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Nobuyuki Nukina

Researcher at Doshisha University

Publications -  228
Citations -  29108

Nobuyuki Nukina is an academic researcher from Doshisha University. The author has contributed to research in topics: Huntingtin & Huntingtin Protein. The author has an hindex of 73, co-authored 221 publications receiving 25632 citations. Previous affiliations of Nobuyuki Nukina include Brigham and Women's Hospital & University of Tsukuba.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)

Takeshi Iwatsubo, +5 more
- 01 Jul 1994 - 
TL;DR: Two familial AD cortices with the mutation of beta-amyloid protein precursor 717 (beta APP717) (Val to Ile) showed a remarkable predominance of A beta 42(43)-positive, A beta 40-negative plaques.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Trehalose alleviates polyglutamine-mediated pathology in a mouse model of Huntington disease.

Motomasa Tanaka, +8 more
- 18 Jan 2004 - 
TL;DR: Oral administration of trehalose, the most effective of these disaccharides, decreased polyglutamine aggregates in cerebrum and liver, improved motor dysfunction and extended lifespan in a transgenic mouse model of Huntington disease, making trehalOSE promising as a therapeutic drug or lead compound for the treatment of polyglUTamine diseases.