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Anil Prasad

Researcher at Beth Israel Deaconess Medical Center

Publications -  66
Citations -  8189

Anil Prasad is an academic researcher from Beth Israel Deaconess Medical Center. The author has contributed to research in topics: Cancer & Adenocarcinoma. The author has an hindex of 31, co-authored 63 publications receiving 6997 citations. Previous affiliations of Anil Prasad include Veterans Health Administration & United States Department of Veterans Affairs.

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Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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MGMT Promoter Methylation and Field Defect in Sporadic Colorectal Cancer

Lanlan Shen, +13 more
- 21 Sep 2005 - 
TL;DR: Some colorectal cancers arise from a field defect defined by epigenetic inactivation of MGMT, and detection of this abnormality may ultimately be useful in risk assessment for coloreCTal cancer.
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Cannabidiol induces programmed cell death in breast cancer cells by coordinating the cross-talk between apoptosis and autophagy.

Ashutosh Shrivastava, +3 more
- 01 Jul 2011 - 
TL;DR: An intricate interplay between apoptosis and autophagy in CBD-treated breast cancer cells is revealed and the value of continued investigation into the potential use of CBD as an antineoplastic agent is highlighted.
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Regulation of CXCR4-mediated chemotaxis and chemoinvasion of breast cancer cells

Aaron Z. Fernandis, +4 more
- 08 Jan 2004 - 
TL;DR: It is shown that CXCL12 enhanced the chemotaxis, chemoinvasion and adhesive properties of breast cancer cells; parameters that are critical for development of metastasis.
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Bile acids in combination with low pH induce oxidative stress and oxidative DNA damage: relevance to the pathogenesis of Barrett’s oesophagus

Katerina Dvorak, +13 more
- 01 Jun 2007 - 
TL;DR: Test the hypothesis that bile acids and gastric acid, two components of refluxate, can induce oxidative stress and oxidative DNA damage and showed that 8-OH-dG is formed mainly in the epithelial cells in dysplastic Barrett’s oesophagus.