Example of Neurochemical Research format
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Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format
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Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format Example of Neurochemical Research format
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open access Open Access

Neurochemical Research — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Biochemistry #116 of 415 up up by 50 ranks
Cellular and Molecular Neuroscience #38 of 88 up up by 17 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 1025 Published Papers | 6374 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 19/06/2020
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Related Journals

open access Open Access

Springer

Quality:  
High
CiteRatio: 5.7
SJR: 0.851
SNIP: 0.946
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Taylor and Francis

Quality:  
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CiteRatio: 13.9
SJR: 4.634
SNIP: 2.046
open access Open Access

Springer

Quality:  
High
CiteRatio: 4.3
SJR: 0.633
SNIP: 1.433
open access Open Access

De Gruyter

Quality:  
High
CiteRatio: 6.5
SJR: 1.246
SNIP: 0.854

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.038

9% from 2018

Impact factor for Neurochemical Research from 2016 - 2019
Year Value
2019 3.038
2018 2.782
2017 2.772
2016 2.581
graph view Graph view
table view Table view

6.2

13% from 2019

CiteRatio for Neurochemical Research from 2016 - 2020
Year Value
2020 6.2
2019 5.5
2018 4.7
2017 4.6
2016 4.5
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 9% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 13% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.102

21% from 2019

SJR for Neurochemical Research from 2016 - 2020
Year Value
2020 1.102
2019 0.91
2018 0.973
2017 1.06
2016 1.017
graph view Graph view
table view Table view

0.933

16% from 2019

SNIP for Neurochemical Research from 2016 - 2020
Year Value
2020 0.933
2019 0.805
2018 0.772
2017 0.873
2016 0.754
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 21% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 16% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Neurochemical Research

Guideline source: View

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Springer

Neurochemical Research

Neurochemical Research is devoted to the rapid publication of studies that use neurochemical methodology in research on nervous system structure and function. The journal publishes original reports of experimental and clinical research results, perceptive reviews of significan...... Read More

Medicine

i
Last updated on
19 Jun 2020
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ISSN
0364-3190
i
Impact Factor
Medium - 0.811
i
Acceptance Rate
66%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
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Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1023/A:1007677003387
Glial Fibrillary Acidic Protein: GFAP-Thirty-One Years (1969–2000)
Lawrence F. Eng1, Roopa S. Ghirnikar1, Yuen L. Lee1
01 Oct 2000 - Neurochemical Research

Abstract:

It is now well established that the glial fibrillary acidic protein (GFAP) is the principal 8-9 nm intermediate filament in mature astrocytes of the central nervous system (CNS). Over a decade ago, the value of GFAP as a prototype antigen in nervous tissue identification and as a standard marker for fundamental and applied re... It is now well established that the glial fibrillary acidic protein (GFAP) is the principal 8-9 nm intermediate filament in mature astrocytes of the central nervous system (CNS). Over a decade ago, the value of GFAP as a prototype antigen in nervous tissue identification and as a standard marker for fundamental and applied research at an interdisciplinary level was recognized (Raine, 135). As a member of the cytoskeletal protein family, GFAP is thought to be important in modulating astrocyte motility and shape by providing structural stability to astrocytic processes. In the CNS of higher vertebrates, following injury, either as a result of trauma, disease, genetic disorders, or chemical insult, astrocytes become reactive and respond in a typical manner, termed astrogliosis. Astrogliosis is characterized by rapid synthesis of GFAP and is demonstrated by increase in protein content or by immunostaining with GFAP antibody. In addition to the major application of GFAP antisera for routine use in astrocyte identification in the CNS, the molecular cloning of the mouse gene in 1985 has opened a new and rich realm for GFAP studies. These include antisense, null mice, and numerous promoter studies. Studies showing that mice lacking GFAP are hypersensitive to cervical spinal cord injury caused by sudden acceleration of the head have provided more direct evidence for a structural role of GFAP. While the structural function of GFAP has become more acceptable, the use of GFAP antibodies and promoters continue to be valuable in studying CNS injury, disease, and development. read more read less

