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Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format Example of Therapeutic Hypothermia and Temperature Management format
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open access Open Access

Therapeutic Hypothermia and Temperature Management — Template for authors

Categories Rank Trend in last 3 yrs
Critical Care and Intensive Care Medicine #35 of 82 down down by 3 ranks
Anesthesiology and Pain Medicine #56 of 110 down down by 12 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 120 Published Papers | 253 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 29/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.1

5% from 2019

CiteRatio for Therapeutic Hypothermia and Temperature Management from 2016 - 2020
Year Value
2020 2.1
2019 2.2
2018 2.7
2017 2.6
2016 1.7
graph view Graph view
table view Table view

0.323

38% from 2019

SJR for Therapeutic Hypothermia and Temperature Management from 2016 - 2020
Year Value
2020 0.323
2019 0.519
2018 0.577
2017 0.549
2016 0.541
graph view Graph view
table view Table view

0.439

20% from 2019

SNIP for Therapeutic Hypothermia and Temperature Management from 2016 - 2020
Year Value
2020 0.439
2019 0.552
2018 0.539
2017 0.56
2016 0.93
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 38% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 20% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Therapeutic Hypothermia and Temperature Management

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Therapeutic Hypothermia and Temperature Management

Approved by publishing and review experts on SciSpace, this template is built as per for Therapeutic Hypothermia and Temperature Management formatting guidelines as mentioned in Mary Ann Liebert, Inc. author instructions. The current version was created on and has been used by 169 authors to write and format their manuscripts to this journal.

Medicine

i
Last updated on
28 Jun 2020
i
ISSN
2153-7658
i
Impact Factor
Medium - 0.919
i
Acceptance Rate
Not provided
i
Frequency
Not provided
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
i
Citation Type
Author Year
(Blonder et al., 1982)
i
Bibliography Example
Blonder, G. E., Tinkham, M., and Klapwijk, T. M. 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and super- current conversion. Phys Rev B, 25(7), 4515– 4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1089/THER.2019.0001
A New Vision for Therapeutic Hypothermia in the Era of Targeted Temperature Management: A Speculative Synthesis.
Travis C. Jackson1, Patrick M. Kochanek1

Abstract:

Three decades of animal studies have reproducibly shown that hypothermia is profoundly cerebroprotective during or after a central nervous system (CNS) insult. The success of hypothermia in preclinical acute brain injury has not only fostered continued interest in research on the classic secondary injury mechanisms that are p... Three decades of animal studies have reproducibly shown that hypothermia is profoundly cerebroprotective during or after a central nervous system (CNS) insult. The success of hypothermia in preclinical acute brain injury has not only fostered continued interest in research on the classic secondary injury mechanisms that are prevented or blunted by hypothermia but has also sparked a surge of new interest in elucidating beneficial signaling molecules that are increased by cooling. Ironically, while research into cold-induced neuroprotection is enjoying newfound interest in chronic neurodegenerative disease, conversely, the scope of the utility of therapeutic hypothermia (TH) across the field of acute brain injury is somewhat controversial and remains to be fully defined. This has led to the era of Targeted Temperature Management, which emphasizes a wider range of temperatures (33-36°C) showing benefit in acute brain injury. In this comprehensive review, we focus on our current understandings of the novel neuroprotective mechanisms activated by TH, and discuss the critical importance of developmental age germane to its clinical efficacy. We review emerging data on four cold stress hormones and three cold shock proteins that have generated new interest in hypothermia in the field of CNS injury, to create a framework for new frontiers in TH research. We make the case that further elucidation of novel cold responsive pathways might lead to major breakthroughs in the treatment of acute brain injury, chronic neurological diseases, and have broad potential implications for medicines of the distant future, including scenarios such as the prevention of adverse effects of long-duration spaceflight, among others. Finally, we introduce several new phrases that readily summarize the essence of the major concepts outlined by this review-namely, Ultramild Hypothermia, the "Responsivity of Cold Stress Pathways," and "Hypothermia in a Syringe." read more read less

Topics:

Targeted temperature management (54%)54% related to the paper
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51 Citations
open accessOpen access Journal Article DOI: 10.1089/THER.2013.0018
Comparative Effectiveness of Therapeutic Hypothermia After Out-of-Hospital Cardiac Arrest: Insight from a Large Data Registry.
Timothy J. Mader1, Brian H. Nathanson, William E. Soares1, Ryan A. Coute1, Bryan McNally2

Abstract:

