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Jun Ren

Researcher at University of Washington

Publications -  350
Citations -  28119

Jun Ren is an academic researcher from University of Washington. The author has contributed to research in topics: Medicine & Autophagy. The author has an hindex of 70, co-authored 298 publications receiving 22884 citations. Previous affiliations of Jun Ren include Peking Union Medical College & Fourth Military Medical University.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Empagliflozin rescues diabetic myocardial microvascular injury via AMPK-mediated inhibition of mitochondrial fission.

Hao Zhou, +6 more
- 01 May 2018 - 
TL;DR: Empagliflozin improved diabetic myocardial structure and function, preserved cardiac microvascular barrier function and integrity, sustained eNOS phosphorylation and endothelium-dependent relaxation, as well as improved microvessel density and perfusion.
Journal ArticleDOI

Mitochondrial biogenesis in the metabolic syndrome and cardiovascular disease.

Jun Ren, +3 more
- 20 Aug 2010 - 
TL;DR: In this paper, a review explores mechanisms of mitochondrial dysfunction with a focus on impaired oxidative phosphorylation and mitochondrial biogenesis in the pathophysiology of metabolic heart disease, and concludes that these mitochondrial abnormalities can predispose a metabolic cardiomyopathy characterized by diastolic dysfunction.