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Zhifen Yang

Researcher at Stanford University

Publications -  25
Citations -  16680

Zhifen Yang is an academic researcher from Stanford University. The author has contributed to research in topics: Autophagy & BAG3. The author has an hindex of 20, co-authored 22 publications receiving 14656 citations. Previous affiliations of Zhifen Yang include University of Michigan & Sanford-Burnham Institute for Medical Research.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Eaten alive: a history of macroautophagy.

TL;DR: This review traces the key findings that led to the current molecular understanding of this complex process, which is involved in physiology, development, lifespan and a wide range of diseases, including cancer, neurodegeneration and microbial infection.
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Mammalian autophagy: core molecular machinery and signaling regulation

TL;DR: Recent advances in identifying and understanding the core molecular machinery and signaling pathways that are involved in mammalian autophagy are highlighted.
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Endoplasmic Reticulum Stress Triggers Autophagy

TL;DR: The results indicate that ER stress can induce an autophagic response, and it is found that Atg1 had high kinase activity during ER stress-induced autophagy.