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Jay E. Brenman

Researcher at University of North Carolina at Chapel Hill

Publications -  52
Citations -  11012

Jay E. Brenman is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: AMPK & AMP-activated protein kinase. The author has an hindex of 30, co-authored 51 publications receiving 9922 citations. Previous affiliations of Jay E. Brenman include University of California, San Francisco & University of California, Berkeley.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Interaction of Nitric Oxide Synthase with the Postsynaptic Density Protein PSD-95 and α1-Syntrophin Mediated by PDZ Domains

Jay E. Brenman, +11 more
- 08 Mar 1996 - 
TL;DR: It is shown that the N-terminus of nNOS, which contains a PDZ protein motif, interacts with similar motifs in postsynaptic density-95 protein (PSD-95) and a related novel protein, PSD-93.
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Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy.

Jay E. Brenman, +4 more
- 08 Sep 1995 - 
TL;DR: In this paper, it was shown that dystrophin complex interacts with an N-terminal domain of nNOS that contains a GLGF motif, which may contribute to preferential degeneration of fast-twitch muscle fibers in Duchenne muscular dystrophy.
Journal Article

Expression of nitric oxide synthase in human central nervous system tumors.

Charles Cobbs, +4 more
- 15 Feb 1995 - 
TL;DR: The data indicate that malignant central nervous system neoplasms express unexpectedly high levels of NOS and suggest that NO production may be associated with pathophysiological processes important to these tumors.
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Cloning and characterization of postsynaptic density 93, a nitric oxide synthase interacting protein.

Jay E. Brenman, +4 more
- 01 Dec 1996 - 
TL;DR: It is demonstrated that the PDZ domain at the N terminus of nNOS is required, but it is not sufficient for interaction with PSD-93/95, which may help account for the functional as well as the physical coupling ofnNOS to NMDA receptors.