M
Mihail Hristov
Researcher at RWTH Aachen University
Publications - 45
Citations - 6173
Mihail Hristov is an academic researcher from RWTH Aachen University. The author has contributed to research in topics: Progenitor cell & Endothelial stem cell. The author has an hindex of 34, co-authored 45 publications receiving 5772 citations. Previous affiliations of Mihail Hristov include Ludwig Maximilian University of Munich.
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Journal ArticleDOI
Delivery of microRNA-126 by apoptotic bodies induces CXCL12-dependent vascular protection
Alma Zernecke,Kiril Bidzhekov,Heidi Noels,Erdenechimeg Shagdarsuren,Lin Gan,Bernd Denecke,Mihail Hristov,Thomas Köppel,Maliheh Nazari Jahantigh,Esther Lutgens,Esther Lutgens,Shusheng Wang,Eric N. Olson,Andreas Schober,Christian Weber,Christian Weber +15 more
TL;DR: It is shown that endothelial cell–derived apoptotic bodies are generated during atherosclerosis and convey paracrine alarm signals to recipient vascular cells that trigger the production of CXCL12.
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Endothelial Progenitor Cells Mobilization, Differentiation, and Homing
TL;DR: Clinical studies using EPCs for neovascularization have just been started; however, the mechanisms stimulating or inhibiting the differentiation of EPC in vivo and the signals causing their migration and homing to sites of injured endothelium or extravascular tissue are largely unknown at present.
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Endothelial progenitor cells: characterization, pathophysiology, and possible clinical relevance
Mihail Hristov,Christian Weber +1 more
TL;DR: Recent clinical studies employing endothelial progenitor cells for neo‐vascularization of ischemic organs have just been published, however, the specificity of the observed positive clinical effects, the mechanisms regulating the differentiation of EPCs and their homing to sites of injured tissue remain partially unknown at present.
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Apoptotic bodies from endothelial cells enhance the number and initiate the differentiation of human endothelial progenitor cells in vitro.
TL;DR: The results suggest that apoptotic bodies from ECs are taken up by EPCs, increasing their number and differentiation state and may represent a new signaling pathway between progenitor and damaged somatic cells.
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Endothelial progenitor cells: isolation and characterization.
TL;DR: The mechanisms stimulating or inhibiting the differentiation of bone marrow-derived EPCs in vivo and the signals causing their adhesion, migration, and homing to sites of injured tissue are largely unknown at present.