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Glenn Randall

Researcher at University of Chicago

Publications -  88
Citations -  13141

Glenn Randall is an academic researcher from University of Chicago. The author has contributed to research in topics: Viral replication & Hepatitis C virus. The author has an hindex of 36, co-authored 72 publications receiving 10608 citations. Previous affiliations of Glenn Randall include Argonne National Laboratory & Rockefeller University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Identification of microRNAs of the herpesvirus family

Sébastien Pfeffer, +19 more
- 16 Feb 2005 - 
TL;DR: To identify other miRNA genes in pathogenic viruses, a new miRNA gene prediction method with small-RNA cloning from several virus-infected cell types was combined and predicted miRNAs in several large DNA viruses.
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Cellular cofactors affecting hepatitis C virus infection and replication

Glenn Randall, +19 more
- 31 Jul 2007 - 
TL;DR: The data define 26 host genes that modulate HCV infection and indicate that the requirement for functional RNAi for HCV replication is dominant over any antiviral activity this pathway may exert against HCV.
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Dengue Virus-Induced Autophagy Regulates Lipid Metabolism

Nicholas S. Heaton, +1 more
- 18 Nov 2010 - 
TL;DR: A role for autophagy is defined in dengue virus infection and a mechanism by which viruses can alter cellular lipid metabolism to promote their replication is provided.
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Dengue virus nonstructural protein 3 redistributes fatty acid synthase to sites of viral replication and increases cellular fatty acid synthesis

Nicholas S. Heaton, +6 more
- 05 Oct 2010 - 
TL;DR: Mechanistic insight into DENV membrane remodeling is provided and the potential for the development of therapeutics that inhibit DENV replication by targeting the fatty acid biosynthetic pathway is highlighted.