagawa,Shinichi Nishi +10 more
TL;DR: It is suggested that maxacalcitol attenuates the progression of diabetic nephropathy by suppression of oxidative stress and amelioration of the Nrf2-Keap1 pathway in nonobese type 2 diabetes without significant changes in blood pressure and glomerular filtration rate.
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Role of oxidative stress in diabetic bone disorder.
TL;DR: The impact of diabetes-induced oxidative stress in the development of diabetic bone disorder is focused on and it is demonstrated that both streptozotocin-induced diabetic mice and spontaneously diabetic Torii rats, an animal model of type 2 diabetes, have low-turnover osteopenia associated with increased oxidative stress.
Journal ArticleDOI
Histomorphometric analysis of diabetic osteopenia in streptozotocin-induced diabetic mice: A possible role of oxidative stress
TL;DR: It is demonstrated that streptozotocin-induced diabetic mice exhibit low-turnover osteopenia associated with increased oxidative stress, and intensified immunostaining of an oxidative stress marker in bone tissue including the osteoblasts of diabetic mice.