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Marina Jendrach

Researcher at Goethe University Frankfurt

Publications -  43
Citations -  13181

Marina Jendrach is an academic researcher from Goethe University Frankfurt. The author has contributed to research in topics: Mitochondrion & Autophagy. The author has an hindex of 29, co-authored 41 publications receiving 11456 citations. Previous affiliations of Marina Jendrach include Ruhr University Bochum & Charité.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Mitochondrial dysfunction: an early event in Alzheimer pathology accumulates with age in AD transgenic mice.

Susanne Hauptmann, +8 more
- 01 Oct 2009 - 
TL;DR: It is concluded that Ass dependent mitochondrial dysfunction starts already at 3 months in this AD model before extracellular deposition of Ass and progression accelerates substantially with aging.
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Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration.

Suzana Gispert, +24 more
- 03 Jun 2009 - 
TL;DR: Older Pink1−/− mice show increasing mitochondrial dysfunction resulting in impaired neural activity similar to PD, in absence of overt neuronal death.
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Mitochondrion-Derived Reactive Oxygen Species Lead to Enhanced Amyloid Beta Formation

Kristina Leuner, +17 more
- 15 Jun 2012 - 
TL;DR: It is shown for the first time that mitochondrion-derived ROS are sufficient to trigger Aβ production in vitro and in vivo and that Aβ itself leads to mitochondrial dysfunction and increased ROS levels.