This study was done to determine the effectiveness of therapeutic hypothermia (TH) after out-of-hospital cardiac arrest (OHCA) among a large cohort of adults in the Cardiac Arrest Registry to Enhance Survival (CARES), with an emphasis on subgroups with a nonshockable first documented rhythm. This was an IRB approved retrospec... This study was done to determine the effectiveness of therapeutic hypothermia (TH) after out-of-hospital cardiac arrest (OHCA) among a large cohort of adults in the Cardiac Arrest Registry to Enhance Survival (CARES), with an emphasis on subgroups with a nonshockable first documented rhythm. This was an IRB approved retrospective cohort study. All adult index events at participating sites from November 2010 to December 2013 were study eligible. All patient data elements were provided. Summary statistics were calculated for all patients with and without TH. For multivariate adjustment, a multilevel (i.e., hierarchical), mixed-effects logistic regression (MLR) model was used with hospitals treated as random effects. Propensity score matching (PSM) on both shockable and nonshockable patients was done as a sensitivity analysis. After predefined exclusions, our final sample size was 6369 records for analysis: shockable=2992 (47.0%); asystole=1657 (26.0%); pulseless electrical activity=1249 (19.6%); other unspecified nonshockable=471 (7.4%). Unadjusted differences in neurological status at hospital discharge with and without TH were similar (p=0.295). After multivariate adjustment, TH had either no association with good neurological status at hospital discharge or that TH was actually associated with worse neurological outcome, particularly in patients with a nonshockable first documented rhythm (i.e., for NS patients, MLR odds ratio for TH=1.444; 95% CI [1.039, 2.006] p=0.029, and OR=1.017, p=0.927 via PSM). Highlighting our limitations, we conclude that when TH is indiscriminately provided to a large population of OHCA survivors with a nonshockable first documented rhythm, evidence for its effectiveness is diminished. We suggest more uniform and rigid guidelines for application. read more read less

Topics:

Pulseless electrical activity (53%)53% related to the paper, Retrospective cohort study (51%)51% related to the paper
47 Citations
Journal Article DOI: 10.1089/THER.2015.0009
Therapeutic Hypothermia for the Treatment of Acute Myocardial Infarction - Combined analysis of the RAPID MI- ICE and the CHILL-MI trials

Abstract:

In the randomized rapid intravascular cooling in myocardial infarction as adjunctive to percutaneous coronary intervention (RAPID MI-ICE) and rapid endovascular catheter core cooling combined with cold saline as an adjunct to percutaneous coronary intervention for the treatment of acute myocardial infarction CHILL-MI studies,... In the randomized rapid intravascular cooling in myocardial infarction as adjunctive to percutaneous coronary intervention (RAPID MI-ICE) and rapid endovascular catheter core cooling combined with cold saline as an adjunct to percutaneous coronary intervention for the treatment of acute myocardial infarction CHILL-MI studies, hypothermia was rapidly induced in conscious patients with ST-elevation myocardial infarction (STEMI) by a combination of cold saline and endovascular cooling. Twenty patients in RAPID MI-ICE and 120 in CHILL-MI with large STEMIs, scheduled for primary percutaneous coronary intervention (PCI) within 30% of the left ventricle) exhibited significantly reduced IS/MaR in the hypothermia group (40.5, 27.0-57.6 vs. 55.1, 41.1-64.4, median, IQR; hypothermia n=42 vs. control n=37, p=0.03), while patients with MaR<30% did not show effect of hypothermia (35.8, 28.3-57.5 vs. 38.4, 27.4-59.7, median, IQR; hypothermia n=15 vs. control n=19, p=0.50). The prespecified pooled analysis of RAPID MI-ICE and CHILL-MI indicates a reduction of myocardial IS and reduction in heart failure by 1-3 hours with endovascular cooling in association with primary PCI of acute STEMI predominantly in patients with large area of myocardium at risk. (ClinicalTrials.gov id NCT00417638 and NCT01379261). read more read less

Topics:

Percutaneous coronary intervention (57%)57% related to the paper, Myocardial infarction (57%)57% related to the paper, Myocardial infarction diagnosis (55%)55% related to the paper, Hypothermia (53%)53% related to the paper, Conventional PCI (51%)51% related to the paper
45 Citations
open accessOpen access Journal Article DOI: 10.1089/THER.2013.0010
Mild hypothermia reduces tissue plasminogen activator-related hemorrhage and blood brain barrier disruption after experimental stroke.

