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Shengkan Jin

Researcher at Rutgers University

Publications -  41
Citations -  22999

Shengkan Jin is an academic researcher from Rutgers University. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 31, co-authored 40 publications receiving 20821 citations. Previous affiliations of Shengkan Jin include University of Medicine and Dentistry of New Jersey.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky, +235 more
- 16 Feb 2008 - 
TL;DR: A set of guidelines for the selection and interpretation of the methods that can be used by investigators who are attempting to examine macroautophagy and related processes, as well as by reviewers who need to provide realistic and reasonable critiques of papers that investigate these processes are presented.
Journal ArticleDOI

Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor.

Zhenyu Yue, +4 more
- 09 Dec 2003 - 
TL;DR: It is demonstrated that beclin 1 is a critical component of mammalian autophagy and a role forAutophagy in tumor suppression is established, and mutations in other genes operating in this pathway may contribute to tumor formation through deregulation of Autophagy.
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Autophagy promotes tumor cell survival and restricts necrosis, inflammation, and tumorigenesis

Kurt Degenhardt, +19 more
- 01 Jul 2006 - 
TL;DR: Limiting autophagy under conditions of nutrient limitation can restore cell death to apoptosis-refractory tumors, but this necrosis is associated with inflammation and accelerated tumor growth.