Institution
Queens College
Education•New York, New York, United States•
About: Queens College is a education organization based out in New York, New York, United States. It is known for research contribution in the topics: Population & Opioid. The organization has 3271 authors who have published 5883 publications receiving 159021 citations.
Topics: Population, Opioid, Politics, Context (language use), Poison control
Papers published on a yearly basis
Papers
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TL;DR: New guidelines for recording ERPs are presented and criteria for publishing the results are presented, which allow different studies to be compared readily.
Abstract: Event-related potentials ~ERPs! recorded from the human scalp can provide important information about how the human brain normally processes information and about how this processing may go awry in neurological or psychiatric disorders. Scientists using or studying ERPs must strive to overcome the many technical problems that can occur in the recording and analysis of these potentials. The methods and the results of these ERP studies must be published in a way that allows other scientists to understand exactly what was done so that they can, if necessary, replicate the experiments. The data must then be analyzed and presented in a way that allows different studies to be compared readily. This paper presents guidelines for recording ERPs and criteria for publishing the results.
2,033 citations
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TL;DR: A theory is proposed for the development of filamentous organisms, based on the assumptions that the filaments are composed of cells which undergo changes of state under inputs they receive from their neighbors, and the cells produce outputs as determined by their state and the input they receive.
1,541 citations
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TL;DR: It is shown that the problem is NP-hard in all but one special case and the complexity of optimal fixed-priority scheduling algorithm is discussed.
1,230 citations
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TL;DR: The unexpected findings in these subjects were higher brain weights and greater number of neurons as compared to age‐matched nursing home control subjects, which suggest people may have had incipient Alzheimer's disease but escaped loss of large neurons, or started with larger brains and more large neurons and thus might be said to have had a greater reserve.
Abstract: Postmortem examination was performed on 137 residents (average age 85.5 years) of a skilled nursing facility whose mental status, memory, and functional status had been evaluated during life. Seventy-eight percent were demented using conservative criteria; 55% had characteristic Alzheimer's disease. Choline acetyltransferase and somatostatin were significantly reduced in the brains of patients with Alzheimer's disease as compared with age-matched nursing home control subjects, although the degree of the reduction was less severe than found in subjects less than 80 years of age. Ten subjects whose functional and cognitive performance was in the upper quintile of the nursing home residents, as good as or better than the performance of the upper quintile of residents without brain pathology (control subjects), showed the pathological features of mild Alzheimer's disease, with many neocortical plaques. Plaque counts were 80% of those of demented patients with Alzheimer's disease. Choline acetyltransferase and somatostatin levels were intermediate between controls and demented patients with Alzheimer's disease. The unexpected findings in these subjects were higher brain weights and greater number of neurons (greater than 90 micron 2 in a cross-sectional area in cerebral cortex) as compared to age-matched nursing home control subjects. These people may have had incipient Alzheimer's disease but escaped loss of large neurons, or alternatively, started with larger brains and more large neurons and thus might be said to have had a greater reserve.
1,163 citations
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Paris Descartes University1, Cornell University2, University of Massachusetts Medical School3, Spanish National Research Council4, University of Rome Tor Vergata5, Boston Children's Hospital6, University of Pittsburgh7, National Scientific and Technical Research Council8, National University of Cuyo9, Albert Einstein College of Medicine10, University of California, San Francisco11, University of New Mexico12, University of Split13, Goethe University Frankfurt14, University of Helsinki15, University of Salento16, German Cancer Research Center17, Virginia Commonwealth University18, St. Jude Children's Research Hospital19, Discovery Institute20, Harvard University21, University of Tromsø22, Eötvös Loránd University23, Hungarian Academy of Sciences24, New York University25, University of Vienna26, Babraham Institute27, University of South Australia28, Howard Hughes Medical Institute29, University of Texas Southwestern Medical Center30, University of Oviedo31, University of Graz32, National Institutes of Health33, Queens College34, City University of New York35, University of Tokyo36, University of Zurich37, Novartis38, Austrian Academy of Sciences39, University of Groningen40, University of Cambridge41, University of Padua42, University of Oxford43, University of Bern44, University of Oslo45, Foundation for Research & Technology – Hellas46, University of Crete47, Francis Crick Institute48, Osaka University49, Icahn School of Medicine at Mount Sinai50
TL;DR: A panel of leading experts in the field attempts here to define several autophagy‐related terms based on specific biochemical features to formulate recommendations that facilitate the dissemination of knowledge within and outside the field of autophagic research.
Abstract: Over the past two decades, the molecular machinery that underlies autophagic responses has been characterized with ever increasing precision in multiple model organisms. Moreover, it has become clear that autophagy and autophagy-related processes have profound implications for human pathophysiology. However, considerable confusion persists about the use of appropriate terms to indicate specific types of autophagy and some components of the autophagy machinery, which may have detrimental effects on the expansion of the field. Driven by the overt recognition of such a potential obstacle, a panel of leading experts in the field attempts here to define several autophagy-related terms based on specific biochemical features. The ultimate objective of this collaborative exchange is to formulate recommendations that facilitate the dissemination of knowledge within and outside the field of autophagy research.
1,095 citations
Authors
Showing all 3328 results
Name | H-index | Papers | Citations |
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Stuart A. Aaronson | 129 | 657 | 69633 |
Karen A. Matthews | 125 | 693 | 57426 |
Sheldon Cohen | 111 | 280 | 96037 |
Stephen B. Manuck | 94 | 369 | 29993 |
Ahmad R. Hariri | 92 | 350 | 36000 |
Hugo D. Critchley | 90 | 354 | 35610 |
Gavril W. Pasternak | 90 | 403 | 28604 |
Robert R. Alfano | 86 | 1252 | 33960 |
Taro Takahashi | 81 | 201 | 30388 |
Teresa J. Bandosz | 80 | 464 | 24499 |
Jeffrey H. Newcorn | 79 | 305 | 23912 |
John Monahan | 72 | 313 | 21833 |
James F. Rusling | 72 | 452 | 21436 |
Robert Bittman | 69 | 422 | 16583 |
Steve Smale | 68 | 161 | 27662 |