Abstract:

Therapeutic hypothermia has shown neuroprotective promise, but whether it can be used to improve outcome in stroke has yet to be determined in patients. Recombinant tissue plasminogen activator (rt-PA) is only given to a minority of patients with acute ischemic stroke, and is not without risk, namely significant brain hemorrh... Therapeutic hypothermia has shown neuroprotective promise, but whether it can be used to improve outcome in stroke has yet to be determined in patients. Recombinant tissue plasminogen activator (rt-PA) is only given to a minority of patients with acute ischemic stroke, and is not without risk, namely significant brain hemorrhage.We explored whether mild hypothermia, in combination with rt-PA, influences the safety of rt-PA. Mice were subjected to middle cerebral artery occlusion (MCAO) using a filament model, followed by 24 hours reperfusion.Two paradigms were studied. In the first paradigm, cooling and rt-PA treatment began at the same time upon reperfusion, whereas in the second paradigm, cooling began soon after ischemia onset, and rt-PA began after rewarming and upon reperfusion. Experimental groups included: tPA treatment at normothermia (37°C), rt-PA treatment at hypothermia (33°C), no rt-PA at normothermia, and no rt-PA treatment at hypothermia. Infarct size, neurological deficit scores, blood brain barrier (BBB) permeability, brain hemorrhage, and expression of endogenous tissue plasminogen activator (tPA) and its inhibitor, plasminogen activator inhibitor (PAI-1) were assessed. For both paradigms, hypothermia reduced infarct size and neurological deficits compared to normothermia, regardless of whether rt-PA was given. rt-PA treatment increased brain hemorrhage and BBB disruption compared to normothermia, and this was prevented by cooling. However, mortality was higher when rt-PA and cooling were administered at the same time, beginning 1–2 hours post MCAO. Endogenous tPA expression was reduced in hypothermic mice, whereas PAI-1 levels were unchanged by cooling. In the setting of rt-PA treatment, hypothermia reduces brain hemorrhage, and BBB disruption, suggesting that combination therapy with mild hypothermia and rt-PA appears safe. read more read less

Topics:

Hypothermia (57%)57% related to the paper, Tissue plasminogen activator (55%)55% related to the paper, Stroke (54%)54% related to the paper, Plasminogen activator (54%)54% related to the paper, Ischemia (52%)52% related to the paper
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43 Citations
open accessOpen access Journal Article DOI: 10.1089/THER.2013.0024
Therapeutic Hypothermia in Children and Adults with Severe Traumatic Brain Injury
Anna Sandestig1, Bertil Romner1, Per-Olof Grände2

Abstract:

Great expectations have been raised about neuroprotection of therapeutic hypothermia in patients with traumatic brain injury (TBI) by analogy with its effects after heart arrest, neonatal asphyxia, and drowning in cold water. The aim of this study is to review our present knowledge of the effect of therapeutic hypothermia on ... Great expectations have been raised about neuroprotection of therapeutic hypothermia in patients with traumatic brain injury (TBI) by analogy with its effects after heart arrest, neonatal asphyxia, and drowning in cold water. The aim of this study is to review our present knowledge of the effect of therapeutic hypothermia on outcome in children and adults with severe TBI. A literature search for relevant articles in English published from year 2000 up to December 2013 found 19 studies. No signs of improvement in outcome from hypothermia were seen in the five pediatric studies. Varied results were reported in 14 studies on adult patients, 2 of which reported a tendency of higher mortality and worse neurological outcome, 4 reported lower mortality, and 9 reported favorable neurological outcome with hypothermia. The quality of several trials was low. The best-performed randomized studies showed no improvement in outcome by hypothermia-some even indicated worse outcome. TBI patients may suffer from hypothermia-induced pulmonary and coagulation side effects, from side effects of vasopressors when re-establishing the hypothermia-induced lowered blood pressure, and from a rebound increase in intracranial pressure (ICP) during and after rewarming. The difference between body temperature and temperature set by the biological thermostat may cause stress-induced worsening of the circulation and oxygenation in injured areas of the brain. These mechanisms may counteract neuroprotective effects of therapeutic hypothermia. We conclude that we still lack scientific support as a first-tier therapy for the use of therapeutic hypothermia in TBI patients for both adults and children, but it may still be an option as a second-tier therapy for refractory intracranial hypertension. read more read less

Topics:

Hypothermia (56%)56% related to the paper, Traumatic brain injury (56%)56% related to the paper, Intracranial pressure (51%)51% related to the paper, Asphyxia (50%)50% related to the paper
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43 Citations
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SHERPA/RoMEO Database

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RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

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S. No. Citation Style Type
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