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Cheryl L. Walker

Researcher at Baylor College of Medicine

Publications -  225
Citations -  23802

Cheryl L. Walker is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Uterine leiomyoma & Tumor suppressor gene. The author has an hindex of 60, co-authored 220 publications receiving 21020 citations. Previous affiliations of Cheryl L. Walker include Texas A&M Health Science Center College of Medicine & Texas A&M University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky, +1287 more
- 01 Apr 2012 - 
TL;DR: These guidelines are presented for the selection and interpretation of methods for use by investigators who aim to examine macroautophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The Energy Sensing LKB1-AMPK Pathway Regulates p27kip1 Phosphorylation Mediating the Decision to Enter Autophagy or Apoptosis

Jiyong Liang, +12 more
- 01 Feb 2007 - 
TL;DR: LKB1–AMPK pathway-dependent phosphorylation of p27 at Thr 198 stabilizes p27 and permits cells to survive growth factor withdrawal and metabolic stress through autophagy, which may contribute to tumour-cell survival under conditions of growth factor deprivation, disrupted nutrient and energy metabolism, or during stress of chemotherapy.
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Cyclins and cell cycle checkpoints

David G. Johnson, +1 more
- 01 Jan 1999 - 
TL;DR: Several therapeutic agents, such as DNA-damaging drugs, microtubule inhibitors, antimetabolites, and topoisomerase inhibitors, take advantage of disruption in normal cell cycle regulation to target checkpoint controls and ultimately induce growth arrest or apoptosis of neoplastic cells.
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ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS

Angela Alexander, +15 more
- 02 Mar 2010 - 
TL;DR: A cytoplasmic pathway for ROS-induced ATM activation of TSC2 to regulate mTORC1 signaling and autophagy is identified, identifying an integration node for the cellular damage response with key pathways involved in metabolism, protein synthesis, and cell survival.