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Usha Nair

Researcher at University of Michigan

Publications -  28
Citations -  8373

Usha Nair is an academic researcher from University of Michigan. The author has contributed to research in topics: Autophagy & Autophagosome. The author has an hindex of 23, co-authored 28 publications receiving 7198 citations. Previous affiliations of Usha Nair include Texas A&M University.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Endoplasmic Reticulum Stress Triggers Autophagy

Tomohiro Yorimitsu, +3 more
- 06 Oct 2006 - 
TL;DR: The results indicate that ER stress can induce an autophagic response, and it is found that Atg1 had high kinase activity during ER stress-induced autophagy.
Journal ArticleDOI

SNARE proteins are required for macroautophagy

Usha Nair, +15 more
- 22 Jul 2011 - 
TL;DR: Evidence is provided for the involvement of exocytic Q/t-SNAREs in autophagosome formation, acting in the recruitment of key autophagy components to the site of autophagic formation, and in regulating the organization of Atg9 into tubulovesicular clusters.
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The Atg1 kinase complex is involved in the regulation of protein recruitment to initiate sequestering vesicle formation for nonspecific autophagy in Saccharomyces cerevisiae.

Heesun Cheong, +3 more
- 12 Dec 2007 - 
TL;DR: PAS assembly during nonspecific autophagy is studied, using an atg11Delta mutant background to eliminate the PAS formation that occurs during vegetative growth and it is found that protein complexes containing the Atg1 kinase have two roles for PAS Formation during nonsPecific autophileagy.
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Atg9 cycles between mitochondria and the pre-autophagosomal structure in yeasts.

Fulvio Reggiori, +3 more
- 11 May 2005 - 
TL;DR: It is demonstrated that Atg9 shuttles between this location and mitochondria, which supports a new model where mitochondria may provide at least part of the autophagosomal lipids and suggest a novel cellular function for this well-studied organelle.