Topics:

GFAP stain (72%)72% related to the paper, Astrogliosis (63%)63% related to the paper, Glial fibrillary acidic protein (61%)61% related to the paper, Astrocyte (53%)53% related to the paper, Neuroglia (51%)51% related to the paper
1,260 Citations
open accessOpen access Journal Article DOI: 10.1007/S11064-015-1581-6
The Glymphatic System: A Beginner’s Guide
Nadia Aalling Jessen1, Anne Sofie Finmann Munk1, Iben Lundgaard1
07 May 2015 - Neurochemical Research

Abstract:

The glymphatic system is a recently discovered macroscopic waste clearance system that utilizes a unique system of perivascular tunnels, formed by astroglial cells, to promote efficient elimination of soluble proteins and metabolites from the central nervous system. Besides waste elimination, the glymphatic system also facili... The glymphatic system is a recently discovered macroscopic waste clearance system that utilizes a unique system of perivascular tunnels, formed by astroglial cells, to promote efficient elimination of soluble proteins and metabolites from the central nervous system. Besides waste elimination, the glymphatic system also facilitates brain-wide distribution of several compounds, including glucose, lipids, amino acids, growth factors, and neuromodulators. Intriguingly, the glymphatic system function mainly during sleep and is largely disengaged during wakefulness. The biological need for sleep across all species may therefore reflect that the brain must enter a state of activity that enables elimination of potentially neurotoxic waste products, including β-amyloid. Since the concept of the glymphatic system is relatively new, we will here review its basic structural elements, organization, regulation, and functions. We will also discuss recent studies indicating that glymphatic function is suppressed in various diseases and that failure of glymphatic function in turn might contribute to pathology in neurodegenerative disorders, traumatic brain injury and stroke. read more read less

Topics:

Glymphatic system (63%)63% related to the paper
View PDF
1,144 Citations
open accessOpen access Journal Article DOI: 10.1007/S11064-006-9128-5
Glycogen Synthase Kinase-3 (GSK3): Inflammation, Diseases, and Therapeutics
Richard S. Jope1, Christopher J. Yuskaitis1, Eléonore Beurel1
22 Mar 2007 - Neurochemical Research

Abstract:

Deciphering what governs inflammation and its effects on tissues is vital for understanding many pathologies. The recent discovery that glycogen synthase kinase-3 (GSK3) promotes inflammation reveals a new component of its well-documented actions in several prevalent diseases which involve inflammation, including mood disorde... Deciphering what governs inflammation and its effects on tissues is vital for understanding many pathologies. The recent discovery that glycogen synthase kinase-3 (GSK3) promotes inflammation reveals a new component of its well-documented actions in several prevalent diseases which involve inflammation, including mood disorders, Alzheimer’s disease, diabetes, and cancer. Involvement in such disparate conditions stems from the widespread influences of GSK3 on many cellular functions, with this review focusing on its regulation of inflammatory processes. GSK3 promotes the production of inflammatory molecules and cell migration, which together make GSK3 a powerful regulator of inflammation, while GSK3 inhibition provides protection from inflammatory conditions in animal models. The involvement of GSK3 and inflammation in these diseases are highlighted. Thus, GSK3 may contribute not only to primary pathologies in these diseases, but also to the associated inflammation, suggesting that GSK3 inhibitors may have multiple effects influencing these conditions. read more read less

Topics:

GSK-3 (56%)56% related to the paper, Inflammation (51%)51% related to the paper
709 Citations
Journal Article DOI: 10.1007/S11064-005-6978-1
Agonistic Properties of Cannabidiol at 5-HT1a Receptors
Ethan B. Russo1, Andrea Burnett1, Brian Hall1, Keith K. Parker1
01 Aug 2005 - Neurochemical Research

Abstract:

