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Lorenzo Galluzzi

Researcher at Cornell University

Publications -  539
Citations -  91168

Lorenzo Galluzzi is an academic researcher from Cornell University. The author has contributed to research in topics: Immune system & Autophagy. The author has an hindex of 118, co-authored 477 publications receiving 71436 citations. Previous affiliations of Lorenzo Galluzzi include University of Modena and Reggio Emilia & University of Paris.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Mitochondrial Membrane Permeabilization in Cell Death

Guido Kroemer, +2 more
- 01 Jan 2007 - 
TL;DR: Once MMP has been induced, it causes the release of catabolic hydrolases and activators of such enzymes (including those of caspases) from mitochondria, meaning that mitochondria coordinate the late stage of cellular demise.
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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.

Lorenzo Galluzzi, +186 more
- 23 Jan 2018 - 
TL;DR: The Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives.
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Classification of cell death: recommendations of the Nomenclature Committee on Cell Death

Guido Kroemer, +29 more
- 25 Oct 2005 - 
TL;DR: This study details the 2009 recommendations of the NCCD on the use of cell death-related terminology including ‘entosis’, ‘mitotic catastrophe”,’ ‘necrosis‚ ‘necroptosis‚’ and ‘pyroptotic’.
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Immunogenic Cell Death in Cancer Therapy

Guido Kroemer, +3 more
- 21 Mar 2013 - 
TL;DR: It is postulate that ICD constitutes a prominent pathway for the activation of the immune system against cancer, which in turn determines the long-term success of anticancer therapies and its subversion by pathogens.