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Cathleen R. Carlin

Researcher at Case Western Reserve University

Publications -  58
Citations -  11443

Cathleen R. Carlin is an academic researcher from Case Western Reserve University. The author has contributed to research in topics: Endosome & Receptor. The author has an hindex of 28, co-authored 56 publications receiving 9965 citations. Previous affiliations of Cathleen R. Carlin include Boston Children's Hospital & Saint Louis University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

STAT3 and epithelial-mesenchymal transitions in carcinomas.

Michael K. Wendt, +3 more
TL;DR: The molecular, cellular, and microenvironmental factors that contribute to the pathophysiological activities of STAT3 during its regulation of EMT programs in human carcinomas are reviewed.
Journal ArticleDOI

Epidermal growth factor receptor is down-regulated by a 10,400 MW protein encoded by the E3 region of adenovirus

Cathleen R. Carlin, +4 more
- 07 Apr 1989 - 
TL;DR: EGF-R is down-regulated in an analogous manner during early infection of a variety of cell types by group C human adenoviruses by a novel mechanism that involves the formation of hetero-oligomers composed of 10.4K and EGF- R.
Journal ArticleDOI

Epithelial-to-Mesenchymal Transition Promotes Breast Cancer Progression via a Fibronectin-Dependent Stat3 Signaling Pathway

Nikolas G. Balanis, +5 more
- 21 Jun 2013 - 
TL;DR: The data demonstrate that matrix-initiated signaling is sufficient to drive STAT3 activation, a reaction that is facilitated by EMT during BC metastatic progression, and demonstrates a novel mechanism by which STAT3 becomes activated by the extracellular matrix independent of the canonical EGF receptor signaling network.