Cannabidiol (CBD) is a major, biologically active, but psycho-inactive component of cannabis. In this cell culture-based report, CBD is shown to displace the agonist, [3H]8-OHDPAT from the cloned human 5-HT1a receptor in a concentration-dependent manner. In contrast, the major psychoactive component of cannabis, tetrahydrocan... Cannabidiol (CBD) is a major, biologically active, but psycho-inactive component of cannabis. In this cell culture-based report, CBD is shown to displace the agonist, [3H]8-OHDPAT from the cloned human 5-HT1a receptor in a concentration-dependent manner. In contrast, the major psychoactive component of cannabis, tetrahydrocannabinol (THC) does not displace agonist from the receptor in the same micromolar concentration range. In signal transduction studies, CBD acts as an agonist at the human 5-HT1a receptor as demonstrated in two related approaches. First, CBD increases [35S]GTPcS binding in this G protein coupled receptor system, as does the known agonist serotonin. Second, in this GPCR system, that is negatively coupled to cAMP production, both CBD and 5-HT decrease cAMP concentration at similar apparent levels of receptor occupancy, based upon displacement data. Preliminary comparative data is also presented from the cloned rat 5-HT2a receptor suggesting that CBD is active, but less so, relative to the human 5-HT1a receptor, in binding analyses. Overall, these studies demonstrate that CBD is a modest affinity agonist at the human 5-HT1a receptor. Additional work is required to compare CBD’s potential at other serotonin receptors and in other species. Finally, the results indicate that cannabidiol may have interesting and useful potential beyond the realm of cannabinoid receptors. read more read less

Topics:

Agonist (62%)62% related to the paper, Endogenous agonist (59%)59% related to the paper, Partial agonist (59%)59% related to the paper, Cannabinoid receptor type 2 (58%)58% related to the paper, GPR18 (58%)58% related to the paper
663 Citations
Journal Article DOI: 10.1007/BF00993263
Quantitative aspects of reactive gliosis: A review
William T. Norton1, Dennis A. Aquino1, Isao Hozumi1, Fung-Chow Chiu1, Celia F. Brosnan1
01 Sep 1992 - Neurochemical Research

Abstract:

Recent studies of gliosis in a variety of animal models are reviewed. The models include brain injury, neurotoxic damage, genetic diseases and inflammatory demyelination. These studies show that reactive gliosis is not a stereotypic response, but varies widely in duration, degree of hyperplasia, and time course of expression ... Recent studies of gliosis in a variety of animal models are reviewed. The models include brain injury, neurotoxic damage, genetic diseases and inflammatory demyelination. These studies show that reactive gliosis is not a stereotypic response, but varies widely in duration, degree of hyperplasia, and time course of expression of GFAP immunostaining, content and mRNA. We conclude that there are different biological mechanisms for induction and maintenance of reactive gliosis, which, depending on the kind of tissue damage, result in different expressions of the gliotic response. read more read less

Topics:

Gliosis (59%)59% related to the paper
545 Citations
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Neurochemical Research format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Neurochemical Research in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Neurochemical Research guidelines and auto format it.

2. Do you follow the Neurochemical Research guidelines?

Yes, the template is compliant with the Neurochemical Research guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Neurochemical Research?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Neurochemical Research citation style.

4. Can I use the Neurochemical Research templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Neurochemical Research.

5. Can I use a manuscript in Neurochemical Research that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Neurochemical Research that you can download at the end.

6. How long does it usually take you to format my papers in Neurochemical Research?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Neurochemical Research.

7. Where can I find the template for the Neurochemical Research?

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8. Can I reformat my paper to fit the Neurochemical Research's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Neurochemical Research is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Neurochemical Research?”

11. What is the output that I would get after using Neurochemical Research?

After writing your paper autoformatting in Neurochemical Research, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Neurochemical Research's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Neurochemical Research?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Neurochemical Research. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Neurochemical Research?

The 5 most common citation types in order of usage for Neurochemical Research are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Neurochemical Research?

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16. Can I download Neurochemical Research in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Neurochemical Research Endnote style according to Elsevier guidelines.

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