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Ioannis P. Nezis

Researcher at University of Warwick

Publications -  80
Citations -  15127

Ioannis P. Nezis is an academic researcher from University of Warwick. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 35, co-authored 70 publications receiving 12412 citations. Previous affiliations of Ioannis P. Nezis include Oslo University Hospital & National and Kapodistrian University of Athens.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Ref(2)P, the Drosophila melanogaster homologue of mammalian p62, is required for the formation of protein aggregates in adult brain.

Ioannis P. Nezis, +8 more
- 24 Mar 2008 - 
TL;DR: It is demonstrated that Ref(2)P localizes to age-induced protein aggregates as well as to aggregates caused by reduced autophagic or proteasomal activity, and a major role is revealed in the formation of ubiquitin-positive protein aggregation both under physiological conditions and when normal protein turnover is inhibited.
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ESCRTs and Fab1 Regulate Distinct Steps of Autophagy

Tor Erik Rusten, +10 more
- 23 Oct 2007 - 
TL;DR: This study links ESCRT function with autophagy and aggregate-induced neurodegeneration, thereby providing a plausible explanation for the fact that ESCRT mutations are involved in inherited neurodegenersative disease in humans.