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Richard Vidal

Researcher at Fermilab

Publications -  686
Citations -  67220

Richard Vidal is an academic researcher from Fermilab. The author has contributed to research in topics: Large Hadron Collider & Standard Model. The author has an hindex of 113, co-authored 685 publications receiving 61464 citations. Previous affiliations of Richard Vidal include University of Rochester & Fairfield University.

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Journal ArticleDOI

Observation of a new boson at a mass of 125 GeV with the CMS experiment at the LHC

S. Chatrchyan, +2863 more
- 17 Sep 2012 - 
TL;DR: In this paper, results from searches for the standard model Higgs boson in proton-proton collisions at 7 and 8 TeV in the CMS experiment at the LHC, using data samples corresponding to integrated luminosities of up to 5.8 standard deviations.
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The CMS experiment at the CERN LHC

S. Chatrchyan, +3175 more
- 14 Aug 2008 - 
TL;DR: The Compact Muon Solenoid (CMS) detector at the Large Hadron Collider (LHC) at CERN as mentioned in this paper was designed to study proton-proton (and lead-lead) collisions at a centre-of-mass energy of 14 TeV (5.5 TeV nucleon-nucleon) and at luminosities up to 10(34)cm(-2)s(-1)
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Combined Measurement of the Higgs Boson Mass in pp Collisions at √s=7 and 8 TeV with the ATLAS and CMS Experiments

Georges Aad, +5120 more
- 14 May 2015 - 
TL;DR: A measurement of the Higgs boson mass is presented based on the combined data samples of the ATLAS and CMS experiments at the CERN LHC in the H→γγ and H→ZZ→4ℓ decay channels.
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CMS physics technical design report, volume II: Physics performance

G. L. Bayatian, +2063 more
- 01 Jun 2007 - 
TL;DR: In this article, the authors present a detailed analysis of the performance of the Large Hadron Collider (CMS) at 14 TeV and compare it with the state-of-the-art analytical tools.
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The Atlantic salmon genome provides insights into rediploidization

Sigbjørn Lien, +47 more
- 12 May 2016 - 
TL;DR: It is found that genes that were retained as duplicates after the teleost-specific whole-genome duplication 320 million years ago were not more likely to be retained after the Ss4R, and that the duplicate retention was not influenced to a great extent by the nature of the predicted protein interactions of the gene products.