D
Douglas L. Feinstein
Researcher at University of Illinois at Chicago
Publications - 186
Citations - 16342
Douglas L. Feinstein is an academic researcher from University of Illinois at Chicago. The author has contributed to research in topics: Nitric oxide synthase & Neuroprotection. The author has an hindex of 63, co-authored 179 publications receiving 14553 citations. Previous affiliations of Douglas L. Feinstein include Veterans Health Administration & Cornell University.
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Journal ArticleDOI
Neuroinflammation in Alzheimer's disease
Michael T. Heneka,Monica J. Carson,Joseph El Khoury,Gary E. Landreth,Frederic Brosseron,Douglas L. Feinstein,Andreas H. Jacobs,Tony Wyss-Coray,Tony Wyss-Coray,Javier Vitorica,Richard M. Ransohoff,Karl Herrup,Sally A. Frautschy,Bente Finsen,Guy C. Brown,Alexei Verkhratsky,Alexei Verkhratsky,Alexei Verkhratsky,Koji Yamanaka,Jari Koistinaho,Eicke Latz,Eicke Latz,Annett Halle,Gabor C. Petzold,Terrence Town,Dave Morgan,Mari L. Shinohara,V. Hugh Perry,Clive Holmes,Clive Holmes,Nicolas G. Bazan,David J. Brooks,Stéphane Hunot,Bertrand Joseph,Nikolaus Deigendesch,Olga Garaschuk,Erik Boddeke,Charles A. Dinarello,John C.S. Breitner,Greg M. Cole,Douglas T. Golenbock,Markus P. Kummer +41 more
TL;DR: Genome-wide analysis suggests that several genes that increase the risk for sporadic Alzheimer's disease encode factors that regulate glial clearance of misfolded proteins and the inflammatory reaction.
Journal ArticleDOI
Synthesis of nitric oxide in CNS glial cells.
Sean Murphy,Martha L. Simmons,Luis Agulló,Agustina García,Douglas L. Feinstein,Elena Galea,Donald J. Reis,Dahlia Minc-Golomb,Joan P. Schwartz +8 more
TL;DR: The intention here is to evaluate the evidence for NO production from non-neuronal CNS sources and thus prompt discussion about potential 'nitrinergic' roles for glial cells.
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Lipid emulsion infusion rescues dogs from bupivacaine-induced cardiac toxicity
TL;DR: It was found that infusing a lipid emulsion during resuscitation from bupivacaine-induced cardiac toxicity substantially improved hemodynamics, pmO2, and pHm and increased survival in dogs.
Journal ArticleDOI
Induction of calcium-independent nitric oxide synthase activity in primary rat glial cultures.
TL;DR: It is concluded that glial cells of the central nervous system can express an inducible form of NOS similar to the inducable NOS of macrophages, which may contribute to the neuronal damage associated with cerebral ischemia and/or demyelinating diseases.
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Inhibition of mammalian nitric oxide synthases by agmatine, an endogenous polyamine formed by decarboxylation of arginine
TL;DR: Agmatine, decarboxylated arginine, may be an endogenous regulator of NO production in mammals and was a competitive NOS inhibitor but not an NO precursor.